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At least 451 records · Page 25Linked to original sources
PubMed1987

[Interaction of Leptospira with the host's body in the infectious process in guinea pigs].

In experiments on guinea pigs the pathogenicity of leptospires is manifested by their adhesion to liver cells, colonization of the surface of these cells, accumulation of leptospires in the renal interstice and their colonization of the nephrothelial surface of proximal convoluted tubules in the kidneys, by toxic microcirculatory disturbances, dystrophy and necrosis of nephrothelial cells. Then the primary toxic action of circulating leptospires, microcirculatory disturbances and hemorrhagic syndrome augment during the colonization of the surface of liver cells, accompanied by their dystrophy and dissociation, as well as by jaundice. The accumulation of leptospires in the renal interstice and their subsequent multiplication on the nephrothelium of the proximal convoluted tubules are responsible for the development of interstitial nephritis and necrotic nephrosis. The persistence of lesions in the liver and kidneys, occurring in some cases in spite of elimination of the infective agent from these organs due to increasing antibody production suggests the toxic action of immune complexes.

Animals↗
PubMedDate not supplied

[Effect of intensive operator activity on lipid peroxidation processes in the human body].

It has been demonstrated that the stress associated with an active mental work for 2 hours leads to a high rate of lipid peroxidation which results in a greater amount of products of lipid peroxidation in blood and pentane in exhaled air. Simultaneous measurements of blood catecholamines have shown their significant increase immediately after exposure to the stress effect. It can therefore be concluded that a stress situation enhances lipid peroxidation in the human body.

Breath Tests↗
PubMed1982

[Action of cooling on cells and the body as a multifactorial process].

The role of different cryogenic factors in the process of freezing and thawing of the cells and other biological systems is discussed. Attention is paid to the fact that cryo injury is arising all over freezing-thawing, to be eventually determined by the effect of the complex of damaging factors. It is shown that a successful anabiosis under very low temperatures depends upon the functional state of the organism before cooling, morphophysiology of cells, their reaction to supercooling, extra- and intracellular crystallization, and on rates and duration of cooling and warming.

Adaptation, Physiological↗