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Biomedical subjects

W H Krause

Publications and source records attributed to W H Krause.

At least 19 recordsLinked to original sources
PubMed1990

[Psychosomatic rehabilitation].

In rehabilitation medicine a multi-axial descriptive and theoretical model must be applied to do justice to the manifold aspects. In particular for psychosomatic medicine, but to the same extent also for disciplines concerned with the individual organs, it will necessary to pay attention to the social-interactional as well as to the development psychology aspects of these diseases and to their anatomic pathology and physiological pathology developments. The tasks and aims of psychosomatic rehabilitation medicine are described: diagnostics, details of hospitalisation and psychotherapy are described. To enhance the therapeutic value of the psychosomatic hospital somatotherapy and psychotherapy are co-ordinated for the patient's benefit. The special importance of psychosomatic rehabilitation for prevention is pointed out. A new, comprehensive re-orientation is aimed at to help patients to lead a generally healthier and more effective life.

Combined Modality Therapy↗
PubMed1977

[Dysfibrinogenemia. A new case: dysfibrinogenemia Giessen III (author's transl)].

A new case of dysfibrinogenemia is reported which shows no signs of a haermorrhagic diathesis (dysfibrinogenemia Giessen III). The abnormal fibrinogen was detected by only slight but characteristic alterations of some parameter of the coagulation analysis (prolonged clotting times after addition of thrombin, Reptilase and thrombin coagulase; low fibrinogen concentration determined by methods based on clot formation in comparison to the immunological fibrinogen determination; delayed fibrin polymerization). In addition, clinical features and diagnosis of dysfibrinogenemia are described in general.

Batroxobin↗
PubMed1975

[Hypodysfibrinogenemia: fibrinogen giessen II (author's transl)].

A new case of facultative hemorrhagic diathesis is described in a 19 year old female and the coagulatory anomaly examined. The experiments show that the coagulatory disturbance should be ascribed to a defective aggregation of fibrin monomers associated with hypofibrinogenemia. Some of its characteristics are the markedly prolonged thrombin-, reptilase-time as well prolonged prothrombin and partial thromboplastin time. The plasma fibrinogen concentration measured by the immunologic method is reduced. The streptokinase induced digestion of fibrinogen Giessen II plasma occurred at a slower rate than normal plasma with persistence of the large fibrinogen degradation products. Fibrinogen Giessen II appears to be a congenital hypodysfibrinogenemia with abnormality of fibrinogen resulting in delayed coagulation and a retarded fibrinogenolysis rate.

Adult↗