Metabolic abnormalities in diabetic peripheral nerve: relation to impaired function.

D A Greene

doi:10.1016/s0026-0495(83)80024-9

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Metabolic abnormalities in diabetic peripheral nerve: relation to impaired function.

D A Greene

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Abstract

An hypothesis is presented relating several well-defined metabolic abnormalities in diabetic peripheral nerve to impaired peripheral nerve function by a sodium-potassium ATPase mechanism. It is proposed that this hypothesis be tested in the most well-defined animal model for human insulin deficiency diabetes currently available--the BB diabetic rat.

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Animals Diabetes Mellitus, Experimental Diabetes Mellitus, Type 1 Energy Metabolism Female Glucose Hyperglycemia Inositol Male Peripheral Nerves Phosphocreatine Rats Rats, Inbred Strains Sodium-Potassium-Exchanging ATPase Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S.

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D A Greene. 1983. Metabolic abnormalities in diabetic peripheral nerve: relation to impaired function.. https://doi.org/10.1016/s0026-0495(83)80024-9

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