PubMed Health⌕ Search

SEARCH · PubMed Health

Results for “SARTORIUS MUSCLE”

Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 451 records · Page 25Linked to original sources
PubMed1983

K+ depolarization and phospholipid metabolism in frog sartorius muscle.

K+ depolarization evokes phosphatidylinositol response, i.e. the increased 32P orthophosphate labelling of phosphatidylinositol in frog sartorii muscles. The phosphatidylinositol response seems to be closely related to K+ depolarization and not to the transient Ca2+ release at the beginning of depolarization. It ceases as soon as the muscles depolarized by 90 mmol/l KCl for a short period of time are repolarized, while it continues when the depolarization is maintained. When the muscles are depolarized with 20 mmol/l KCl, the phosphatidylinositol response is also observed. This response is not suppressed by drugs that block Ca2+ mobilization. Other agents like caffeine, azide or EGTA which induce some effects similar to that of K+ depolarization, do not evoke phosphatidylinositol response. Rather, they simply cause a decrease in the labelling of phospholipids, phosphatidylinositol being the least affected. In muscles derived from frogs maintained under healthy conditions Ca2+ release in the early phase of K+ depolarization does not cause significant changes in phospholipid labelling. However, in muscles from frogs starving for many months, a large decrease in the labelling of phospholipids is observed in the early phase of K+ depolarization. It is postulated that the changes in the physicochemical state of the membrane and not Ca2+ gating mechanism or free cell Ca2+ level are crucial in the phosphatidylinositol response in the frog sartorii muscles depolarized by high K+.

Animals↗
PubMed1994

Sartorius muscle contracture in a dog.

An adult male German Shepherd dog presented with a chronic non-painful, non-weight bearing lameness of the right hind limb. On clinical examination a contracted sarotorius muscle was palpable. Histopathology of the affected muscle confirmed replacement fibrosis and muscle contracture. Surgical excision of the muscle resulted in complete recovery.

Animals↗
PubMed1977

[Effect of proserine and eserine on synaptic potentials of the frog sartorius muscle].

Anticholinesterases proserine (2-10(-7)-2-10(-6) M) and eserine (2-10(-6)-2-10(-5) M) prolonged end-plate poteutials (e. p. p. s.) and miniature end-plate potentials (m. e. p. p. s.) and increased their amplitude; higher concentrations of these drugs first increased and then decreased the amplitude and duration. Thus, higher concentrations, in addition to anticholinesterase effect, exert also the inhibiting influence on postjunction membrane. These two effects are reversible when washing out the drugs. Neither proserine nor eserine affect the frequency of m. e. p. p. and the quantum content of e. p. p. No cholinssensitive effect of these drugs was observed.

Animals↗