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At least 289 records · Page 16Linked to original sources
PubMed1986

[Central pontine and extra-pontine myelinolysis following rapid correction of hyponatremia--a case report].

An autopsy case of central pontine and extrapontine myelinolysis following rapid correction of hyponatremia is reported. Hyponatremia was induced by massive devastation of adrenal cortex due to tuberculosis. Severe hyponatremia (approximately 100 mEq/l of serum sodium level) was rapidly corrected by infusion of hypertonic saline solution within two days. After transient regression of her consciousness, the patient developed decorticated state and then complete paralysis of all limbs. She died of pneumonia approximately one month after the last illness. The autopsy revealed myelynolysis in the center of the pontine basis. The nerve cells and axon cylinders were relatively well preserved. In addition, the similar lesions were seen in the subcortical gray matter and cerebral cortex. The thin white matter at the site characterized by an extensive admixture of gray and white matter was selectively involved. The pathogenesis of this disorder is briefly discussed.

Adrenal Cortex Diseases↗
PubMed1979

[Differential diagnosis of salivary gland swellings (excluding neoplasms)--the ORL view (author's transl)].

Important investigatory methods for the diagnosis of non-neoplastic salivary gland disease are discussed. The difficulties found with these different methods are emphasized, and biopsy is recommended as the most reliable means for diagnosis. Some salivary gland disorders are caused by hypotensive drugs. Chronic recurrent parotitis is presented as an example of the difficulties in differential diagnosis and treatment.

Biopsy↗
PubMedDate not supplied

[Computed tomography in the differential diagnosis of hypocorticism].

A comprehensive study of 52 patients with hypocorticism was conducted. Some of them were examined using the method of computerized tomography (CT) which provided important evidence for the diagnosis of hypocorticism, assessment of its etiology and differential diagnosis of the process. 12 of 16 patients demonstrated changes which were characteristic for hypocorticism: in 4 patients (no signs of tuberculous involvement of the adrenals) a decrease in their sizes with the preserved regular shape was noted, in 8 patients (with clinicolaboratory findings of tuberculous involvement) enlarged sizes, bilateral deformity of the contours and the presence of petrificates were detected. The authors presented some differential-diagnostic CT signs of tuberculous and metastatic involvement of the adrenals having some features in common.

Adolescent↗
PubMed1985

[Tuberculosis of the contralateral adrenal gland: a case report].

Unilateral adrenal tuberculosis is a very rare disease. A 66-year-old woman presented with epigastric discomfort and general fatigue. Abdominal CT scan revealed a homogeneous mass shadow in the right adrenal region. Findings of physical examination were normal except that the patient was obese. Hormonal data were in normal range. Adrenal scintiscanning demonstrated no RI uptake in the right adrenal gland. Right adrenalectomy was performed under the diagnosis of nonfunctioning tumor of the right adrenal gland. Histopathological examination, however, revealed typical tuberculosis with Langhans' type of giant cells and infiltrated lymphocytes. Of 322, 148 autopsies performed during the twelve years between 1970 and 1981 in Japan, 228 cases of adrenal tuberculosis were recognized. Furthermore, only 18 cases had tuberculous regions in the adrenal gland alone.

Adrenal Gland Diseases↗
PubMed1985

[Adrenal tuberculosis in 13 patients with Addison's disease. CT findings].

A series of thirteen cases of adrenal tuberculosis with Addison's disease is presented. At the onset of disease, CT findings consisted of an enlargement of both glands, with peripheral enhancement after contrast medium was injected. The CT differential diagnosis among tubercular, metastatic and amyloidotic etiology was not possible. When the course of the illness was favourable, CT showed a generalized glandular atrophy with calcifications. In two fatal cases, the diagnosis could be made only at post-mortem examination.

Addison Disease↗