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Results for “GLUCOSEPHOSPHATE DEHYDROGENASE”

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At least 37 records · Page 2Linked to original sources
PubMed1977

[Histochemistry of glucosephosphate dehydrogenase and lactate dehydrogenase of the neural cells of chick embryo].

A high intensity of the reactions to lactate dehydrogenase and glucous-6-phosphate dehydrogenase was demonstrated histochemically in the neural anlage of the chick embryos after 15, 20, 24, 30, 44, 48 and 72 hours of incubation. At early stages of the development cranio-caudal gradient of enzymatic reactions was distinctly traced. A high intensity of the reactions was observed in certain cells of the neural groove in 24-hours' embryos and in the neural tube of 48-hours' embryos. These cells are supposed to develop into neuroblasts.

Animals↗
PubMedDate not supplied

[Glucosephosphate dehydrogenase activity and reduced glutathione level in the erythrocytes of children with lymphoblastic leukemia].

The activity of glucose-6-phosphate dehydrogenase and the level of reduced glutathione were determined in the erythrocytes of children with acute lymphoblastic leukaemia treated by intensive combined chemotherapy (according to the authors from Memphis). The determinations were done before, during and after completion of treatment lasting about 3 years. A significant rise was observed in the activity of this enzyme and in the level of reduced glutathione in the groups of treated children and their fall to normal values after completion of treatment.

Adolescent↗
PubMedDate not supplied

[Various genetic and clinical aspects of patients with hemolytic anemia caused by glucosephosphate dehydrogenase deficiency (DG6F)].

Five families with DG6F deficiency were studied. Out of the first family, neinatal hyperbilirubinemia was evident in 3 children. Exchange transfusion was given to the first two. Possibly, the hemolytic crisis was subsequent to the application of vitamin K. In the next family group, there is a history of 6 children dead from jaundice and deep dark urine. Partial deficiency was found in the wife and 3 children from the third family. Fortunately, the father who is the brother of a deficient patient, shows normal levels. In family number four, both the father and the mother show below normal levels of DG6F. They have a sick son and daughter with subnormal levels. In the last family, the mother carried and the father shows abnormal levels of the enzyme; consequently, a son and their 2 daughters complain from the diseases. It is pointed out that some drugs, infections and several foods may precipitate the hemolytic crisis and brief comments are made on diagnostic and therapeutic resources.

Adult↗