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PubMed · 6184766

Krypton red laser photocoagulation for subretinal neovascularization.

Abstract

A series of 75 well-defined, consecutive patients with subretinal neovascularization secondary to a variety of macular diseases were treated with krypton red laser (KRL) photocoagulation and are reported on prospectively. Two thirds of the patients had subretinal neovascularization secondary to involutional (senile) macular degeneration. Most patients had clinical and/or fluorescein angiographic manifestations of neovascularization that posed a poor visual prognosis. The technique of treatment using the krypton red wavelength, as well as special considerations in the pre- and post-operative management, are reviewed. The results suggest that this wavelength is effective in the successful, anatomic elimination of subretinal neovascularization and improvement of the visual status with certain potential advantages compared to conventional argon blue-green laser (ABGL) radiation. Since this clinical study is void of randomization and controls, no absolute conclusions can be made with regard to the effectiveness of this form of treatment compared to other laser treatments or to the natural course of the macular diseases themselves. However, the results of this study are extremely encouraging, warranting further investigation.

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BibTeXRIS

L A Yannuzzi. 1982. Krypton red laser photocoagulation for subretinal neovascularization.. https://doi.org/10.1097/00006982-198200210-00004

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Horseshoe-like macular tear following recurrent branch retinal vein occlusion.

BACKGROUND: A macular hole can develop as a late complication secondary to a branch retinal vein occlusion (BRVO). We report about an atypical horseshoe-like tear occurring in the fovea after recurrent BRVO. METHODS: An interventional case report. RESULTS: In 1997, a 53-year-old man was seen with an occlusion of macular part of inferior temporal vein of the retina on the left eye. After experiencing several recurrent BRVO in this eye, 6 years later he presented with a horseshoe-like tear in the fovea. Visual acuity was 20/200. The patient underwent standard three-port vitrectomy and installation of C3F8 16%. Intraoperatively, massive traction of the vitreous was detected on the edges of the tear. Six months after the operation, the tear remained attached. The visual acuity was 20/200. CONCLUSIONS: The uniqueness of the presented case is the occurrence of a macular tear following recurrent BRVO, its horseshoe-like shape and foveal location. To the best of our knowledge, this is the first report on a horseshoe-like tear seen in the fovea secondary to BRVO. We assume that chronic macular edema and retinal ischemia following BRVO were additional factors beside the vitreous traction, contributing to the formation of the macular tear. Anatomical closure of the tear and stabilisation of visual acuity can be achieved by vitreoretinal surgery.

Fluorescein Angiography↗