PubMed Health⌕ Search

SEARCH · PubMed Health

Results for “Feeding Behavior”

Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 469 records · Page 26Linked to original sources
PubMed1988

Liver phosphorylase activation during early phase of feeding behavior: a neurohumoral regulation.

The mechanisms of liver glycogen depletion during the early phase of spontaneous feeding were examined in adult rats. A 40% increase in the activity of phosphorylase a, the active form of a key glycogenolytic enzyme, was detected 5 min after the onset of feeding, with a concomitant decrease of glycogen in the liver after 5 and 15 min. Within 60 min after the onset of feeding, the enzyme activity returned to the basal level and the glycogen content was restored. These metabolic responses were impaired by hepatic sympathetic denervation or by bilateral adrenodemedullation. It was concluded that the onset of meals may evoke an arousal of the sympathoadrenal system including the hepatic innervation, and that activation of this system is important in stimulating liver phosphorylase activity and glycogenolysis during the early phase of spontaneous feeding.

Adrenal Medulla↗
PubMed1989

[Dexfenfluramine and feeding behavior. Clinical and pharmacoclinical studies].

Dexfenfluramine is undoubtedly the most studied molecule in the field of feeding behaviour and it has increased our fundamental knowledge of food intake and body mass regulation. Animal and human studies have shown that dexfenfluramine increases the degree of satiety induced by food intake. Dexfenfluramine seems to act directly on the fundamental mechanisms of energetic regulation. Dexfenfluramine also has an original effect on some food intake disorders. In "carbohydrate cravers", dexfenfluramine reduces total food intake by selectively reducing carbohydrate snacks without reducing protein intake significantly.

Animals↗
PubMed2004

The relationship between parental eating problems and children's feeding behavior: a selective review of the literature.

This review describes the research evidence linking parental eating problems with processes that affect children's food selection and rejection, including child feeding difficulties, and child dieting practices and attitudes. First, studies that assess the relationship between parental eating disorder and child feeding are reviewed. Secondly, research that examines the relationship between parent and child dieting behavior and attitudes is reviewed. Finally, the eating characteristics of parents whose children have feeding difficulties are discussed. There is no consensus in the literature about the relationships between parent and child feeding and eating disturbance, and mechanisms for the intergenerational transmission of these behaviors have yet to be established.

Adult↗
PubMed1993

Effect of neonatal androgenization on the circadian rhythm of feeding behavior in rats.

The effect of neonatal androgenization with testosterone propionate (TP) on growth, food intake, and feeding circadian and ultradian rhythms was studied in male and female rats. TP-female rats but not TP-male rats weighed more and ate more in adulthood than control animals injected with oil. TP administration to male rats induced few changes in the circadian organization of food intake and meal size. However, neonatal androgenization of female rats produced an approach of feeding parameters to those showed by male rats: diminished meal number and increased meal size and food intake. Moreover, the spectral analysis of meal frequency revealed that TP induced in female rats an enlargement in the 4.8 h ultradian component. Although hormone secretions produced in adult animals contribute to sexual dimorphism, it is noticeable that androgen presence during the organizational period is largely responsible for differences between sexes.

Animals↗
PubMed1995

Changed central and peripheral catecholamines and indoleamines in one-way crossed-intestine rats: relationship to changes in feeding behavior and metabolism.

The involvement of central and peripheral catecholamines and serotonin (5-HT) in regulation of feeding and energy metabolism was examined in one-way crossed intestine rats that show large and sustained changes in daily food intake. Five to six weeks after the crossed-intestinal surgery, catecholamines and indoleamines in dissected major brain regions and in the heart, intrascapular brown adipose tissue (IntBAT), pancreas, and serum were determined using HPLC with electrochemical detection. The food-losing rats increased daily food intake from 70.8 to 126.3 g, whereas their partners decreased daily food intake from 67.1 to 38.7 g (p < .001). Compared with the partners and sham-operated controls, the food-gaining rats had increased 5-hydroxy-indoleacetic acid (5-HIAA) throughout the brain [hypothalamus, 442.9 vs. 383.5 (p < .05) and 404.2 ng/g (p < .05), Lateral cortex plus amygdala (LC + A), 236.6 vs. 216.8 (p < .05) and 212.0 ng/g (p < .05), brain stem, 282.9 vs. 238.7 (p < .05) and 245.1 ng/g (p = .05), cerebellum, 56.7 vs. 49.6 (p < .05) and 44.4 ng/g (p < .05)]. Higher 5-HIAA in food-gaining rats that were undereating are consistent with serotonin's role in inhibiting food intake. Peripherally, the rats gaining food showed significantly lower NE, 5-HT, and 5-HIAA in IntBAT compared with their partners (NE, 994.2 vs. 1236 ng/g, 5-HT 338.0 vs. 527 ng/g, and 5-HIAA, 39 vs. 51 ng/g). Because NE and 5-HT have been shown to exert stimulating effects on BAT-mediated thermogenesis, lower levels of NE, 5-HT, and 5-HIAA in IntBAT of food gaining rats are compatible with the lower metabolic rate observed in these animals. The results show that both central and peripheral catecholamines and serotonin are involved in regulation of food intake and energy metabolism.

Analysis of Variance↗
PubMed1993

Antagonism of estrogenic effects on feeding behavior by central implants of anisomycin.

The following experiment determined whether the estrogenic suppression of food intake is dependent upon changes in protein synthesis within neurons of the paraventricular nucleus of the hypothalamus (PVN). Ovariectomized rats were treated centrally with anisomycin-filled or empty (control) cannulae in the PVN. Females were injected with either 2.0 micrograms of estradiol benzoate (EB) or the oil vehicle and the inner cannulae were removed 2 h later. EB injections significantly lowered food and water intake in the central control group but not in animals given PVN implants of anisomycin. Body weight gain decreased for all females. EB induced comparable levels of female sexual behavior in both groups, demonstrating that anisomycin implants did not affect the ability of estradiol to stimulate lordosis. These findings indicate that the effects of estradiol on food intake require the activation of protein synthesis in estrogen-sensitive PVN neurons.

Animals↗
PubMed1965

EATOMETER: A DEVICE FOR CONTINUOUS RECORDING OF FREE-FEEDING BEHAVIOR.

Minor modifications of a commercially available food cup permit automatic recording of a rat's contacts with common dry food. It is thus possible to measure the frequency and duration of the "meals" that rats take over extended periods of time, and, in conjunction with existing devices for measuring drinking, to record contingencies between eating and drinking.

Animals↗
PubMed2000

Prey transport kinematics in Tupinambis teguixin and Varanus exanthematicus: conservation of feeding behavior in 'chemosensory-tongued' lizards.

Although lizards have been predicted to show extensive intraoral prey-processing behaviors, quantitative analyses of the types of prey-processing behavior they demonstrate and of their kinematics have been limited. The more basal lizard lineages (Iguanians) have undergone some study, but the prey-processing repertoires of crown taxa have not been thoroughly examined and quantitative comparisons of behaviors within or among species have not been made. In this study, the prey transport behavior of the savannah monitor (Varanus exanthematicus) and gold tegu (Tupinambis teguixin) are described. Although these two lineages have independently evolved tongues that are highly specialized for chemoreception, we found that they share the same three distinct types of transport behavior. These behavior patterns are (i) a purely inertial transport, (ii) an inertial transport with use of the tongue, and (iii) a non-inertial lingual transport. The tongue is used extensively in both the inertial and the purely lingual transport behaviors. More than 75 % of all transport behaviors involved tongue movements. These species appear to exhibit a conservation of feeding kinematics compared with patterns known for basal lizards. A hypothesis for the evolution of inertial feeding is proposed.

Animals↗
PubMed2005

[Feeding behavior of Serangium japonicum (Coleoptera: Coccinellidae), a predator of Bemisia tabaci (Homoptera: Aleyrodidae)].

Serangiurm japonicum is an important predator of whiteflies in China. In this study, the Bemisia tabaci-attacking behavior of S. japonicum adults and larvae was observed by a videocassette recorder. The results showed that the predatory behavior of S. japonicum adults was characteristic with six events, i.e., walking, feeding, clearing, resting, vibrating wings, and excreting. The starved larvae of S. japonicum was fond spending all times in feeding and walking on host eggs, with no time in resting or clearing throughout the process. The time included in feeding and walking was not significant different among different instars of S. japonicum. The handing time per egg decreased with S. japonicum larvae developed from instar I to IV, but differed little between instar III and IV. The starved females of S. japonicum spent longer time on egg feeding, but less time on clearing, resting and walking than satiated females. The time for starved female to predate on B. tabaci nymphs and pupae increased as the host nymphs developed.

Animals↗