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W Zuzewicz

Publications and source records attributed to W Zuzewicz.

12 recordsLinked to original sources
PubMed1982

[Multi-shift work and work under conditions of jet lag. Questionnaire studies].

The authors discuss the disturbances in the biorhythm - sleep - waking in the workers who are on duty at the posts of command and in communication ouration pilots surpassing the time zones. The questionnaire method was used. The resultant desynchronization of the biological rhythm is exhibited mainly through the subjective feeling of fatigue and sleepiness. Adaptation and readaptation to changing working conditions occurs.

Adaptation, Physiological↗
PubMedDate not supplied

Ethyl alcohol effect on the visual evoked potential.

The investigations were carried out in 30 clinically healthy pilots who were given 1 g/kg of body weight of ethanol orally. Visual evoked potentials were recorded 30 and 60 minutes after alcohol ingestion in the occipital leads using an ANOPS-10 computer. It was found that ethanol caused prolongation of the latency time of all components parallel with rising concentration of ethanol in the blood. The amplitude of the late components, i.e.-N2-P2-P2-N3 and N3-P3 30 minutes after alcohol ingestion was raised, and after 60 minutes it was reduced. The analysis of the individual visual evoked potentials demonstrated three types of individual reactions to ethanol. Type I--increasing depression of the amplitude of the components with rising blood alcohol concentration, type II--rising amplitude, mainly of the late components, type III--amplitude rise in the 30th minute followed by amplitude fall in the 60th minute without reaching, however, the initial level. These changes of the visual evoked potentials suggest that alcohol exerts in the first place an effect on the subcortical structures and that this effect is a phasic one.

Adult↗
PubMedDate not supplied

Variability of the averaged evoked potentials in healthy subjects after 24-hour sleep deprivation.

Variability of the averaged evoked visual potentials in healthy subjects after 24-hour sleep deprivation. Acta Physiol. Pol., 1977, 28 (1): 61-69. The authors evaluated the effects of 24-hour sleep deprivation on visual evoked potentials in 20 pilots aged 20 to 40 years, clinically healthy and with normal bioelectric cerebral activity. A slight reduction in the latency time of components was observed, mainly in the non-specific part of the visual evoked potentials, and a rise in the P3-N4 amplitude. An analysis of the duration of potential components suggests that sleep deprivation causes disturbances in the equilibrium between the processes of excitation and inhibition from non-specific structures (with increased excitation). All parameters assessed may evidence increased excitation of the central nervous system after activation with insomnia + light flashes, and may be useful for extending the diagnostic possibilities in cases of borderline pathological changes in cerebral bioelectric activity.

Adult↗
PubMedDate not supplied

[Effect of phenactil (chlorpromazine) on cortical visual evoked potentials in clinically healthy subjects with normal and abnormal electroencephalograms].

The results of a study of averaged cortical visual evoked potentials in healthy subjects with abnormal EEG records and in subjects with normal EEG are presented. The investigations were performed at rest and after activation with chlorpromazine. The potentials were recorded from typical sites in the visual cortex (O2--CZ and O1--CZ). Stimulation with flashes at 1 c/sec., 40 msec. duration, 2000 lux light intensity at a distance of 60 cm from the eyes was applied. The pupils were dilated with homatropine. The mean amplitudes of potential components measured by the method peak-to-peak, the mean latency time, and the mean duration of components were measured. It was found that at rest the inter-group differences included an amplitude rise at the time of change of the specific part into non-specific and the change of the evoked potential to the after-discharge, and the duration of positive components in the non-specific part prevailed over the negative ones. Chlorpromazine changed somewhat the amplitude of components (fall in the specific part and rise in the non-spefific one) but the differences were statistically not significant. Similar changes were observed in the latency time of various components (prolongation of latency time of late components was statistically not significant). The results suggest differences in the reactivity of cerebral structures of these groups at rest as well as after chlorpromazine activation.

Chlorpromazine↗