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PubMed · 10070550

Daylight simulators and colour vision tests.

Abstract

Our previous studies have shown that the influence of the illuminating source used for colour vision examination on the intended function of the test is very test dependent, some being relatively unaffected by source characteristics while others can be significantly affected. The effects can even differ between the plates of one pseudo-isochromatic plate test set. In addition, in previous studies we have considered a large range of sources on one test at a time. For this study the emphasis is on four fluorescent tube sources, all of which meet the requirements of the CIE method for assessing the quality of daylight simulators. The colour vision tests include sorting tests (the D-15 tests [Standard, Lanthony Desat and Adams Desat]), the Lanthony New Colour Test and the FM100 Hue Test, pseudo-isochromatic plate tests (Ishihara, Standard Pseudo-isochromatic Plates Volume 1, Hahn New Colour Test and Lanthony Tritan Album) and a matching test (TCU Test). Two examples are quoted here. The tests were assessed on the basis of alignment of the colours of the tests to protanopic and deuteranopic confusion axes and, where appropriate, to the tritanopic confusion axis. The data and analysis indicate that the four fluorescent tubes are essentially equivalent.

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BibTeXRIS

S J Dain. 1998. Daylight simulators and colour vision tests.. https://pubmed.ncbi.nlm.nih.gov/10070550/

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Diagnosing protan heterozygosity using the Medmont C-100 colour vision test.

BACKGROUND: A surprisingly high 15 per cent of women in Caucasian societies are carriers of the genes for abnormal colour vision but there is no clinical method to identify them. It has long been known that heterozygotes for the protan colour vision deficiencies can demonstrate a reduced luminous sensitivity to red light. This is known as Schmidt's sign, which is thought to arise from mosaicism (Lyonisation). The Medmont C-100 colour vision test measures relative spectral sensitivity using flicker photometry to differentiate protans and deutans. It should be able to diagnose Schmidt's sign. METHOD: We tested six known protan heterozygotes (four whose sons have a protan colour vision deficiency and two whose fathers are protan) with the Medmont C-100 test. RESULTS: All six heterozygotes made average settings of -1.75 or more negative at the Medmont C-100 test, settings which are at or beyond the boundary of the distribution of settings made by observers with normal colour vision. There have been two previous cases reported in the literature of protan heterozygotes, who made protan settings on the Medmont C-100 or its predecessor test, the OSCAR. We also tested six daughters of the known heterozygotes, 50 per cent of whom are likely to be heterozygotes. Four of the six (66 per cent) made protan settings on the Medmont C-100. The other two made normal 0.0 settings. CONCLUSION: We conclude that the Medmont C-100 can be used clinically to diagnose carriers of protan colour vision deficiency.

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