PubMed · 5120997
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A Hustin. 1971. [Noise].. https://pubmed.ncbi.nlm.nih.gov/5120997/
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Noise-induced hearing loss has previously been reported among rock musicians. This study sought to determine whether a hazard of noise-induced hearing loss exists for music club employees other than musicians themselves. Sound levels at eight live-music clubs were measured and symptoms of noise exposure in 31 music club employees were assessed by questionnaire. The average sound level at the various clubs during performances ranged from 94.9 to 106.7 dBA. The overall sound level average, including both performance and ambient levels, ranged from 91.9 to 99.8 dBA. Symptoms of noise exposure, such as tinnitus and subjective hearing loss, correlated with sound intensity. Only 16% reported regular use of hearing protection. We conclude that employees of music clubs are at substantial risk of developing Noise-Induced Hearing Loss, due to chronic noise exposure which consistently exceeded safe levels. Hearing protection is used much too infrequently. The development of hearing conservation programs for this large group of workers is essential.
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Histological and functional derangement of the vestibular system has been reported in laboratory animals exposed to high levels of noise. However, reports of clinical series give contradictory results with regard to vestibular disturbances in industrial workers and military personnel suffering from noise-induced hearing loss (NIHL). This study evaluates vestibular function in 22 men with documented NIHL and in 21 matched controls, using electro-nystagmography (ENG) and the smooth harmonic acceleration (SHA) test. There was a significantly lower vestibulo-ocular reflex gain (p = 0.05), and a tendency towards decreased caloric response in the patients. There were no differences between patient and control groups in: incidence of vertigo and of spontaneous, positional and positioning nystagmus; directional preponderance and canal paresis in the ENG; or in phase and asymmetry parameters in the SHA test. These results demonstrated a symmetrical, centrally compensated decrease in vestibular end-organ response associated with symmetrical hearing loss in the patients. Statistically significant correlations were found between average hearing loss, and decrease in average vestibulo-ocular reflex gain (p = 0.01) and ENG caloric lateralization (p = 0.02). These correlations might indicate a single mechanism for both cochlear and vestibular NIHL. The results imply subclinical, well compensated malfunction of the vestibular system associated with NIHL.