[Cytotoxic activity of alkyl-sulfonates. II. Carcinostatic effect of 2-chloro-ethylmethylsulfonate on benzopyrene sarcoma & rat myeloma].
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Alkanesulfonates↗
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Results for “SULFONIC ACIDS/effects”
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[Structure of antitussive agents belonging to the sulfonate group].
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Alkanesulfonates↗
[Therapy of lead poisoning; experimental study with piro-na].
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Catechols↗
[Effect of various polysaccharides, sulfonic derivatives of polysaccharides & aromatic salts of sulfa-acids on the complement].
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Complement System Proteins↗
[Anticellular activity of alkylsulfonates. IV. Effects on blood cells].
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Alkanesulfonates↗
[Anticellular activity of alkylsulfonates. V. Morphological and functional study of the bone marrow].
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Alkanesulfonates↗
[Bacteriological investigations on albothyl used in leukorrhea therapy].
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Cresols↗
[Effects of atropine sulfate and sodium decylsulfonate on isolated muscle preparations].
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Atropine↗
[The growth inhibiting effect of the sulfonic acid derivatives on tubercle bacilli].
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Acids↗
Factors related to the growth of psittacosis virus (strain 6BC). IV. Certain amino acids, vitamins, and other substances.
The analogues of amino acids, beta-2-thienylalanine, ethionine, and 6-methyltryptophane, inhibited the growth of psittacosis virus (6BC) in tissue culture without evidence of serious toxicity for the host cells. Of a number of vitamin analogues tested, only salicyl-beta-alanide inhibited viral multiplication in the absence of toxic effects on the host cells. 6,7-Diethylriboflavin, desoxypyridoxine, and oxythiamine reduced viral growth in concentrations that possessed some toxicity for host tissue. In tolerated amounts, 3-acetylpyridine, pyridine-3-sulfonic acid, pantoyl sulfanilamide, and desthiobiotin did not effect viral multiplication. Sodium malonate inhibited psittacosis virus growth in non-toxic amounts, whereas sodium monofluoroacetate was ineffective. Colchicine suppressed multiplication of virus only after a prolonged period of exposure and subsequent delay before producing inhibition, suggesting that the effect was secondary to its antimitotic action which suppressed multiplication of the host cells.
Alanine↗