PubMed Health⌕ Search
Home/Archive/PubMed

PubMed · 8554404

[Emphysematous pyelonephritis].

J Monllor Gisbert·F Galbis Palau
View original source ↗Export metadata ↓

Abstract

OBJECTIVES: The etiopathogenic and therapeutic aspects of emphysematous pyelonephritis, an uncommon disease entity, are discussed. METHODS/RESULTS: We describe the clinical history of a diabetic with intercurrent gas-forming infection in the form of emphysematous pyelonephritis that was successfully treated. CONCLUSIONS: This disease entity, which is almost exclusive to diabetic patients, is unique. It produces a type of organism that warrants early and aggressive treatment.

Explore related subjects

Chronic DiseaseDiabetes Mellitus, Type 2EmphysemaFemaleHumansMiddle AgedNephrectomyProteus InfectionsProteus mirabilisPyelonephritisCase ReportsJournal ArticleReview

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

J Monllor Gisbert, F Galbis Palau. 1995. [Emphysematous pyelonephritis].. https://pubmed.ncbi.nlm.nih.gov/8554404/

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

PubMed2001

Genome-wide expression analysis reveals dysregulation of myelination-related genes in chronic schizophrenia.

Neuropathological and brain imaging studies suggest that schizophrenia may result from neurodevelopmental defects. Cytoarchitectural studies indicate cellular abnormalities suggestive of a disruption in neuronal connectivity in schizophrenia, particularly in the dorsolateral prefrontal cortex. Yet, the molecular mechanisms underlying these findings remain unclear. To identify molecular substrates associated with schizophrenia, DNA microarray analysis was used to assay gene expression levels in postmortem dorsolateral prefrontal cortex of schizophrenic and control patients. Genes determined to have altered expression levels in schizophrenics relative to controls are involved in a number of biological processes, including synaptic plasticity, neuronal development, neurotransmission, and signal transduction. Most notable was the differential expression of myelination-related genes suggesting a disruption in oligodendrocyte function in schizophrenia.

Chronic Disease↗