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Results for “GINGIVAL HYPERPLASIA”

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At least 469 records · Page 26Linked to original sources
PubMed1998

[Gingival hyperplasia in pregnancy. II. Etiopathogenic factors and mechanisms].

The principal aspects of the hyperplastic pathology which frequently appears during pregnancy with a particular regard to the etiopathogenetic hypotheses are presented. The etiopathogenesis of the so-called pregnancy granuloma is rather complex, because in addition to the common irritant factors like bacterial plaque and calculus it is necessary to recognize the influence that hormonal modifications in pregnancy have on the predisposition to the development of gingival lesions. The hormonal influence acts by an intense inflammatory response and by a selective growth of some periodontal-pathogen and aggressive microbes, like Prevotella intermedia, whose increase in the subgingival plaque from the 3 degrees-4 degrees month of pregnancy coincide with the beginning of hyperplastic lesions. Sexual hormones seems to act as growth factors for the subgingival bacterial flora, in particular for Gram-negative anaerobic rods. The worsening of gingival conditions appears prevalently in the first quarter of pregnancy, in concomitance with the increase of urinary excretion of gonadotropins, that is in relation with an increased concentration of estrogens and progesteron in circulating blood. Sexual hormones could cause great modifications in peripheric vascular system, with a marked increase of vascular permeability and the following oedema of gingival tissues. In pregnancy the maternal immunological system presents some particular modifications, that allows to avoid the immunological rejection between mother and fetus, but reduce the reactivity of gingival tissues against local irritating factors.

Female↗
PubMed1975

[Gingival hyperplasia in bar joint prostheses].

The occurrence of mucosal hyperplasias under 57 bars (according to Dolder) was examined. It was found that apart from the time factor, cleaning habits of the patient and the construction of the dentures played an important role. Short bars are more difficult to clean than those with wide distances between the abutments. The influence of additional factors can only be demonstrated in combination with other factors, this suggests rather complex connections.

Denture Design↗
PubMed2005

[Cyclosporine induced gingival hyperplasia in kidney transplants].

OBJECTIVE: Assess the influence of cyclosporine on the gingival growth of 60 patients with kidney transplant through a research carried out at the Hospital Cajuru in Curitiba, (April to October of 2002). METHODS: Regardless of age, gender, ethnic or social condition, all patients received cyclosporine daily and had, at least, one dental segment. They monthly returned to the Hospital for medical control and for several laboratory exams, among them the cyclosporine serum level. This permitted observation of these cases, especially with respect to the reaction of gingival growth. Clinical dental exams of the patients comprised filling out a form on their personal data, height and weight and number of dental segments. Also an observation of the degree of the gingival and bacterial plaque index was recorded. These data were treated statistically. RESULTS: Of the patients treated only with cyclosporine, 47.2% showed no alterations of the gingival structure while 52.8% presented gingival growth, of them 33.3% with a > 2 degree. Of the patients treated with both cyclosporine and nifedipine, 33.3% had a normal gingiva and 66.7% presented gingival growth. In 45.8% of them, the alteration was of > 2 degree, with no significant difference between results of the male and female patients. A positive correlation between excess fat and the corrected cyclosporine dose and serum concentration per unit of Body Mass Index was found, also between excess fat and the bacterial plaque index. A significant association between the extent of increased gingival growth and the bacterial plaque index was also observed. CONCLUSION: Use of cyclosporine when associated with nifedipine brings about an increase of gingival growth.

Adult↗
PubMed2002

[Treatment of periodontitis associated with cyclosporin-induced severe gingival hyperplasia with regular mechanical plaque removal in a patient with renal transplantation (Case report)].

A clinical case of a middle-aged kidney transplanted woman is presented. The woman has been taking Cyclosporin-A for over 13 years and has had advanced periodontitis and severe gingival swellings and gingival inflammation. The kidney transplant patient was treated and followed up for approx. four years. The treatment protocol included very thorough mechanical scaling and root planing, oral hygienic instructions and the regular professional maintenance program resulted in complete remission of the gingival overgrowth and stabilization of the periodontal condition. The gingival and periodontal conditions showed a continuing improvement over the time despite of the continuous CSA administration.

Cyclosporine↗
PubMed1993

Cyclosporin-induced gingival hyperplasia: an ultrastructural study of the oral epithelial prickle cells.

In this study the ultrastructural characteristics of the prickle cells of the human gingival epithelium after cyclosporin-A treatment are described. The prickle cell layer shows hyperplasia, along with the well known hyperplasia of the connective tissue components. The prickle cells show large numbers of free ribosomes, rough endoplasmic reticulum profiles and well developed Golgi complexes. The intercellular space is usually large and the neighboring cells are joined by desmosomes. In many cases the intercellular space is almost diminished and the cells are joined by a lot of desmosomes. Occasionally, a single cilium was observed to project into the intercellular space but the functional significance of this observation is unknown. The tonofilaments exhibit a specific arrangement in the cytoplasm of the prickle cells and it is believed that they contribute to the formation of a 'tonofilament network' which, along with the desmosomes, provide strength to the epithelium. Possible mechanisms regarding the gingiva overgrowth are discussed.

Actin Cytoskeleton↗
PubMed1992

Soft tissue management during implant maintenance.

Peri-implant soft tissue problems seen during the maintenance phase of implant therapy include an inadequate zone of keratinized tissue, mobility of grafted tissue, mucosal "pouching," gingival hyperplasia, gingival fistulas, and gingivitis. Techniques to alleviate or reduce these problems include free soft tissue autografts, apically positioned flaps, reduction of tissue thickness by a flap procedure or gingivectomy, and increased attention to the control of inflammation.

Adult↗
PubMed2001

Inter-abutment and peri-abutment mucosal enlargement with mandibular implant overdentures.

A variety of terms have been used to describe changes in the oral mucosa around abutments underneath fixed and removable implant prostheses such as mucosal inflammation, peri-implant mucosal hyperplasia, gingival hyperplasia, hyperplastic tissue, mucosal proliferation, proliferative gingivitis and peri-implant mucositis. Although such terms have become part of the evidence-based literature, there is no histological evidence to support the descriptive terminology used by authors. The use of the alternative term mucosal enlargement for clinical observations underneath mandibular implant overdentures is proposed. Prospective and retrospective reports have failed to use similar criteria to measure periodontal parameters in relation to these mucosal changes, both in keratinized or non-keratinized tissues. Therapeutic remedies for mucosal enlargement are dictated by authors' opinions on the possible aetiology. This literature review, coupled with clinical observations during a 5-year prospective study, prompts this proposal of the term mucosal enlargement underneath splinted and unsplinted mandibular implant overdentures.

Dental Abutments↗
PubMed2005

Common benign oral soft tissue masses.

This article reviews some of the more common benign oral soft tissue masses with emphasis on their etiology, epidemiology, clinical presentation, histopathology, and treatment. These lesions include traumatic fibroma, mucocele, warts/papilloma, pyogenic granuloma, peripheral giant cell granuloma, generalized gingival hyperplasia, gingival fibromatosis, lateral periodontal cyst, lipoma, and denture-induced hyperplasia.

Adolescent↗
PubMed2007

Long-term uncontrolled hereditary gingival fibromatosis: a case report.

Hereditary gingival fibromatosis (HGF) is a rare condition characterized by varying degrees of gingival hyperplasia. Gingival fibromatosis usually occurs as an isolated disorder or can be associated with a variety of other syndromes. A 33-year-old male patient who had a generalized severe gingival overgrowth covering two thirds of almost all maxillary and mandibular teeth is reported. A mucoperiosteal flap was performed using interdental and crevicular incisions to remove excess gingival tissues and an internal bevel incision to reflect flaps. The patient was treated 15 years ago in the same clinical facility using the same treatment strategy. There was no recurrence one year following the most recent surgery.

Adult↗
PubMedDate not supplied

[The causes of gingival overgrowth].

Gingival overgrowth includes a series of diseases with many clinical appearances. The pathological mechanisms being obscure there were used many terms for defining it. Thus, "gingival hyperplasia" and "gingival hypertrophy" were the definitions used to define this pathology. Therefore, the term of "gingival overgrowth" replaced in last decades the above two terms. This article have the goal of trying a classification of the entities forming the large family of gingival overgrowth.

Dentures↗
PubMed2000

Simultaneous hyperplasia, metaplasia, and neoplasia in an 8 year-old boxer dog: a case report.

Generalized gingiva hyperplasia and multiple gingival masses were observed in an 8 year-old boxer dog. The masses were removed surgically. Three of them were biopsied. Microscopic examination was reported as fibrous gingival hyperplasia, fibrous epulis with osseous metaplasia, and mast cell tumor. Twenty-two months following surgery, moderate generalized gingival hyperplasia was present, with no clinical evidence of regrowth of the neoplasms.

Animals↗