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At least 37 records · Page 2Linked to original sources
PubMed1995

Posttraumatic childhood lumbosacral plexus neuropathy.

A 13-month-old male received crush injury to the abdomen resulting in paraparesis due to lumbosacral plexus neuropathy. The child was monitored with serial clinical examinations and electromyography/nerve conduction studies. He had complete clinical recovery. Lumbosacral plexus neuropathy is unusual in childhood and has not been previously reported as a result of abdominal trauma. This patient is presented with details of the clinical course, electrodiagnostic studies, discussion, and literature review.

Abdominal Injuries↗
PubMed1996

[Idiopathic lumbosacral plexus neuropathy in child. Case report].

We describe a lumbosacral plexus neuropathy case in childhood in which detailed investigation, including electromyography and magnetic resonance imaging, was normal. Muscle biopsy showed mild denervation. No underlying condition was detected. The patient presented with pain, weakness and light atrophy in left lower limb, reduced reflex at the ankle, loss of the quadriceps reflex and paresthesy in involved limb. Recovery after one year was almost complete, with persistent slight weakness and atrophy.

Child↗
PubMed1985

A rare cause of lumbosacral plexus neuropathy.

This is the second report of a case in which a uterine myoma, one of the most common benign tumours of women, was the cause of a lumbosacral plexus neuropathy. The possibility of uterine myoma should be considered in the differential diagnosis of neuropathy of the lumbosacral plexus in women.

Aged↗
PubMed2002

Percutaneous arterial interventional treatment of exercise-induced neurogenic intermittent claudication due to ischaemia of the lumbosacral plexus.

Radiological interventional therapy is described in seven patients with a distinct clinical syndrome of exercise-induced neurogenic intermittent claudication due to a reversible ischaemia of the lumbosacral plexus during walking accompanied by transient neurologic deficits. This condition was presumably caused by a reversible vascular steal phenomenon during exertion. The underlying vascular conditions were stenoses of the internal and/or common iliac arteries. All patients underwent percutaneous transluminal angioplasty (PTA) during the period from 1988 to 2001; an additional stent was placed in two patients. After a mean follow-up period of 18 months, four patients were asymptomatic, two had an improvement in walking-distance of 300 m and 800 m, respectively, and one patient developed a peripheral intermittent claudication without neurological complaints. In four patients, however, a further intervention was required. In patients with intermittent claudication due to exercise induced ischaemia of the lumbosacral plexus, a successful treatment is possible by means of PTA. Repeat intervention is justified if symptoms recur.

Aged↗
PubMed1993

Idiopathic lumbosacral plexus neuropathy in two children.

Two children diagnosed as having idiopathic lumbosacral plexopathy are presented. Although an apparently rare condition, it is probably underdiagnosed since it is difficult to recognise. Before such a diagnosis, careful exclusion of compressive and infiltrative lesions of the spinal cord, cauda equina and lumbosacral plexus is of utmost importance.

Blood Chemical Analysis↗
PubMed1981

Lumbosacral plexus neuritis.

We studied four patients with neurologic disorders of the lumbosacral plexus. Except for location of symptoms, the disorder confirmed to criteria established for the clinical diagnosis of brachial plexus neuritis. Acute onset of pain in one or both legs was followed by weakness, loss of stretch reflexes, and sometimes atrophy of affected muscles. The disorder affected in individual nerves or parts of the plexus. Gradual recovery occurred in all four patients.

Adult↗
PubMed1996

[Damage to the lumbosacral plexus in psoas hitch operation].

The psoas hitch operation is used for the management of ureteral disease requiring replacement of varying lengths of the ureter. During this procedure the bladder is fixed to the psoas by 2 or 3 hitches of nonresorbable suture. Astonishingly, peripheral nerve injury to the lumbosacral plexus has not yet been described as a complication even though it has been seen when the psoas muscle was hitched unintentionally during abdominal surgery. Within 14 days we saw 2 patients with damage to parts of the lumbosacral plexus following a psoas hitch. In one case, hypesthesia and exercise-related pain in the areas supplied by the genitofemoral, the ilioinguinal and femoral nerve persisted, suggesting inclusion of at least parts of those nerves in the hitch. Initial paresis of hip flexion did not persist and the second patient recovered without sequelae. According to the literature this kind of complication does not occur if visible nerves are avoided and stitches an used that are no deeper than 3 mm into the psoas muscle.

Adult↗
PubMed1988

Lumbosacral plexus avulsion injury: clinical, myelographic and computerized tomographic features.

A rare case of lumbosacral plexus avulsion studied with computerized tomography (CT) in a 5-year old Saudi involved in a vehicular accident with resultant left lower limb monoparesis is presented. Myelography and CT were superior to electromyographic findings in this case. Such cases should be closely followed and surgical exploration and decompression offered if any evidence of cauda equina compression develops.

Child, Preschool↗
PubMed1988

[Successful corticoid treatment of lumbosacral plexus neuropathy in heroin abuse. Clinical aspects, electrophysiology, therapy and follow-up].

A 33 year old heroin addict developed a lumbosacral plexopathy which clinically and electrophysiologically resembled an "idiopathic lumbosacral plexus neuropathy". He complained of severe neuralgic pain, marked paresis, and distinct sensory loss in the right leg. The plexopathy was accompanied by rhabdomyolysis. He was treated with corticosteroids in high dosage. Within 3 days the disabling symptoms had almost completely disappeared. Clinical and electrophysiological findings are described in the course of the disease and are compared with the data in the literature.

Adult↗
PubMed1991

Ischemic injury to the spinal cord or lumbosacral plexus after aorto-iliac reconstruction.

Between January 1, 1980, and June 30, 1989, 9 patients (6 males and 3 females) developed ischemic injury to the spinal cord or lumbosacral plexus following 3,320 operations on the abdominal aorta (0.3%). The incidence of this complication was 0.1% (2 of 1,901) after elective and 1.4% (3 of 210) after emergency abdominal aortic aneurysm repair, and 0.3% (4 of 1,209) after repair for occlusive disease. Three of the latter had prior clinical evidence of distal embolization. Eight grafts were bifurcated (aorto-iliac:four, aorto-femoral: three, aorto-ilio-femoral:one). One patient underwent extra-anatomic revascularization. Only two patients had supraceliac aortic cross-clamping and one patient underwent exclusion of both internal iliac arteries. Four patients had hypotension. Early mortality was 22% (two of nine). Severe perioperative complications, mostly due to associated visceral and somatic ischemia and sepsis, were present in seven of the nine patients. The extent and type of the neurologic injury correlated with long-term outcome. Patients with ischemic injury of the lumbosacral roots or plexus had better recovery. Attention to the pelvic circulation and the collateral blood supply is important. Use of gentle technique to prevent embolization, avoidance of hypotension and prolonged supraceliac cross-clamping, revascularization of at least one internal iliac artery, and the use of heparin may decrease but not eliminate paraplegia. Once this unexpected complication occurs, careful neurologic evaluation should be done to localize the lesion and aid prognosis.

Aged↗
PubMed1999

Lumbosacral plexus in Hoxa9 knockout mice with special reference to their nerve variations identified according to whether they were interphenotypic or intergenotypic differences.

The lumbosacral plexus in specific Hox gene mutant mice originating from C57bl/60, i.e., Hoxa9 +/+, +/- and -/- genotypes (10 specimens of each), were dissected minutely and detailed examinations were made of the eight nerves in the plexus and the lowest intercostal nerve. We identified three types of nerve variation in mice with Hox mutations: interphenotypic variations, intergenotypic variations and common variations regardless of the genotype axial phenotype. The interphenotypic variations involved a caudal shift of the nerve origin in mice with the Hoxa9 -/- axial phenotype. We divided these variations into three patterns according to whether or not nerve configurations in mice with the wild and/or Hoxa9 -/- axial phenotypes were consistent, as follows: 1) the iliohypogastric and pudendal nerve morphologies of either of the phenotypes were consistent; 2) the femoral, obturator and sciatic nerve morphologies of the wild axial phenotype were consistent, but those of the -/- phenotype showed several variations and 3) both phenotypes showed several variations of the ilioinguinal and genitofemoral nerves. The intergenotypic variations, were limited to two examples: the common trunk formation of the ilioinguinal and iliohypogastric nerves in mice with the +/- and -/- genotypes, regardless of their axial phenotypes, and the territory of the ilioinguinal nerve touched the lateral margin of the thigh in the wild genotype, but extended beyond the margin onto the posterior aspect in the -/- genotype. The common variations regardless of either axial phenotype or genotype, included the lateral cutaneous nerve of the thigh which showed a limited, common variation in both of the nerve origin, ramification pattern and territory among either the three genotypes or two axial phenotypes. We consider the interphenotypic variations to be consistent with previous experimental findings showing a discrepancy between the nerve origins and their cues for nerve pathfinding. However, this developmental sequence did not seem to apply to the intergenotypic or common variations.

Animals↗
PubMed2006

Lumbosacral plexus injury and brachial plexus injury following prolonged compression.

We report the case of a 36-year-old woman who developed right upper and lower limb paralysis with sensory deficit after sedative drug overdose with prolonged immobilization. Due to the initial motor and sensory deficit pattern, brachial plexus injury or C8/T1 radiculopathy was suspected. Subsequent nerve conduction study/electromyography proved the lesion level to be brachial plexus. Painful swelling of the right buttock was suggestive of gluteal compartment syndrome. Elevation of serum creatine phosphokinase and urinary occult blood indicated rhabdomyolysis. The patient received medical treatment and rehabilitation; 2 years after the injury, her right upper and lower limb function had recovered nearly completely. As it is easy to develop complications such as muscle atrophy and joint contracture during the paralytic period of brachial plexopathy and lumbosacral plexopathy, early intervention with rehabilitation is necessary to ensure that the future limb function of the patient can be recovered. Our patient had suspected gluteal compartment syndrome that developed after prolonged compression, with the complication of concomitant lumbosacral plexus injury and brachial plexus injury, which is rarely reported in the literature. A satisfactory outcome was achieved with nonsurgical management.

Adult↗
PubMed1985

Lumbosacral plexus stretch injury following the use of the modified lithotomy position.

The modified lithotomy position is used to provide simultaneous operative exposure to the abdomen and perineum. We report 3 lumbosacral plexus complications following use of this position. A mechanism involving stretch secondary to hyperabduction seems most likely. Electromyography is helpful in the diagnosis and the prognosis seems to be good.

Adolescent↗
PubMed1998

Anatomy and evaluation of the lumbosacral plexus.

Although not as common as brachial plexopathies, lumbosacral plexopathies do occasionally occur. Most plexopathies are due to masses that compress or infiltrate the pelvis, traction on the plexus itself from dislocation of the hemipelvis, or ischemia. Diagnosis and evaluation of the lumbosacral plexus may be made using electrodiagnostic testing, MR imaging, or CT scans, in addition to the patient's history, thorough physical examination, and knowledge of the anatomy of the plexus and its associated anatomical structures.

Electrodiagnosis↗
PubMed1982

[Reconstruction of the lumbosacral plexus of an embryo at stage 23 (25mm) and an embryo at stage 16 (7,5mm)].

Two embryos, one at stage 23 and another at stage 16, are reconstructed in view of studying the evolution of the lumbosacral plexus. At stage 16, the sacral plexus begins its development with anastomosis of L5, S1 and S2 roots, while the pelvic member is scarcely sketched. At stage 23, the evolution is complete, the pelvis is entirely constitued and the femoral, sciatic and obturator nerves of adult type. The coccygian plexus is in process of construction.

Humans↗