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Biomedical subjects

W Lisch

Publications and source records attributed to W Lisch.

49 records · Page 3Linked to original sources
PubMed1982

[Differential diagnosis of hereditary vitreoretinal degenerations ].

Vitreoretinal degenerations constitute the main cause of manifestation of an amotio retinae during youth. Seven different hereditary forms can be distinguished. For the individual types, characteristic or obligatory symptoms can be postulated. In this connection, five main symptoms of hereditary vitreo-retinal degenerations are presented: (1) foveal retionoschisis, (2) "white plains", (3) peripheral retinoschisis, (4) vitreal strands with insertion on the retina, (5) peripheral band-shaped vitreoretinal degeneration.

Diagnosis, Differential↗
PubMed1980

[The various stages of vitelliform macular degeneration].

Hereditary autosomal dominant vitelliform macular degeneration and Best's macular degeneration are synonyma. The different namens for this disease rise from its varying clinical manifestation. The age at which it becomes manifest also varies. Our findings in two families with this hereditary foveal disease confirm that the macular changes are polymorphous. Nine different characteristic states of vitelliform macular degeneration can be distinguished. The vitelliform stage may represent a previtelliform or a resorptive stage. The differential diagnosis between vitelliform disc and vitelliform cyst can only be made unequivocally by fluorescein angiography. If the vitelliform cyst is scarred directly the scar is sharply delineated. A vitelliform stage or a pseudohypopyon stage may develop, especially form larger vitelliform cysts. In such cases the final stage may be a poorly delineated foveal scar. Occassionally, a mixed picture of two stages is observed, e.g., of cystic and scarred states. Exact knowledge of all stages of the disease is of great interest with resepct to differential diagnosis.

Adolescent↗
PubMed1980

[Clinical appearance of corneal lattice-type dystrophy (author's transl)].

Autosomal dominant hereditary lattice-type corneal dystrophy is a purely corneal hereditary metabolic disturbance, i.e. a localized amyloidosis. The author describes the findings in a family presenting with this disease. The condition progresses slowly; three stages may be distinguished, namely stage A, characterized by paracentrally arranged reticular lattice lines; stage B, characterized by paracentral lattice lines and patchy central subepithelial opacifications, and stage C, distinguished by paracentral lattice lines and diffuse white central subepithelial opacifications. Frequently, lattice-type corneal dystrophy is accompanied by symptoms of a pseudo-inflammation, e.g., photophobia, epiphora, "red eye" and pain due to recurrent corneal erosions. With regard to penetrating keratoplasty, changes of the donor cornea after a certain period of latency may be observed lattice-type corneal dystrophy as a result of which three types of opacifications may occur: 1) peripheral lattice lines and spots in the stroma; 2) diffuse whitish subepithelial opacifications, and 3) recurrent straited eruptions of the corneal epithelium.

Adult↗
PubMed1976

[Symmetrical carotid aneurysm on both sides (author's transl)].

The case of a 44-year-old female patient with angiographically established symmetrical aneurysm of the internal carotid artery on both sides near the origin of the arteria ophthalmica is reported. The right ophthalmic artery is not perceptible for a distance of 2 mm in its early upper portion. The left ophthalmic artery is well filled. The visual capacity of the right eye is reduced to 1/24, the left eye has one of 1.0. The lower half of the visual field including the fixation point fails. The left visual field is characterized by a pronounced irregular concentrical narrowing with preserved fixation point. The different filling of the ophthalmic arteries is responsible for the difference in the visual capacity and in the visual fields of both eyes. Because, neurosurgically, only a resection of both aneurysms comes into question, one desists from this proceeding because of the extraordinary high risk of such an operation.

Adult↗
PubMed1976

[Primary hereditary band-shaped corneal dystrophy and its association with other hereditary corneal lesions (author's transl)].

The results of examinations of members of a large Tyrolian family tree with primary band-shaped corneal dystrophy and other hereditary corneal leasons are reported. Corneal lesions occurred in 16 family members. In three male members, the primary band-shaped corneal dystrophy is associated either with parenchymal opacification and endo-epithelial dystrophy or epithelial dystrophy or with parenchymal opacification and epithelial dystrophy. In some cases, various corneal dystrophies (parenchymal opacification, endo-epithelial dystrophy, cornea guttata, epithelial dystrophy, band-shaped nasal corneal dystrophy) exist without band-shaped corneal dystrophy. A polyphenic gene is responsible for the manifestation of the various corneal lesions. The occurrence of typical primary band-shaped corneal dystrophy only in male family members can be explained by sex-linked inheritance. Because of some flutuations in the expression, the primary band-shaped corneal dystrophy can only be manifested slightly on the nasal border. The hereditary character of the occurrence of the primary band-shaped corneal dystrophy and of the closely related other corneal dystrophies together with the already in 1974 observed association with keratokonus is stressed. Our observations show the variety of the manifestations of a polyphenic gene in the corneal region.

Adolescent↗
PubMed1976

[Iridoschisis (author's transl)].

The alterations characteristic for Iridoschisis (type I and II) are discussed in the light of our own observations. Iridoschisis must be differentiated from the essential progressive iris atrophy, from the dysgenesis mesodermalis corneae et iridis (Rieger), the Chandler syndrome and from the genuine polycoria. Because of the genetic inter-relationships between Iridoschisis, essential promalis corneae and iridis, a predisposition for the occurrence of these alterations has to be taken into account.

Aged↗
PubMed1976

[Corneal lesion by vacuum extraction (author's transl)].

The case of a 6-year-old boy is reported who was delivered out of a frontal position by application of the vacuum cup to the left part of the forehead. His left eye exhibits a pronounced somewhat irregular astigmatism. There are some vertical descemet tears. Because of the application of the vacuum pump close to the left bulbus, an increase of the tissue tension and consequently a deformation of the bulbus occurred by the vacuum in the bulbar region. The described lesions parallel the vertical descement tears which occur in forceps-delivery. Owing to the greater horizontal corneal diameter, a more pronounced extension of the cornea in the horizontal meridian occurs, which would explain the vertical course of the descemet tears. The obstetric contraindication for the application of the vacuum extraction in case of a frontal or facial position of the fetus is justified also from the ophthalmological point of view because of the risk of occurrence of irreversible corneal lesions.

Astigmatism↗