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Xiaozhong Shen

Publications and source records attributed to Xiaozhong Shen.

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Timing matters: Impact of covalent BTK inhibitor dose modifications on outcomes in chronic lymphocytic leukemia/small lymphocytic leukemia-A 7-year real-world study.

BACKGROUND: Covalent BTK inhibitors (cBTKis) are the cornerstone of chronic lymphocytic leukemia (CLL)/small lymphocytic leukemia (SLL) therapy, yet real-world data on dose modifications and their differential impact on long-term outcomes remain incompletely defined. This study investigated the incidence, timing, and the effectiveness of drug switching in a real-world CLL cohort. METHODS: In this 7-year retrospective real-world study, 324 CLL/SLL patients treated at a specialized Shanghai outpatient clinic (April 2018-April 2025; median follow-up, 42 months) were analyzed. Dose modifications were classified as dose interruption (DI) or dose reduction (DR). Their prognostic impact on progression-free (PFS) and overall survival (OS) was assessed by Kaplan-Meier analysis and multivariate Cox regression. RESULTS: The 42-month PFS rate was 70.2%. Of 324 patients, 229 (70.7%) experienced dose reductions or interruptions; infections were the predominant cause (61.9%). The full-dose (FD) group (n&#xa0;=&#xa0;90) demonstrated superior 4-year PFS (93% vs. 58%, p&#xa0;<&#xa0;.001) and OS (98% vs. 76%, p =&#xa0;.007). Early modifications (0-3 months) were independent predictors of inferior PFS (hazard ratio [HR], 3.93, p =&#xa0;.008) and OS (HR,&#xa0;3.29, p =&#xa0;.014). Prolonged DI (>14 days) was associated with inferior PFS (HR,&#xa0;2.64) and OS (HR,&#xa0;2.15), whereas DR and short DI (&#x2264;14 days) had negligible impact. Early (0-3 months) prolonged DI was devastating&#xa0;(3-year PFS, 41.2%; HR,&#xa0;3.84, p&#xa0;<&#xa0;.001). cBTKi switching (n&#xa0;=&#xa0;82; 100% nonprogression-driven) shortened DI (median, 6 vs. 14 days) and was independently associated with superior OS (HR,&#xa0;0.34, p =&#xa0;.018) and PFS (HR,&#xa0;0.36, p =&#xa0;.022). CONCLUSIONS: Early prolonged DI is the dominant adverse prognostic factor in cBTKi-treated CLL/SLL. Proactive switching minimizes treatment gaps and improves survival, supporting a timing-aware, DI- versus DR-informed approach to dose management.

Humans