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[Paraplegia caused by vertebral compression fractures in senile osteoporosis].

Paraplegia followed spontaneous deformed osteoporotic spine is very rare. Our own 3 cases treated conservatively in two and operatively in one with success and 11 cases reported in the literatures were analyzed. All patients were recovered from paraplegia. One patient was died one and a half year after the recovery of her paralytic episode. Thorough autopsy study of this patient showed no organic lesion in the regarded spinal cord due to such kind of spine deformity.

Aged↗
PubMed2004

Risk factors of new compression fractures in adjacent vertebrae after percutaneous vertebroplasty.

PURPOSE: To evaluate the risk factors related to the development of new fractures in adjacent vertebrae after vertebroplasty. MATERIAL AND METHODS: The study was conducted on 106 patients in whom 212 vertebroplasties were performed during a period of 3 years. Evaluations of the five vertebrae superior and inferior to the treated vertebra were performed. Consequently, 913 vertebrae were evaluated and the fracture-free interval of the vertebral body adjacent to the treated vertebra was calculated. Survival analysis was performed using the Kaplan-Meier method and Cox proportional hazards regression analysis. RESULTS: Seventy-two (7.9%) new fractures were revealed. The Kaplan-Meier estimate of the 1-year fracture-free rate was 93.1%. The mean fracture-free interval was 32 months (95% CI, 32 to approximately 33 months). The greater degree of height restoration of the treated vertebra, the location of the adjacent vertebra in the TL junction, and the shorter distance between the treated and the adjacent vertebra increased the risk of new vertebral fractures. CONCLUSION: Thoracolumbar junction, shorter distance from the treated vertebrae, and greater degree of height restoration of the cemented vertebrae may increase the fracture risk of vertebrae adjacent to cement vertebrae after vertebroplasty.

Adult↗
PubMed2002

Osteoporotic vertebral compression fracture causing neurologic deficit.

Osteoporotic vertebral fractures rarely cause neurologic deficits. We discuss a 69-year-old man who presented with the signs of impaired function of the left L5 nerve root. Imaging revealed a fracture of the posterior upper edge of the fifth lumbar vertebra with a bony fragment protruding into the spinal canal. The fracture was attributed to sneezing and coughing in this man with osteoporosis secondary to hypogonadism. Literature search revealed 64 cases of neurologic deficit associated with osteoporotic vertebral fracture. Most osteoporotic fractures with neurologic deficit (72%) occur at the thoracolumbar junction. The symptomatology of girdle-like pain and hypesthesia is not specific and may be misinterpreted as an abdominal problem or renal pain. A typical sign is the slowly progressive neurologic deficit, occurring spontaneously or after minor trauma such as a fall on the buttocks. Surgical treatment confirms the diagnosis and usually leads to remission of the neurologic signs. With increasing frequency of osteoporotic fractures in the developed world because of the expanding elderly population, cases with neurologic problems become more important.

Journal Article↗
PubMed1994

Bone mineral density and vertebral compression fracture rates in ankylosing spondylitis.

OBJECTIVE: To examine the relationship between disease severity and bone density as well as vertebral fracture risk in patients with ankylosing spondylitis (AS). METHODS: Measurements were taken for bone mineral density (BMD) and vertebral fracture rates in 87 patients with AS. BMD was measured at the hip (femoral neck -FN), lumbar spine (L1-L4-LS) and for the whole body using a hologic-QDR-1000/W absorptiometer. An algorithm based on normal female ranges of vertebral heights was used to define a fracture as occurring when two vertebral ratios were each three standard deviations below the calculated mean of the controls. RESULTS: Patients with AS had significantly lower FN-BMD in proportion to disease severity (based on a Schober index) and disease duration. LS-BMD was also reduced in early disease, but in patients with advanced AS it had increased considerably. Nine vertebral fractures (10.3%) were identified which was considerably higher than expected when compared with a fracture of 1.9% in a control population of 1035 females of a similar age range. Patients with AS with fractures were significantly older, more likely to be male, had longer disease duration and more advanced spinal limitation with less mobility. There was no significant reduction in lumbar spine or femoral neck bone density in the fracture group. CONCLUSIONS: Vertebral fractures that result from osteoporosis are a feature of longstanding AS. BMD used as a measure of osteoporosis of the spine in advanced AS is unreliable probably as a result of syndesmophyte formation and does not predict the risk of vertebral fracture. Alternative sites such as the neck of the femur should be used for sequential assessment of BMD in AS.

Adult↗
PubMed1987

Vertebral compression fractures and mineral metabolism in chronic obstructive lung disease.

Chronic obstructive lung disease has been reported as a cause of osteoporosis, though whether this association is due to the disease itself or to corticosteroid treatment has not been elucidated. We studied 44 male patients with chronic obstructive lung disease (mean (SD) FEV1 39% (14%) of predicted normal) who were not having long term corticosteroids. No differences in a vertebral deformity score or in metacarpal index were found between them and a control group of similar age. Indices of bone formation (serum osteocalcin) and bone resorption (urinary hydroxyproline) were normal and parathyroid hormone and 1,25-dihydroxyvitamin D were also normal. Serum 25-hydroxyvitamin D was decreased, indicating depleted vitamin D. Calcitonin concentrations were higher in the patient than in the control group of the same age. There was no increase in the prevalence of osteoporosis in patients with chronic obstructive lung disease who had not received long term corticosteroid treatment. Increased concentrations of calcitonin may protect the skeleton from the detrimental effect of hypovitaminosis D.

Bone and Bones↗