Industrial occupational exposure in the Taiwan Province of the Republic of China.
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The authors investigated a population group of 660 men and 440 women aged 35-60 years selected in the Skoda works and examined as part of the integrated programme of prevention of chronic non-infectious diseases. The risk profile of subjects associated with the life-style of the population was evaluated with regard to sex, age and education. It was revealed that the blood pressure rises with age and body weight in men and women. With rising body weight rises also the triglyceride and uric acid level and in women also the total cholesterol level. The authors confirmed the relationship between smoking and the thiocyanate blood level. The magnesium level declines with age in both sexes and correlates with the reported alcohol intake. Men and women with higher education smoke less, have a lower body weight and lower triglyceride blood levels. Women have also a lower total cholesterol level. University educated men consume less alcohol, while the reverse is true in university educated women.
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Miners working in the North are exposed to more pronounced effects of unfavourable occupational factors (hand drillers, bulldozer operators, etc.) and are found to have higher morbidity rates. With their longer stay in the North, the incidence of gastrointestinal disorders increases. Among the migrants from the regions with contrasting climatic conditions. (Caucasus, Central Asia), the morbidity is higher than among those from other regions of this country.
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The authors designed a method to evaluate possible influence of occupational hazards on lower life expectancy. Initial application of the method proved that average life expectancy of metallurgists is shorter by 2.6 years vs that of general male population in Tula Province.
Occupational or professional asthma is defined as adult asthma, i.e., an inflammatory respiratory disease characterized by the presence of variable airflow limitation or bronchial hyperreactivity secondary to conditions and causes associated with a given occupational or working environment - not with stimuli found outside the workplace. Depending on the physiopathological mechanism involved, a distinction is made between immune asthma (with or without IgE mediation) and non-immune asthma. It is difficult to establish the relationship among the symptoms of asthma, the patient's professional activity and the presence or absence of sensitization to certain agents in the working environment. Guided compilation of the case history and measurement of nonspecific bronchial hyperreactivity and bronchial inflammation are currently essential in the diagnostic approach to occupational asthma. Whenever possible, allergists should establish the cause-effect relationship in occupational asthma, as required by the medical-legal and social implications of the disease. Occupational asthma remains a minority diagnosis among occupational diseases in general. Adequate personnel training and the creation of diagnostic centers may help to ensure correct and rapid detection of this disease.