Fat-deficiency disease of rats. The effect of doses of methyl arachidonate and linoleate on fat metabolism, with a note on the estimation of arachidonic acid.
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Journal Article↗
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Results for “DEFICIENCY DISEASES”
Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.
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Fat-deficiency disease of rats. The relation of the essential unsaturated acids to tumour formation in the albino rat on normal diet.
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Journal Article↗
Fat-deficiency disease of rats. The storage of fat in the fat-starved rat.
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Journal Article↗
Fat-deficiency disease of rats. The influence of tumour growth on the storage of fat and of polyunsaturated acids in the fat-starved rat.
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Journal Article↗
An explanation for variations in the clinical and biochemical symptoms of lysosomal-enzyme deficiency diseases such as GM1 gangliosidosis [proceedings].
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Alleles↗
[Metopism in iron deficiency disease. A roentgenological investigation (author's transl)].
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Adult↗
[Christmas--a light deficiency disease?].
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Brain↗
The liver in acquired immune deficiency disease.
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Acquired Immunodeficiency Syndrome↗
Anti-LW antibody production in a child with combined immune deficiency disease (thymic alymphoplasia).
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Agammaglobulinemia↗
Levels of selenium in blood and tissues associated with some selenium deficiency diseases in New Zealand sheep.
Whole bloods from weaned lambs with severe selenium responsive unthriftiness usually contain <5 ng Se/ml. Mildly or moderately affected lambs have blood levels of 5-10 ng/ml. Selenium responsive infertility in ewes appears to be associated with whole blood selenium levels below 10 ng/ml.
Journal Article↗
Serum glycoproteins in protein-calorie deficiency disease.
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Blood Protein Electrophoresis↗
Assessment of biomarker selection in selenium-deficiency disease.
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Biomarkers↗
Lymphopenic forms of congenital immunologic deficiency diseases.
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Agammaglobulinemia↗
Histiocytosis X--an immune deficiency disease? Studies on antibody-dependent monocyte-mediated cytotoxicity.
Six children with histiocytosis X, all in clinical remission, were investigated for antibody-dependent cytotoxicity mediated by monocytes and neutrophils. Monocytes demonstrated a reduced cytotoxicity and a normal Fc receptor activity. Judged by light microscopy the monocytes were normal. Preincubation with the patient's serum did not influence the cytotoxicity of normal monocytes. Neutrophils from patients with histiocytosis X showed a normal cytotoxic activity. We postulate a functional defect of the mononuclear phagocyte system in histiocytosis X.
Antibody-Dependent Cell Cytotoxicity↗
Graft versus host reaction in Wiskott-Aldrich syndrome: antemortem diagnosis of human GVH in an immunologic deficiency disease.
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Autopsy↗
Cobalt and copper deficiency in Swedish landrace pelt sheep. Application of diagnostics in flock-related deficiency diseases.
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Animals↗
Heredity counselling for sex-linked recessive deficiency diseases.
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Counseling↗
Three different hepatocyte transplantation techniques for enzyme deficiency disease and acute hepatic failure.
The effects of three different techniques of hepatocyte transplantation were investigated: transplantation of free hepatocytes into the spleen and intraperitoneal transplantation of microcarrier-attached hepatocytes or of microencapsulated hepatocytes. The liver-supportive functions of these transplanted hepatocytes were analyzed using either the Gunn rat (hyperbilirubinemia) or rats with acute liver failure. In the Gunn rat intraperitoneal transplantation of microcarrier-attached hepatocytes resulted in a significant reduction of plasma bilirubin for 28 days whereas intraperitoneal transplantation of microencapsulated hepatocytes was ineffective notwithstanding immunosuppression by cyclosporin A. Intrasplenic hepatocyte transplantation was only effective in reducing plasma bilirubin for 14 days. During acute liver failure, liver support was achieved temporarily by hepatocyte transplantation in the spleen, by intraperitoneally transplanted microcarrier-attached hepatocytes, and by microencapsulated hepatocytes to equal extents, the microencapsulated hepatocytes being the least effective after 8 h of liver ischemia.
Animals↗