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PubMed1987

[A plan for the medical prevention of pneumoconiosis in coal mining].

Some assumptions of a system of pneumoconiosis prevention for coal miners have been presented. An essential element of the system consists in replacing fluororadiograms with full-size X-ray pictures, as a method of primary selection. Provisions have been also made for promotion of the detection and treatment of chronic bronchitis and some other respiratory system diseases, along with obligatory spirometric tests. It is planned that the examinations will also involve pensioners. The current analysis of the collected information will be made using the electronic data processing.

Anthracosilicosis↗
PubMed1985

[New morphological aspects of pathogenesis of pneumofibrosis in anthracosis].

The death of the alveolar macrophage stimulating the activation of fibroblast in pneumoconiosis is not a single trigger mechanism of pneumofibrosis in anthracosis. Alteration of the microcirculatory bed and lung aerohematic barrier plays an essential pathogenetic role in coal pneumoconiosis. Polycarbonic acids which are formed in the organism from coal as a result of its oxidative-hydrolytic degradation are the cause of the damage and loss of the structural components of the aerohematic barrier.

Anthracosilicosis↗
PubMed1985

Lung function in different types of pneumoconiosis.

The results of pulmonary function tests in patients with silicosis, pneumoconiosis in coal miners and arc welders, asbestosis, as well as in persons exposed to asbestos without abnormal x-ray findings of the chest, and in nonexposed healthy control persons and the development after 6 years are compared between each other and with the x-ray findings. There are significant differences between the several groups. There is no correlation (parallelism) between increase of lung function impairment and progression of x-ray findings.

Anthracosilicosis↗
PubMed1985

[Liver and blood enzyme spectra of rats with model anthracosis based on feeding a diet with additional amounts of methionine and pyridoxine].

The data obtained during studies of the enzymic spectrum of the liver and blood of rats with experimental anthracosis fed the diet containing additional quota of methionine and pyridoxine are presented. It was established that introduction of additional quota of methionine and pyridoxine in the animals' diet with an optimal fat content reduced the negative manifestations on the part of the enzymic systems of the liver and blood characteristic of experimental anthracosis and thus promoted retardation of fibrous process in the lungs. Additional introduction into the diet of rats with experimental anthracosis of methionine alone appeared ineffective.

Animals↗