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At least 451 records · Page 25Linked to original sources
PubMed1991

Mediastinal abscess resulting from dental infection.

A case of dental sepsis is presented which resulted in a deep neck infection and eventually caused a mediastinal abscess. The microbiology of such a mixed infection is briefly discussed with emphasis on the synergistic activity of the organisms. The anatomy of the fascial spaces is also described to draw attention to the possible routes of spread of this potentially life threatening condition.

Abscess↗
PubMed1992

Intraoral examination in pyogenic facial lesions.

Pyogenic cutaneous lesions of the cervicofacial region may be due to a variety of causes. One possibility that should be considered is an odontogenic infection secondary to untreated dental caries, periodontal disease or previous maxillofacial trauma. An intraoral examination is mandatory to evaluate the oral cavity for signs of pathology that may be manifested as a purulent cutaneous lesion. Patients with odontogenic infection should be referred to a dentist for definitive treatment, which may consist of either endodontic therapy or extraction of the involved tooth and curettage of any abscesses or fistulous tracts.

Adult↗
PubMed1999

[Clinical studies on the pathophysiology of odontogenic abscesses].

In 26 patients with abscesses in the maxillofacial area, the electrolyte concentrations, pH and osmotic and hydrostatic pressures of the pus fluid were measured and calculated. The main cations identified were sodium (134 +/- 38 mmol/l) and potassium (37 +/- 16 mmol/l) and as anions chloride (183 +/- 46 mmol/l) and bicarbonate (10 +/- 4 mmol/l). The pH value of the pus liquid was 6.164 +/- 0.233. The calculated mean osmotic pressure of the pus liquid was 7910 +/- 1455 mm Hg, whereas the measured physical pressure inside the abscess was 49 +/- 13 mm Hg. Both pressure types show time-dependent pressure curves. With time, the real pressure inside the abscess cavity increases, whereas the osmotic pressure decreases. There was no relationship between the two pressure types and the different species of microorganisms responsible for the inflammation. The results of the study reveal that abscesses can be regarded as osmotically active systems, and the mechanism by which the abscess is formed might be as follows. After penetration of virulent microorganisms into the tissue space, the area of acute inflammation is walled off by the collection of inflammatory cells. Destruction of tissue by products of the polymorphonuclear leucozytes takes place and results in liquefactive necrosis and a hypertonic abscess cavity. The inwards-directed flow of tissue fluids into the cavity via the abscess membrane causes volume expansion and generates pressure, two facts that can explain the swelling dynamics and typical symptoms of abscesses in the maxillofacial area.

Humans↗
PubMed1978

Abscesses of the frontal lobe of the brain secondary to covert dental sepsis.

The bacterial species found in pus aspirated from brain abscesses in two patients were typical of those found in dental sepsis. Subsequently apical-root abscesses were demonstrated in the upper jaws of both patients. This evidence strongly suggests that these cerebral abscesses were secondary to dental sepsis which could have spread from the teeth to the frontal lobes by several possible antaomical pathways.

Actinobacillus Infections↗
PubMed1986

Isolation of capsulate anaerobic bacteria from orofacial abscesses.

The presence of capsulate Bacteroides spp. and anaerobic gram-positive cocci was investigated in pus specimens from 182 children with chronic orofacial infections or abscesses and in pharyngeal swabs from 26 children without inflammation. Of 216 Bacteroides spp. and anaerobic cocci isolated from clinical infections, 170 (79%) were capsulate, compared with 34 (35%) of 96 isolates from normal pharyngeal flora (p less than 0.001). The commonest organisms found to be capsulate more often from infected patients than from controls belonged to the B. melaninogenicus group. The possible evolution of encapsulation in these organisms and their importance in mixed orofacial infections are discussed.

Abscess↗
PubMed1982

Fusobacterium necrophorum septicemia following oropharyngeal infection.

Fusobacterium necrophorum septicemia developed in five patients after an oropharyngeal infection. Four patients had sore throat or neck pain, and two had findings of jugular vein septic thrombophlebitis. Metastatic abscesses, including embolic pneumonia, empyema, septic arthritis, and osteomyelitis, also occurred. Four patients recovered and one died. Proper treatment requires recognition of the oropharyngeal source of the septicemia and its differentiation from endocarditis. Antibiotic therapy should be prolonged, and metastatic abscesses drained.

Adolescent↗
PubMed1996

Burkitt's lymphoma mimicking an acute dentoalveolar abscess.

Burkitt's lymphoma is a monoclonal proliferation of B lymphocytes classified histologically as a poorly differentiated lymphocytic lymphoma. The jaw and retroperitoneal structures are the most commonly involved sites. Prognosis is highly dependent on the stage of the disease. In some cases, the first manifestation of Burkitt's lymphoma is in the jaws, and symptoms may be misdiagnosed as infection. Dental radiographs can play an important role in the diagnosis. A case of a peculiar Burkitt's lymphoma involving the mandible that was misdiagnosed as an acute dentoalveolar abscess is presented.

Adolescent↗
PubMed1988

Assessment of the pathogenicity of bacterial species isolated from acute dentoalveolar abscesses.

The pathogenicity of 20 strains belonging to nine bacterial species isolated from acute dentoalveolar abscesses was assessed individually and in two species combinations by subcutaneous inoculation of mice. Infections were produced by all the bacteria although variations were seen both in the type of lesion produced and the subsequent recovery of viable bacteria. Anaerobic gram-negative bacilli were recovered more often (p less than 0.05) at high concentrations (10(6)-10(9) cfu/ml) and produced a localised abscess with peripheral necrosis more frequently (p less than 0.001) than either Streptococcus milleri or anaerobic gram-positive cocci. Lesions induced by a combination of bacteria comprising anaerobic gram-negative bacillus and any other species yielded both strains at high concentration more often (p less than 0.001) than a combination comprising anaerobic gram-positive cocci and S. milleri. It is concluded that anaerobic gram-negative bacilli are major pathogens in acute dentoalveolar abscesses.

Acute Disease↗