SEARCH · PubMed Health

Results for “Body Temperature”

Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 343 records · Page 19Linked to original sources

PubMed1980

Optimal features of basal body temperature recordings associated with conceptional cycles.

Oral basal body temperature (BBT) recordings of 46 women that conceived by donor insemination and who had midcycle monitoring of luteinising hormone (LH) were analysed to establish features associated with an optimal cycle. All cycles exhibited a biphasic temperature shift associated with the follicular (mean + SD, 36.5 degrees C +/- 0.22) and luteal phases (36.8 degrees C +/- 0.19). Whilst a mean body temperature rise occurred on Day +1 when all cycles were analysed, individual patterns were seen at ovulation including no change or a decrease in BBT between Day 0 and Day +1. The BBT of the postovulatory phase was stable and only 4.5% of the 644 observations made showed a change of more than 0.2 degrees C from day to day. It was concluded that the BBT charting has limitations when used to recognize the day of ovulation, and that some variable patterns of the early luteal phase are consistent with conception. Finally, optimal luteal phases demonstrated remarkable stability.

Body Temperature↗

PubMed1997

[Clinical significance of peak body temperature, white blood cell count, and C-reactive protein level in febrile episodes among geriatric inpatients].

To investigate the clinical implication of peak body temperature, peripheral blood white blood cell (WBC) count, and serum C-reactive protein (CRP) level in febrile symptoms among geriatric hospitalized patients, they were analyzed in 968 febrile episodes obtained from 433 hospitalized patients in the referred hospital. Episodes of one day duration were most frequent (41.6%). WBC count was elevated over 8000/microliters in 475 episodes (49.1%) and CRP exceeded 1.0 mg/dl in 770 episodes (79.5%). Frequency of WBC elevation decreased and frequency of CRP elevation increased according to the time course. The mean value of CRP increased significantly according to the time course. The frequency of WBC count increase and CRP elevation and their averages correlated to the peak body temperature. The peak body temperature displayed the most striking correlation to the length of febrile episodes among three clinical indicators, peak body temperature, WBC count, and CRP level. These results indicate that the elevation of WBC count and/or CRP level is frequent in geriatric patients with febrile symptoms. Peak body temperature may serve as a clinical indicator of the severy of the febrile disease occurring in geriatric patients.

Aged↗

PubMed1977

Analogs of endoperoxide precursors of prostaglandins: failure to affect body temperature when injected into primary and secondary central temperature controls.

It is known that central administration of prostaglandins of the E series has marked effects on body temperature. The purpose in the present experiments was to learn whether stable analogs of the cyclic endoperoxide precursors of PGE2, PGF2alpha and PGD2, injected into the primary temperature control in the preoptic/anterior (PO/AH) hypothalamic region and into a presumed secondary control in the medulla oblongata, can produce rises in body temperature similar to those caused by PGE2. Injection of the analogs U-44069 and U-46619 (1.0 and 2.0 microng) into the PO/AH region of the rat, where both PGE2 and PGE1 caused hyperthermia, had no effect on Tre. Likewise, injections into the medulla oblongata, in the region where PGE2 and PGE1 caused hypothermia, were ineffective in altering body temperature. That neurons important to the control of body temperature are selectively sensitive to PGE2 and not to analogs of prostaglandin precursors suggests that local cyclic endoperoxides can influence body temperature only through bioconversion to prostaglandin.

Animals↗

PubMed1997

Circadian rhythm estimation by core body temperature filtered with simultaneously recorded physiological data.

In field measurements, monitoring of core body temperature is influenced by physical activities; therefore, the estimation of circadian rhythm from the data may not be exact. The purpose of this study is to design a core body temperature filter in order to reduce artifacts induced by physical activities using simultaneously recorded physiological data such as heart rate data. The effects of physical activities on core body temperature and heart rate are assessed through three experiments. Based on the above knowledge, a core body temperature filter was designed. The filter removes part of rectal temperature data as artifact when heart rate rises above a predetermined threshold. As a result, most of the spike-like noise was removed and the filtered temperature data showed sinusoidal variation more than the unfiltered data. The mesor of the estimated rhythm significantly decreased. This filtering method can provide more precise information about circadian rhythm, especially in field measurements.

Adult↗

PubMed2003

Evaluation of chemical dot thermometers for measuring body temperature of orally intubated patients.

BACKGROUND: Recent research indicates that oral measurement of body temperature is a reliable option in orally intubated patients. In situations such as protective isolation, where dedicated electronic thermometers are not available, are single-use chemical dot thermometers an acceptable alternative? OBJECTIVE: To determine the accuracy of single-use chemical dot thermometers in orally intubated adult patients. METHODS: Subjects included a convenience sample of 85 adult patients admitted to 1 of 2 intensive care units (surgical trauma and neuroscience). For each patient, oral temperatures were measured concurrently (within 5 minutes) with a chemical dot thermometer and an electronic thermometer. The sequence of temperature measurements was alternated with each subsequent patient. Both thermometers were placed in the same posterior sublingual pocket opposite the side of the endotracheal tube. RESULTS: Measurements obtained with electronic and single-use chemical dot thermometers correlated strongly (r = 0.937). With the chemical dot thermometer, body temperature was overestimated in 11.8% of the measurements and underestimated in 10.8% of the measurements by 0.4 degree C or more. The difference between oral temperatures measured with the 2 different thermometers was not related to the patient's age, sex, or sublingual pocket location or to the order of thermometer use. CONCLUSION: The chemical dot thermometer is useful and reliable for measuring body temperature of orally intubated patients. When measurements of body temperature have important consequences for decisions about treatment, clinicians should use an electronic thermometer to confirm measurements made with a chemical dot thermometer.

Adolescent↗

PubMedDate not supplied

Effects of reduced regional or body temperatures on responses of pial arterioles.

We have investigated the effects of a reduced regional or a reduced body temperature, on pial arteriolar response to a variety of vasoactive stimuli. This has been done by studying the effects of these stimuli on groups of mice with different body temperatures, or with different temperatures of the artificial cerebrospinal fluid (CSF) irrigating the pial surface. Responses in mice with body temperatures of 30 degrees or 22 degrees C showed little or no difference from responses in mice with body temperatures of 37 degrees. This was so whether the surface irrigant was maintained at 37 degrees or at 23 degrees. On the other hand, significant reductions in pial vascular responses were observed when mice with "CSF" temperatures of 22 degrees were compared with those having "CSF" temperatures of 37 degrees. The data suggest that regional cooling is more effective than cooling the body in reducing the response of pial arterioles. The data also indicate that marked reductions in body temperature would have to occur before a detectable effect on pial vascular responses is produced, at least in mice anesthetized with urethane.

Animals↗

PubMed1998

The relationships of body temperature to weight gain, feed consumption, and feed utilization in broilers under heat stress.

Commercial broiler males were used to evaluate body temperature response to heat stress and its relationship to gain, feed consumption, and feed conversion ratio. Individual gain, feed consumption, and feed conversion ratio were obtained from 28 to 49 d in a heat stress environment (32 C) and compared to a 21 C environment. Body temperatures were measured at 28, 35, and 42 d, then daily from 43 to 48 d, and correlated with 28 to 49 d gain, feed consumption, and feed conversion ratio. Feather cover was measured at 51 d of age as a percentage of BW. Initial 28-d body temperature was not significantly different between 21 and 32 C. Body temperature was significantly higher in the 32 C environment after 7 d of heat stress and was significantly higher than at 21 C at all points through 21 d of heat stress, with mean differences ranging from 0.5 to 1.0 C. Feather weight as a percentage of BW was significantly greater in the 21 C environment (6.22%) than the 32 C environment (5.03%). Mortality was not affected by the heat stress environment. Correlations between body temperature and gain, feed consumption, and feed conversion ratio were low and nonsignificant for most periods in the 21 C environment. In the 32 C environment, body temperature was significantly correlated with gain, feed consumption, and feed conversion ratio after 7 d of heat stress, with mean correlations of -0.40 for gain, -0.31 for feed consumption, and 0.24 for feed conversion ratio in Replicate 1 and -0.44 for gain, -0.40 for feed consumption, and 0.43 for feed conversion ratio in Replicate 2.

Aging↗

PubMed1992

Short-term memory, alertness and performance: a reappraisal of their relationship to body temperature.

Previous studies have led to the beliefs: (1) that short-term memory is best during the night when the body temperature is at its nadir, and (2) that the circadian rhythms of short-term memory and subjective alertness are driven by oscillators independent from each other and from the body temperature cycle. Unfortunately, these conclusions, which would have major implications for understanding the organization of the human circadian timing system, are largely based on field and laboratory studies, which in many cases sampled data infrequently and/or limited data collection to normal waking hours. In order to investigate these points further, we have monitored behavioural variables in two different protocols under controlled laboratory conditions: (1) during a period of 36-60 h of sustained wakefulness; and (2) during forced desynchrony between the body temperature and sleep/wake cycles, allowing testing of non-sleep-deprived subjects at all circadian phases. Contrary to earlier findings, we report here that the circadian rhythm of short-term memory varies in parallel with the circadian rhythms of subjective alertness, calculation performance, and core body temperature under both these experimental conditions. These results challenge the notion that short-term memory is inversely linked to the body temperature cycle and suggest that the human circadian pacemaker, which drives the body temperature cycle, is the primary determinant of endogenous circadian variations in subjective alertness and calculation performance as well as in the immediate recall of meaningful material.

Adolescent↗

PubMed1993

Patients who develop postanesthesia shaking increase body temperature at the same rate as those who do not develop shaking.

Postanesthesia shaking is a common complication occurring after general anesthesia. Although the cause is unknown, some nurses believe that shaking is useful and beneficial for patients because it increases body temperature and that shaking stops when patients are no longer hypothermic. The primary purpose of this study was to examine changes in body temperature among patients who developed and who did not develop shaking. Secondary purposes were to examine changes in body temperature among patients who received or who did not receive intravenous narcotic-analgesic medication treatment to stop shaking and among those who either stopped shaking or who continued to shake until spontaneous cessation occurred minutes to hours later. The convenience sample consisted of 36 shaking patients and 56 nonshaking patients who were extubated, were over 18 years of age, had an intact intravenous line, and received isoflurane anesthesia. Shaking was established when patients reached and sustained grade 2 or 3 shaking on a 0-to-3 visual scale for 3 minutes (no shaking to forceful shaking). Shaking was determined as having stopped when patients achieved grade 2 on a 0-to-2 visual scale (continues shaking to stopped shaking). Axillary temperature was measured on PACU admission and again after 60 minutes. The six hypotheses were supported. Patients who developed shaking (mean = .67 degrees C [1.2 degrees F]) changed body temperature at the same rate as those who did not develop shaking (mean = .72 degrees C [1.3 degrees F]). Administration of intravenous narcotic-analgesic medication to stop shaking did not alter the rate of body temperature change in patients who received or who did not receive treatment for shaking and those who did or who did not stop shaking. Findings suggest that postanesthesia shaking does not increase body temperature. This finding does not support the long-held belief that shaking is useful and beneficial for patients because it increases body temperature. Findings also indicated that the administration of intravenous narcotic-analgesic medications to stop shaking does not alter the rate of body temperature change. Suggestions for further research focus on systematically examining nursing interventions currently implemented to stop shaking. Research findings will provide evidence that either supports or fails to support the implementation of therapeutic interventions that effectively stop shaking within 5 minutes.

Adult↗

PubMed2004

Elevated body temperature independently contributes to increased length of stay in neurologic intensive care unit patients.

OBJECTIVE: Elevated temperature results in worse outcome in experimental models of cerebral ischemia and brain trauma. In critically ill neurologic and neurosurgical patients, elevated body temperature is common and is associated with neurologic deterioration and poor outcome. We sought to determine whether, after controlling for age, severity of illness, and complications, elevated body temperature remained an important predictor of intensive care unit (ICU) and hospital length of stay, mortality rate, and hospital disposition in a large cohort of patients emergently admitted to a neurologic ICU. DESIGN: Prospectively collected data (demographics, diagnosis, Acute Physiology and Chronic Health Evaluation II score, Glasgow Coma Scale score, daily maximum temperature, complications, disposition) were retrospectively reviewed. SETTING: A 20-bed neurology/neurosurgery ICU in a tertiary care academic, level I trauma, referral center. SUBJECTS: From 6,759 admissions, those admitted after an elective procedure with length of stay < or = 1 day, those <18 yrs old, and those with incomplete data were excluded, leaving 4,295 patients for this analysis. First, a hierarchical multiple regression analysis was performed to determine whether elevated body temperature was an independent predictor of length of stay. Second, a path analysis was performed to define the relationships among elevated body temperature, complications, and length of stay. Finally, a matched, weighted sample was developed to quantify the difference in length of stay. INTERVENTIONS: None. MEASUREMENTS AND MAIN RESULTS: We measured ICU and hospital length of stay, mortality rate, and discharge disposition. The presence of elevated body temperature was associated with a dose-dependent longer ICU and hospital length of stay, higher mortality rate, and worse hospital disposition. The most important predictor of ICU length of stay was the number of complications (beta =.681) followed by elevated body temperature (beta =.143). In the matched, weighted population, the presence of elevated body temperature was associated with 3.2 additional ICU days and 4.3 additional hospital days. CONCLUSION: In a large cohort of neurologic ICU patients, after we controlled for severity of illness, diagnosis, age, and complications, elevated body temperature was independently associated with a longer ICU and hospital length of stay, higher mortality rate, and worse outcome.

APACHE↗

PubMed1981

Body temperature, oviposition, and food intake in the hen during continuous light.

Deep-body temperature of the laying hen was measured with an implantable radio transmitter. Food and water intake and oviposition time were recorded, remotely, based on electric signals from microswitches attached to the individual cages. Body temperature and feeding activity of the laying hen under continuous light displayed a circadian rhythm. The length of the body-temperature cycle was 25.2 h, and the feeding cycle was 25.3 h. These values appear to be associated with the animal's laying cycle which was 25.3 h. The laying cycle of the hen can play an important role in the maintenance of circadian rhythms of body temperature and feeding activity. Both the marked temperature rise found at the time of oviposition and the prelaying (or nesting) behavior may be controlled by a preovulatory surge of luteinizing hormone (LH) and progesterone.

Animals↗

PubMed2004

Mean body temperature does not modulate eccrine sweat rate during upright tilt.

Conflicting reports exist about the role of baroreflexes in efferent control of eccrine sweat rate. These conflicting reports may be due to differing mean body temperatures between studies. The purpose of this project was to test the hypothesis that mean body temperature modulates the effect of head-up tilt on sweat rate and skin sympathetic nerve activity (SSNA). To address this question, mean body temperature (0.9.internal temperature + 0.1.mean skin temperature), SSNA (microneurography of peroneal nerve, n = 8), and sweat rate (from an area innervated by the peroneal nerve and from two forearm sites, one perfused with neostigmine to augment sweating at lower mean body temperatures and the second with the vehicle, n = 12) were measured in 13 subjects during multiple 30 degrees head-up tilts during whole body heating. At the end of the heat stress, mean body temperature (36.8 +/- 0.1 to 38.0 +/- 0.1 degrees C) and sweat rate at all sites were significantly elevated. No significant correlations were observed between mean body temperature and the change in SSNA during head-up tilt (r = 0.07; P = 0.62), sweating within the innervated area (r = 0.06; P = 0.56), sweating at the neostigmine treated site (r = 0.04; P = 0.69), or sweating at the control site (r = 0.01; P = 0.94). Also, for each tilt throughout the heat stress, there were no significant differences in sweat rate (final tilt sweat rates were 0.69 +/- 0.11 and 0.68 +/- 0.11 mg.cm(-2).min(-1) within the innervated area; 1.04 +/- 0.16 and 1.06 +/- 0.16 mg.cm(-2).min(-1) at the neostigmine-treated site; and 0.85 +/- 0.15 and 0.85 +/- 0.15 mg.cm(-2).min(-1) at the control site, for supine and tilt, respectively). Hence, these data indicate that mean body temperature does not modulate eccrine sweat rate during baroreceptor unloading induced via 30 degrees head-up tilt.

Adult↗

PubMed1983

Heat stroke: an upward shift of temperature regulation set point at an elevated body temperature.

Explore the source record for details and available documents.

Adaptation, Physiological↗

PubMed2004

Comparative study on circadian rhythms of body temperature, heart rate, and locomotor activity in three species hamsters.

Circadian rhythms of body temperature, heart rate, and locomotor activity were observed in the unanesthetized and unrestrained Syrian hamsters, Djungarian hamsters and Chinese hamsters, and the differences in these biological characters among the three species of hamster were investigated. In each species, body temperature, heart rate, and locomotor activity in the dark period were higher than those in the light period. Heart rate of Chinese hamsters was higher than that of the others in both the light and dark periods. In addition, it was found that the body temperature of Djungarian hamsters decreased rapidly one time a day. These results show species differences in body temperature, heart rate and locomotor activity of Syrian, Djungarian and Chinese hamsters.

Animals↗

PubMed1994

Dark adaptation curve during body temperature decrease.

To investigate the effect of body temperature decrease which occurred along with cold exposure and menstrual cycle on visual acuity, the dark adaptation curve (DAC), heart rate (HR) and tympanic membrane temperature (Tty) were measured at thermoneutral (29 degrees C) and cold (15 degrees C) in both follicular and luteal phases in menstrual cycle. Tty at thermoneutral and cold exposure in follicular phase were significantly lower than those in luteal phase. The degree of decrease in Tty by cooling in follicular phase was not significantly larger than that in luteal phase. HR was decreased significantly by cooling in both phases, while the difference between the two phases was not significant. DAC during cold exposure was lower than that during thermoneutral condition in both phases. Furthermore, the curve obtained during cooling in follicular phase was lower than that during cooling in luteal phase. However, DAC obtained by a subject who showed an increase of Tty by cold exposure in both phases was higher than that at thermoneutral. Even in thermoneutral in luteal phase, DAC during head cooling was significantly lower than that during thermoneutral. These results suggest that the brightness sensitivity in scotopic vision might be increased by a decrease of body temperature and the light threshold during dark adaptation threshold may be related to the change of Tty.

Adult↗

PubMed1986

[Effect of phosphatidylcholine on the body temperature and activity of posterior hypothalamus neurons in animals].

The experiments on rats and rabbits have shown that exogenous phosphatidyl choline (PC) was capable of altering the body temperature and bioelectrical activity of posterior hypothalamus neurons following intravenous and intracerebroventricular administration. Intracerebroventricular PC was more effective in raising the body temperature of rats. The experiments on rabbits have demonstrated that the influence of PC (intravenous administration) on the body temperature depended on the initial body temperature. In rabbits, the changes in the impulse activity of certain non-thermosensitive posterior hypothalamus neurons induced by intracerebroventricular PC administration were found to be dependent on the initial firing rates. PC inhibited the increase in bioelectrical activity of thermosensitive neurons in posterior hypothalamus caused by the rise in the brain temperature secondary to body temperature elevation.

Animals↗

PubMed2000

Telemetric evaluation of body temperature and physical activity as predictors of mortality in a murine model of staphylococcal enterotoxic shock.

BACKGROUND AND PURPOSE: Hypothermia and death are used as experimental markers in murine models of staphylococcal enterotoxic shock. This study determined whether body temperature and physical activity, monitored telemetrically, could predict impending death and provide an earlier, more humane experimental endpoint. METHODS: The study consisted of two iterations (experiments 1 and 2) to determine reproducibility of the model. Each experiment consisted of 24 BALB/c mice surgically implanted with intra-abdominal telemetry transmitters and then injected intraperitoneally with sublethal or lethal doses of staphylococcal enterotoxin B (SEB) and/or lipopolysaccharide (LPS). Core body temperature and physical activity were continuously monitored in all mice for 10 days before, and 5 days after, injections. Additionally, in experiment 2, subcutaneous temperatures were compared with core body temperatures obtained by telemetry. RESULTS: Body temperature and physical activity were reduced in mice after administration of SEB and LPS, or LPS alone, but not SEB only. There was a significant (P < 0.05) correlation between mortality and body temperature (P = 0.0077), but not physical activity (P = 0.97). CONCLUSION: Body temperature proved to be an early indicator of mortality in this murine model of staphylococcal enterotoxic shock.

Animals↗

PubMed2004

Elevated body temperature enhances the laryngeal chemoreflex in decerebrate piglets.

Hyperthermia and reflex apnea may both contribute to sudden infant death syndrome (SIDS). Therefore, we investigated the effect of increased body temperature on the inhibition of breathing produced by water injected into the larynx, which elicits the laryngeal chemoreflex (LCR). We studied decerebrated, vagotomized, neonatal piglets aged 3-15 days. Blood pressure, end-tidal CO(2), body temperature, and phrenic nerve activity were recorded. To elicit the LCR, we infused 0.1 ml of distilled water through a polyethylene tube passed through the nose and positioned just rostral to the larynx. Three to five LCR trials were performed with the piglet at normal body temperature. The animal's core body temperature was raised by approximately 2.5 degrees C, and three to five LCR trials were performed before the animal was cooled, and three to five LCR trials were repeated. The respiratory inhibition associated with the LCR was substantially prolonged when body temperature was elevated. Thus elevated body temperature may contribute to the pathogenesis of SIDS by increasing the inhibitory effects of the LCR.

Animals↗