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At least 181 records · Page 10Linked to original sources
PubMed1990

[Treatment of an infected inguinal wound following vascular reconstruction with a sartorius muscle flap].

Infection or breakdown of a wound in the groin is a dreaded complication in vascular surgery. On 8 patients with such impending or declared wound complications, a distally pedicaled sartorius muscle-flap was performed immediately after wound debridement, to cover the autologeous or alloplastic vascular graft. In all but one case the groin healed promptly. In 6 cases the vascular repair remained patent.

Blood Vessel Prosthesis↗
PubMed1979

[Morphometric and histochemical characteristics of the frog sartorius muscle in ontogeny].

Morphometric and histochemical investigation of musculus sartorius was performed in ontogenesis in Rana ridibunda and Rana temporaria. Muscular composition was characterized according to the type of muscular fibres. Spectrum of lactatdehydrogenase isoenzymes was studied at different developmental stages. As the studies demonstrated, musculus sartorius underwent some essential changes in ontogenesis which manifested themselves in increasing number of muscular fibres and their areas, in changing LDG isoenzymic spectrum. Differentiation of the muscular fibres three types takes place at the 30th stage after P. V. Terentiev and depends on the nerve system maturation.

Acetylcholinesterase↗
PubMed1981

Effect of adenosine on oxygen uptake and electrolyte content of frog sartorius muscle.

Adenosine and ATP depressed resting O2 uptake in frog sartorius. This action was blocked by low levels of caffeine and by 8-phenyltheophylline. It was mimicked by a polymer of adenosine. Adenosine also decreased radioactive calcium uptake in muscles. Potassium and magnesium content were increased while sodium and calcium content were decreased by adenosine. Adenosine did not decrease oxygen uptake in muscles from frogs sacrificed in winter months. However, adenosine deaminase, which inactivates adenosine by removing the amino group, increased oxygen uptake, calcium content and lactate release of these muscles. Our results suggest that adenosine reduces resting metabolism, possibly through reducing passive leaks of electrolytes.

Adenosine↗
PubMed1980

[Differentiation of the sartorius muscle during frog ontogeny].

Stages of morphological differentiation of muscular fibres in the musculus sartorius were studied at light optic and electron microscopic levels on tadpoles and mature frogs (Rana ridibunda). Three stages were detected in the development of the muscle--stage of mature myotubes, stage of young and mature fibres. At the 29th stage of development, the sarcomere length is statistically greater, at subsequent stages it is smaller and does not practically differ from that of mature animals. The T-tubes thickness and the A-discs width in the tadpole muscular fibres at different stages of development and those of mature animals do not differ statistically. Muscular differentiation and acquirement of features peculiar for mature animals continue after metamorphosis is completed.

Animals↗
PubMed1981

Elucidation of the mechanism and site of action of quinuclidinyl benzilate (QNB) on the electrical excitability and chemosensitivity of the frog sartorius muscle.

The effects of the muscarinic antagonist quinuclidinyl benzilate (QNB) on transmission at the frog sartorius neuromuscular junction have been examined. QNB decreases endplate potential (EPP) amplitude without affecting miniature endplate (MEPP) frequency or resting potential. QNB also increased the latency of the EPP and the nerve terminal spike in a frequency dependent fashion, suggesting the site of action is the unmyelinated nerve terminal. Since the rate and amplitude of muscle action are potentials decreased it is likely that QNB causes a blockade of electrically excitable sodium channels; the agent also blocks ionic channels associated with nicotinic acetylcholine receptors. It is possible that these effects of QNB may explain some of the behavioral disturbances produced by its administration.

Animals↗
PubMed1983

[Dependence of the rate of development of Zenker's necrosis in the fibers of the frog sartorius muscle on the concentration of chloral hydrate, urea and urethane].

The spreading necrosis in dissected m. sartorius of Rana temporaria changes its speed in the phasic mode in relation to the concentrations of chloral hydrate, urea and uretan, added to the Ringer solution. The number and size of the phases are also dependent on the temperature and the initial physiological state. The concentrations of the above chemicals decelerating necrosis are 2-4 times less than those that increase the survival time of isolated muscles.

Animals↗
PubMed1985

[Ion currents of a nerve ending in the sartorius muscle of the frog].

Nerve terminal responses evoked by motor nerve, stimulation were recorded from frog sartorius nonmyelinated endings by external electrodes. At proximal parts of nerve terminal a triphasic waveform (+ - +) was observed. Ionophoretic application of tetrodotoxin, tetraethylammonium and 4-aminopyridine indicated that the negative phase reflected inward sodium current while the third positive phase--outward potassium current. Using solutions with tubocurarine from which calcium was omitted and an electrode filled with CaCl2 a late slow negative response component was recorded. This component depended on Ca2+ concentration in the electrode, increased with addition of tetraethylammonium and 4-aminopyridine and was abolished by Co2+. A similar component was recorded by microelectrodes filled with solutions containing Sr2+ and Ba2+ ions. It is concluded that the slow component of the response reflects the current across potential-dependent calcium channels in the presynaptic membrane. The time courses of calcium current and transmitter release in the synapse are compared.

Animals↗
PubMed1981

[Morpho-functional changes in the frog sartorius muscle fibers during the winter].

The electrical characteristics, sensitivity to acetylcholine, structure of nerve terminals and SDH activity were studied in the fibers of frog's m. sartorius in winter and summer. The resting MP of muscle fibers remains unchanged through the year but passive electrical characteristics of muscle electrogenic membrane were changed. Sensitivity to acetylcholine increased two-fold in winter. Silver method revealed some degenerative signs in the neuro-muscular junctions during winter.

Acetylcholine↗
PubMed1977

[Effect of maintaining the grassfrog sartorius muscle in a 0.002 M solution of alpha-dinitrophenol on its sensitivity to a 0.004 M solution of that substance].

A study was made of the effect of a preliminary maintenance of m. sartorius of Rana temporaria in 0.002 M alpha-dinitrophenol (alpha-DNP) on the resistance of muscles to 0.004 M concentration of this substance. The incubation of muscles in 0.002 M alpha-DNP for 10--20 min results in a statistically significant increase in their resistance to 0.004 alpha-DNP (24.8--30.7%). The highest increase in resistance was observed after a 20 minutes' maintenance of muscles. Therefore on studying the effect of 0.002 M alpha-DNP on the resistance of muscles to the injurious concentrations of chloral hydrate (0.08 M), ethanol (3.48 M), or 36 degrees, the muscles were maintained in the inhibitor for 20 minutes. In the case of a 20 minutes' maintenance of muscles in 0.002 M alpha-DNP, their resistance to chloral hydrate increased by 24.8%, whereas that to ethanol or heating decreased by 23.3 and 37.8--64.6%, respectively.

Animals↗
PubMed1977

[Effect of maintaining frog sartorius muscles in a number of chloral hydrate solutions on their resistance to the harmful action of this substance].

A study was made of the effect of maintenance of m. sartorius of the grass frog in several concentrations of chloral hydrate (0.0025 M, 0.005 M, 0.008 M, 0.01 M, and 0.02 M) on their resistance to 0.08 M concentration of this substance. The increase in resistance of muscles was observed after the maintenance in all the several concentrations of the agent except one (0.02 M). The increase in resistance was maximal after a 5 minute maintenance in any concentration. The optimal dose of the effect was a 5 minute maintenance in 0.005 M chloral hydrate solution. In this case the increase in resistance to 0.08 chloral hydrate was the highest (about 23%). The study of the effect of optimal doses on the resistance of muscle tissue to 0.12 M solution fluoride, 3.48 M ethyl alcohol and to 36 derees showed a statistically significant increase in resistance from 22.7 to 39.4%.

Adaptation, Physiological↗