PubMed HealthSearch

PubMed · 6400330

Cardiovascular profiling.

Abstract

As in all other medical evaluations, the evaluation of the cardiovascular system in the competitive athlete or the nonathlete wishing to exercise must begin with a carefully performed history and physical examination. Subsequent studies, including chest radiograph and electrocardiogram, may be waived in certain populations of young athletes, but in older individuals wishing to exercise, these should be included and an exercise stress test should also be done to assess the ability of the heart to withstand moderate levels of exercise without ischemia. The mainstay of evaluation of exercise performance is the graded exercise test on either a bicycle ergometer or a treadmill. These protocols are designed to provide increments in workload until the subject reaches maximal oxygen consumption or is symptom limited. Heart rate monitoring and blood pressure monitoring often are adequate to determine individual response to exercise and to provide appropriate exercise advice. When an absolute measure of physical fitness is required, maximal oxygen consumption should be measured. Following the guidelines of thorough medical evaluation plus stress testing, it is unlikely that an athlete or the exercising nonathlete will develop serious cardiovascular problems with exercise.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

R W Squires, A A Bove. 1984. Cardiovascular profiling.. https://pubmed.ncbi.nlm.nih.gov/6400330/

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

Congestive heart failure due to hypertensive ventricular diastolic dysfunction.

Left ventricular (LV) diastolic dysfunction is the first discernible manifestation of heart disease in hypertensive patients. Arterial hypertension with LV hypertrophy leads to reduced preload followed by impaired cardiac output (systolic dysfunction stemming from primary diastolic dysfunction). Diastolic dysfunction leads more often than systolic dysfunction to hypertensive heart failure and is in many cases clearly distinguishable from heart failure with low ejection fraction (EF). Mortality due to heart failure from impaired inotropism is higher than mortality due to diastolic dysfunction, but morbidity is lower. Hypertensive cardiomyopathies can be divided into 4 ascending categories, according to the pathophysiologic and clinical impact of hypertension on the heart: Degree I: LV diastolic dysfunction with no associated LV hypertrophy Degree II: LV diastolic dysfunction with echocardiographic LV hypertrophy Degree IIA: Normal exercise capacity in terms of maximal oxygen consumption Degree IIB: Impaired exercise capacity in terms of maximal oxygen consumption Degree III: Congestive heart failure (severe dyspnea and radiographically determined pulmonary edema with normal (> or = 50%) EF Degree IIIA: LV mass/volume ratio > 1.8 with little or no myocardial ischemia Degree IIIB: LV mass/volume ratio < 1.8 with significant myocardial ischemia Degree IV: Profile of dilated cardiomyopathy; LV hypertrophy and impaired EF (< 50%).

Cardiac Output