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Results for “Pacemaker, Artificial”

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PubMedDate not supplied

Treatment of sick sinus syndrome with permanent cardiac pacemaker. (Sick sinus syndrome and artificial pacemaker).

Twenty nine patients with sick sinus syndrome were treated from November, 1972 through July, 1976. Six cases had sinus bradycardia, 14 cases had sinoatrial block/sinus arrest and 9 cases had bradycardia-tachycardia syndrome. Twenty five patients received the implantation pacemaker with good result. There was found that an adequate pacing of the heart was effective for the control of the attack of tachyarrhythmia in the case with bradycardia-tachycardia syndrome.

Adolescent↗
PubMed2003

[Electrocardiographic diagnosis of ischemic lesions of the myocardium in patients with artificial pacemaker].

137 patients with artificial cardiac pacemaker (ACP) were examined to ascertain origin and changes of the end of spontaneous ventricular complexes (SVC) in ventricular pacing. The examination program included the ACP inhibition test, K test, veloergometry (VEM), echocardiography. Intermittent pacing has revealed depression of ST segment and/or inversion of T wave of SVC in 109 (79.6%) patients. After inhibition (switch off) of ACP in spontaneous heart rhythm 45 (41.3%) patients got free of the above shifts or showed a less number of pathological ECG leads (Chaterie's syndrome). In positive K test conducted after inhibition of ACP normalization of the SVC end in additional 11.0% cases. True Chaterie's syndrome is characterized by reversibility of ST wave depression and/or inversion of T wave of SVC in long spontaneous rhythm and a positive potassium test. In ischemic damage to the myocardium or postinfarction scars, changes of the SVC end part were stable, combined in many cases with pathological changes of QRS-complex, positive VEM test. Hyperdiagnosis of microfocal myocardial infarction in intermittent ventricular pacing was observed in 78% cases, especially due to Chaterie's syndrome. Thus, analysis of ECG changes is important for the choice of a valid policy of management of patients with ACP.

Aged↗
PubMed1993

A stochastic network model of the interaction between cardiac rhythm and artificial pacemaker.

The electrical interaction between the heart and an artificial pacemaker is often complex. Because of the sophistication and diversity of dual-chamber device algorithms, even experienced cardiologists can have difficulty interpreting paced electrocardiograms (ECG's). In order to study heart-pacemaker interaction (HPI), a computer model of the cardiac conduction system has been developed which includes the effects of artificial pacemaker function and failure. The stochastic network model of cardiac conduction consists of five vertices, each representing a functional electrophysiologic element. Electrophysiologic multidimensional conditional probability functions determine the depolarization status of each vertex. The atrioventricular (AV) node is emulated using a mathematical model which includes the influence of past cycle lengths on AV nodal conduction time. Twenty-three classes of arrhythmias may be simulated and, for pacing simulation, one of 12 antibradycardia pacing modes may be chosen. Random effects of pacemaker malfunction including oversensing, undersensing, or failure-to-capture may be simulated through the use of probability distribution functions. This model should prove useful in the development of pacemaker algorithms, determining patient-specific pacemaker therapy, and predicting causes for apparent pacemaker malfunction. The model has been used in the development of an expert system to analyze paced ECG's for pacemaker function and malfunction.

Algorithms↗
PubMedDate not supplied

[The effect of obzidan on the cardiohemodynamics of subjects with an artificial pacemaker who have hypertension].

Overall thirty six persons with artificial pacemaker (AP), presenting with stage I-II hypertensive disease were examined. Effects of obzidan on cardiohemodynamics (CHD) were studied with single administration and course treatment over two weeks depending on the type of circulation. In hyperkinetic type effects of obzidan on AP and CHD were more pronounced than in eu- and hypokinetic types. But latter subjects developed clinical signs of cardiac insufficiency growing progressively worse. It was found out that cardiohemodynamic effects of obzidan in patients with AP depend upon particular features of intracardiac hemodynamics in ventricular electric stimulation (fixed cardiac rhythm, atrioventricular dissociation, pathologic asynchrony of ventricles) and initial type of circulation.

Adrenergic beta-Antagonists↗
PubMed1980

Favorable effects of oral digoxin therapy on the left ventricular performance in patients with artificial pacemakers.

The contribution of atrial systole in the left ventricular (LV) function and the effects of oral digoxin maintenance therapy on the LV function were evaluated noninvasively in patients who had artificial pacemakers but no clinical manifestation of congestive heart failure (NYHA Class I). Diastolic dimension (Dd), ejection time (ET), and stroke volume were constant in 8 patients without P waves on their electrocardiograms, but in 20 patients with P waves they were variable from beat to beat because of the variation of the PR intervals which caused them to reach their maximum values when the PR intervals were 160 to 200 msec. Ten out of the 28 patients were given 0.25 mg of digoxin daily for 10 days. Dd did not change significantly, but the ET and the systolic dimension were significantly shortened (P < 0.001). Posterior wall excursion, ejection fraction, and mean posterior wall velocity were significantly increased (P < 0.001). It is concluded that (1) atrial contraction is important to the LV function in patients with artificial pacemakers, and (2) that favorable effects on the LV function can be obtained by a small dose of digoxin administered to the patients who had artificial pacemakers but no congestive heart failure.

Adult↗