Postradiation chondroblastic sarcoma of bone: case report and review of the literature.
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Aged↗
Biomedical subjects
W S Lacorte
Publications and source records attributed to W S Lacorte.
Cryptococcal intracerebral mass lesions: CT-pathologic considerations.
Two patients with cryptococcal intracerebral disease showed CT evidence of multiple round hypodense nonenhancing lesions in the gray matter. These lesions proved to be gelatinous pseudocysts rather than granulomas or abscesses. Pseudocysts may occur alone or with meningitis or granulomas. Recognition of these will avoid confusion with other infectious, vascular, or neoplastic conditions.
Adult↗
Computerized tomographic abnormalities in patients with transient episodes of focal neurological dysfunction.
Forty patients with episodes of transient neurological dysfunction lasting less than 24 hr had abnormal CT scan findings. Eight patients had intracranial mass lesions and 32 had vascular lesions i.e. ischemic, infarction, hematoma. Seventeen patients had hypodense lesions consistent with tissue infarction; whereas 12 patients had isodense enhancing lesions consisting with tissue ischemia. In eight patients with isodense enhancing lesions, this was no neurological deterioration following carotid endarterectomy. In 3 patients who had transient ischemic attacks with CT evidence of hypodense lesions there was clinical and CT deterioration following carotid surgery.
Cerebral Hemorrhage↗
Enhancing cerebellar infarction.
Five patients with CT evidence of enhancing cerebellar infarcts are analyzed. This represented 12% of cerebellar infarcts diagnosed by CT scan. The pattern of cerebellar linear enhancing bands which are contiguous with a hypo- or isodense lesion is quite characteristic of cerebellar infarction. In two cases, the lesion appeared isodense on the plain scan and therefore the enhanced scan was crucial to establishing the diagnosis of a cerebellar infarction. The presence of nodular or ring enhancement is sometimes seen with cerebellar infarction but is more common of neoplastic disorders, vascular malformations, or infectious-inflammatory disorders. The enhancement pattern of the cerebellar infarctions developed within the initial seven days after the onset of neurological deficit, and this is earlier than the enhancement pattern of supratentorial ischemic vascular lesions.
Cerebellum↗