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Results for “CEREBROVASCULAR CIRCULATION”

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At least 397 records · Page 22Linked to original sources

PubMed2001

[EDAS (encephalo-duro-arterio-synangiosis) for occlusive/stenotic cerebrovascular disease].

The efficacy of encephalo-duro-arterio-synangiosis (EDAS) using superficial temporal artery was evaluated for the treatment of the occlusive/stenotic cerebrovascular disease. Nine patients with the occlusive/stenotic cerebrovascular disease underwent EDAS in our hospital. The mean follow-up period was 6.6 months. Postoperative angiography showed no collateral formation via EDAS in any of the nine patients. We analyzed the following points: 1) operative procedure, 2) follow up period after surgery, 3) preoperative cerebral blood flow, and 4) age of the patients. Results showed that EDAS as a treatment of occlusive/stenotic cerebrovascular disease was not effective. This study failed to reinforce the suggestion that indirect extracranial/intracranial bypass surgery is effective as the treatment of occlusive/stenotic cerebrovascular disease.

Adult↗

PubMed1991

[Doppler ultrasonography in Takayasu syndrome].

The authors by means of ultrasound Doppler examined 26 patients with non-specific aortoarteritis involvement of the aortic arch branches. The use of compressive tests permitted accurate diagnosis of the routes of blood overflow to the affected segment of the arterial basin of the brain. It was established that the main route of blood inflow in patients with non-specific aortoarteritis was the system of intracranial anastomoses; extracranial blood supply of the brain was noted in occlusion of the internal carotid artery.

Adult↗

PubMed1991

[Long-term changes in cerebral blood flow in patients with atherothrombotic and embolic brain infarction].

Cerebral blood flow (CBF) was measured twice at years interval using xenon-133 inhalation technique in patients with atherothrombotic and embolic supratentorial brain infarction. The purpose of the study was to elucidate factors influencing long-term change in CBF in two subtypes of brain infarction of different mechanisms. Those patients were excluded from the study, who had bilateral hemispheric lesions, significant arterial lesion in the contralateral carotid axis and recurrent stroke before the second measurement of CBF. Of 46 patients studied, 23 (17 men and 6 women) were classified as atherothrombotic and 23 (15 men and 8 women) as embolic infarction based on the diagnostic criteria reported earlier. Their age at onset was 60.6 +/- 8.9 years old (mean +/- S.D.) for atherothrombotic patients, and 60.2 +/- 11.0 years old for embolic patients. The first measurement of CBF was performed between 31 and 87 days and the second measurement between 13 and 99 months after onset. In atherothrombotic group, mean hemispheric CBF (mCBF) of the affected side tended to be higher in patients examined at intervals shorter than 30 months, but tended to decrease in those at longer interval. The multivariate stepwise regression analysis indicated only "the interval between 2 measurements" to have a significant effect on reduction of mCBF, although in addition to the interval (p less than 0.05), infarct-size (p less than 0.01) and change in PaCO2 (p less than 0.05) were suggested to be possible factors by single regression analysis. In embolic group, an increase in hematocrit had a significant effect on reduction of mCBF, even when being evaluated with multivariate stepwise regression analysis.(ABSTRACT TRUNCATED AT 250 WORDS)

Aged↗

PubMed1988

Can differences in cerebral and coronary autoregulation and O2-extraction explain why antihypertensive treatment prevents stroke but not myocardial infarction?

It is proposed that the failure of current antihypertensive treatment to reduce the incidence of coronary heart disease in patients with hypertension is due to severe, possibly irreversible restriction of coronary vascular reserve in the deeper layers of the left ventricle. Contrary to the heart, the brain can maintain a normal oxidative metabolism when the blood pressure is lowered, by extracting more oxygen from the blood. The brain is thus better suited than the heart to take advantage of the beneficial effect of antihypertensive treatment in terms of protection against hypertensive organ damage. This hypothesis is supported by studies of cerebral and coronary autoregulation and the J-shaped relation between death from myocardial infarction and treated diastolic blood pressure now reported from a number of studies.

Cerebrovascular Circulation↗

PubMed1986