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Results for “HEMORRHAGE, GASTROINTESTINAL”

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PubMedDate not supplied

[The characteristics of the DIC syndrome in gastrointestinal hemorrhages].

With the purpose of assessing the part DIC syndrome has in hemorrhage, blood coagulation system was studied in patients presenting with gastrointestinal hemorrhage. Thrombin, a blood coagulation IIa factor, which is one of the chief markers of DIC syndrome, has been found in great amounts in blood in 50% of cases. But this did not lead to disseminated transformation of fibrinogen to fibrin because of the action of the inhibitor of coagulation during the stage of activation of fibrinogen by thrombin. Antithrombin III had but a minor part in these processes. It is suggested that rise in concentration of thrombin that is not accompanied by formation of disseminated fibrin might be of compensatory and adaptive significance. Another characteristic of blood coagulation system in the patients is depressed plasminolysis, while increase, against this background, in concentration of products of fibrin/fibrinogen degradation that is most common, reflects disturbance in the processes of their elimination from the bloodstream.

Blood Coagulation Tests↗
PubMed2006

[New epidemiology of acute gastrointestinal hemorrhage].

Due to progress in general medicine and especially in gastroenterology, the incidence of acute gastrointestinal hemorrhage could be expected to have decreased during the last 25 years. However, published epidemiological data cannot, in general, fulfill this hope. The interpretation of potential trends is, however, often limited by low study quality. For example, questionable bleeding sources such as erosions in the upper gastrointestinal tract or colon diverticula are often rather uncritically named the definitive causes of bleeding. However, there is clear evidence of changes in grouping of patients. After the almost complete disappearance of Helicobacter pylori in younger indigenous populations of most industrialized countries, it is mostly elderly comorbid people with additional risk factors (NSAID use, low-dose aspirin, anticoagulation) who are affected. Not unexpectedly, this group has generally experienced no change in incidence and in fact shows a deterioration of prognosis in case of acute bleeding.

Acute Disease↗
PubMed1981

Gastric sarcoid: unusual cause of upper gastrointestinal hemorrhage.

An unusual presentation of sarcoidosis was noted in a patient who developed severe upper gastrointestinal hemorrhage. The bleeding was attributed to a sarcoid ulcer of the stomach. Serial gastroscopic studies demonstrated complete resolution of the ulcer within five months on conventional antacid therapy. The only residual lesions were gastric mucosal nodularities and moderate hyperemia with granulomas on biopsy. Sarcoidosis was diagnosed by positive Kveim test and abnormal diffusing lung capacity despite a normal chest roentgenogram.

Female↗
PubMed1990

Recurrent massive upper gastrointestinal hemorrhage due to Strongyloides stercoralis infection.

A 29-yr-old black male immigrant from Africa presented with recurrent life-threatening upper gastrointestinal bleeding due to massive duodenal infection by Strongyloides stercoralis. The diagnosis was missed by repeated examinations of fresh stool specimens for ova and parasites and by an initial esophagogastroduodenoscopy. The diagnosis was made by pathologic examination of a duodenal biopsy and of a duodenal aspirate obtained at a second esophagogastroduodenoscopy. This has been previously reported as a cause of massive upper gastrointestinal hemorrhage in four cases. Patients with obscure gastrointestinal bleeding who have travelled or lived in an endemic area should have a duodenal aspiration performed at esophagogastroduodenoscopy.

Adult↗
PubMedDate not supplied

Primary choriocarcinoma of the stomach presenting as gastrointestinal hemorrhage: report of a case.

A case of primary choriocarcinoma of the stomach presenting as gastrointestinal hemorrhage in a 58-year-old postmenopausal woman is presented. Arteriographic features included a large hypervascular gastric mass and multiple liver masses with vascular poolings, mimicking those of cavernous hemangioma. Postcontrast CT including delayed scan showed a large central nonenhanced area with marginal peripheral contrast enhancement. Close observation of the angiographic findings revealed peripheral vascular poolings and central avascularity, which are characteristic of uterine choriocarcinoma. Human chorionic gonadotrophin (HCG) titer was significantly elevated, and extragenital choriocarcinoma of the stomach was confirmed by autopsy.

Angiography↗