PubMed Health⌕ Search
Home/Archive/PubMed

PubMed · 2413535

[Post-traumatic deficit syndromes].

G Assal
View original source ↗Export metadata ↓

Abstract

We discuss the evolution of psychological disorders following head injury. Even when the trauma seems mild at first, it may then give rise to cerebral lesions, particularly in the brain stem. Cerebral contusions in closed head injuries are responsible for consciousness disorders of varying duration, which are followed by mental confusion. At this point akinetic mutism or a Kluver and Bucy syndrome may appear. We consider memory disorders and other cognitive ones, the focus being on psychiatric disturbances of various kind.

Explore related subjects

Akinetic MutismAphasiaBrain ConcussionBrain Damage, ChronicBrain InjuriesBrain StemCerebral CortexConfusionDiagnosis, DifferentialDominance, CerebralHumansNeurocognitive DisordersNeuropsychological TestsEnglish AbstractJournal Article

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

G Assal. 1985. [Post-traumatic deficit syndromes].. https://pubmed.ncbi.nlm.nih.gov/2413535/

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

PubMed2006

Abulia following penetrating brain injury during endoscopic sinus surgery with disruption of the anterior cingulate circuit: case report.

BACKGROUND: It is common knowledge that the frontal lobes mediate complex human behavior and that damage to these regions can cause executive dysfunction, apathy, disinhibition and personality changes. However, it is less well known that subcortical structures such as the caudate and thalamus are part of functionally segregated fronto-subcortical circuits, that can also alter behavior after injury. CASE PRESENTATION We present a 57 year old woman who suffered penetrating brain injury during endoscopic sinus surgery causing right basal ganglia injury which resulted in an abulic syndrome. CONCLUSION: Abulia does not result solely from cortical injury but can occur after disruption anywhere in the anterior cingulate circuit--in the case of our patient, most prominently at the right caudate.

Akinetic Mutism↗
PubMed2006

[MM1 variant of sporadic Creutzfeldt-Jakob disease with long duration akinetic mutism state].

INTRODUCTION: When patients present with a characteristic clinical picture of Creutzfeldt-Jakob disease (CJD) associated with positive 14-3-3 assay, periodic sharp wave complexes, high-signal of the striatum on magnetic resonance imaging, and homozygosis methionine (M) in codon 129, the median survival is 4 to 6 months. CLINICAL CASE: We report a 58-year-old woman with these typical features who survived 21 months, 19 of them in an akinetic mutism state. The autopsy confirmed the diagnosis of the most common CJD phenotype (MM1), usually associated with a shorter survival, and demyleinitation of the white matter (panencephalopathic form). CONCLUSIONS: The MM1 variant of CJD, with a rapidly progressive course leading into an akinetic mutism shortly after disease onset can be followed by a long akinetic mutism state. This profile is suggestive of panencephalopathic form and should be taken into account when counselling about survival.

Akinetic Mutism↗