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PubMed1979

Reiter's disease: modern concepts.

Reiter's disease (RD) is a reactive arthritis syndrome in which arthritis, conjunctivitis, dermatitis, and urethritis are the main features. It may be an acute and self-limited disease or it may cause severe debilitation. It generally follows an infectious episode, such as Shigella dysentery or nonspecific urethritis. Recent evidence has shown that patients in whom RD develops have a genetic disposition since the presence of HLA-B27 correlates highly with RD. Therapy of RD involves supportive care.

Adolescent↗
PubMed1993

[The diagnostic criteria of Reiter's disease].

Previously selected criteria have been tried in the diagnosis of 36 Reiter disease and 89 rheumatic fever patients. Evaluation of the informative value, sensitivity and specificity of the signs allowed recognizing 19 of them as Reiter disease diagnostic criteria. By diagnostic significance the signs were divided into principal, adjuvant and associated. This is thought helpful in the symptoms systematization and diagnosis.

Adolescent↗
PubMed1985

Unusual manifestations of Reiter's disease in a child.

Reiter's disease in a child, manifesting as circumscribed, thick, hyperkeratotic, rupioid lesions over the skin and later in the course of the disease developing pyopericardium and cardiac failure, is reported. Recovery of Chlamydia from the urethral discharge, too, was significant.

Arthritis, Reactive↗
PubMed1988

[Vulvovaginitis circinata in Reiter's disease].

A case of Reiter's disease with circinate vulvovaginitis is presented. As a clinical analogue to circinate balanitis, it should have the same diagnostic value. A combined treatment of prednisone and aromatic retinoid proved to be effective.

Adult↗
PubMed1957

[Reiter's disease].

Explore the source record for details and available documents.

Arthritis, Reactive↗
PubMed1976

[Reiter's disease].

In the Reiter syndrome apart from the symptomatology on the part of the urogenital tract, the changes of eyes and joints a series of further diagnostically important participations of organs appear. In most cases is at the beginning an intestinal infection which, after characteristic latency periods, is followed by urethritis, arthritis and changes of the eyes. Characteristic is further an affection of skin and mucous membrane; facultatively a participation of the heart appears. Different types of course render the diagnosis difficult. The investigation for the occurrence of HL-A 27 is useful.

Adult↗
PubMed1979

Methotrexate in Reiter's disease.

Three cases of severe Reiter's disease were treated successfully with methotrexate after failure of conservative therapy, including systemic corticosteroids in 2 instances. The usefulness and potential hazards of such therapy are discussed, and some evidence that corticosteroids may aggravate the dermatological manifestations of Reiter's disease is presented.

Adolescent↗
PubMed1975

Reiter's disease in three boys.

Three cases of Reiter's disease occurring in boys under the age of 16 are reported. One of these presented with a Salmonella enteritidis diarrhoea. This conforms to the 'dysenteric' form of Reiter's disease usually seen in Europe and rarely reported in England. Another presented with a monarticular arthritis of the knee, and the third has developed a chronic relapsing erosive arthritis as a result of sexually acquired Reiter's disease--an occurrence not previously reported in this age group. We draw attention to the frequency of diarrhoea in these children and the sex incidence of 1 female to 4--5 males, which agrees more with Reiter's disease of dysenteric origin than that acquired venereally.

Adolescent↗
PubMed1995

Vasculitis as the basis of cutaneous lesions in Reiter's disease.

The cutaneous lesions of Reiter's disease (RD) and pustular psoriasis (PP) are said to be histologically similar and often indistinguishable. We encountered three cases of RD in which biopsy specimens of lesions clinically compatible with keratoderma blenorrhagicum showed a pustular psoriasiform tissue reaction in conjunction with a subjacent superficial leukocytoclastic vasculitis (LCV). In an attempt to ascertain if these changes were distinctive and unique to cutaneous RD, the incidence of such changes in lesions of PP was examined using light microscopy and immunohistochemistry. The role of chlamydial infection in the pathogenesis of the observed vascular changes also was explored by assessing for the presence or absence of vascular deposition of chlamydial antigen in cutaneous RD compared with that in a control group that included cases of LCV and PP. In addition to conventional light microscopic analysis, immunoperoxidase studies to identify immunoglobulin deposition were performed on formalin-fixed, paraffin-embedded tissue from two of three patients with RD and on skin biopsy specimens from 11 patients with PP. Direct immunofluorescence (DIF) studies with antibodies to immunoglobulin (Ig)G, IgM, IgA, C3, and chlamydial antigens were performed on frozen tissue from one patient with RD, two patients with PP, three patients with LCV, one patient with nonspecific dermatitis, and one patient with Behçet's disease, who had a high antichlamydia antibody titer. All three specimens of RD showed a pustular psoriasiform diathesis in conjunction with a subjacent superficial LCV that was of maximal intensity in the dermal papillae capillaries. Through an immunoperoxidase technique performed on formalin-fixed tissue, the RD cases for which tissue was available for study demonstrated Ig deposition in injured blood vessels; using the same technique one of 11 PP biopsy specimens showed vascular Ig deposition in concert with LCV. This patient's biopsy was from a lesion of drug-induced LCV. None of the other specimens showed either light microscopic or immunohistochemical evidence of vasculitis. In the one specimen of RD studied by DIF, vascular deposition of IgG, IgM, C3, chlamydia heat shock protein 60 (CHSP60), and chlamydia-specific lipopolysaccharide (LPS) was observed. In the two specimens of PP studied, vascular deposition of C3, fibrin, CHSP 60, and chlamydia-specific LPS was not observed. Two specimens of LCV and the one specimen of dermatitis with concomitant nonspecific vascular injury showed vascular Ig and C3 deposition; in contrast, no vascular deposition of CHSP 60 or chlamydia-specific LPS was observed.(ABSTRACT TRUNCATED AT 400 WORDS)

Adult↗
PubMedDate not supplied

Macular degeneration in a case of Reiter's disease.

Posterior uveitis is not a documented feature of Reiter's disease. We describe here a patient of Reiter's disease, who after a bout of dysentery in January 2002 developed macular edema of the right eye with visual acuity of 6/36. In December 2002, she was found to have phlyctenular conjunctivitis. In June 2004, when she reported with scaly plaques over both palms, repeat ophthalmological examination revealed macular degeneration of the right eye. The simultaneous occurrence of Reiter's disease and macular degeneration in this patient may be fortuitous but the immunological basis of both diseases suggests a possible association.

Anti-Inflammatory Agents↗
PubMed1986

The natural history of Reiter's disease--21 years of observations.

Seventeen patients with Reiter's disease who were included in an earlier prospective study, were reviewed 21 years after their initial episode. Two patients had active synovitis and this correlated with the duration of the initial episode, the extent of the initial synovitis and the peak ESR. In contrast the risk of developing ankylosing spondylitis, present in five patients, was unrelated to the duration of the initial episode or the extent of the initial peripheral synovitis but was greatest in those with a higher peak ESR. A comparison of radiological features at the onset and later showed that five patients had developed bilateral sacroiliac joint fusion although two of these had some sclerosis around the sacroiliac joints at the onset. Eight patients had multifocal marginal syndesmophytes which, in four, occurred without sacroiliac changes. Plantar spur formation and hip and shoulder disease were associated with spondylitis while destructive small joint changes were a feature of Reiter's disease itself. Ten patients were HLA B27 positive. The clinical features at onset were unrelated to the B27 type. Sacroiliitis, osteitis pubis, pelvic whiskering and vertebral squaring were only present in B27 positive patients but syndesmophyte formation was unrelated to B27 type.

Adult↗