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Results for “AMELOGENESIS IMPERFECTA”

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PubMedDate not supplied

Ectodermal dysplasias.

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Amelogenesis Imperfecta↗
PubMed2005

Impaired tooth eruption: a review.

Eruption is the continuous process of movement of a tooth from its developmental location inside the jaw to its functional location in the mouth. Impaired tooth eruption, where this process is disturbed, is common in dental practice. It may manifest either as delayed or complete absence of eruption. Although unerupted teeth are usually asymptomatic, they may cause cosmetic and pathologic complications. The purpose of this article is to provide a review on the pathogenesis and differential radiographic interpretation of impaired tooth eruption.

Amelogenesis Imperfecta↗
PubMedDate not supplied

[A rare dental abnormality: "the phantom tooth" or "odontodysplasia". Apropos of a case].

"Phantom tooth" or "odontodysplasia" is one of the most unusual and rare of dental abnormalities, being characterised: Clinically, by the topography of the lesions, the involvement of both sets of teeth and abnormalities in the size and form of the teeth which remain unrupted or only partially evolved. Radiologically, by transparency of the teeth to X-rays. Histologically, by disorganisation of the dental structures and a highly characteristic appearance of the enamel and, in particular, dentine. On the basis of one case, the authors review in detail each aspect of these characteristics, paying special attention to histology and the information obtained using the sweep electron microscope.

Amelogenesis Imperfecta↗
PubMedDate not supplied

Molecular determinants during dental morphogenesis and cytodifferentiation: a review.

Craniofacial development provides a number of opportunities to investigate the cellular and molecular biology of morphogenesis, cytodifferentiation, tissue-specific extracellular matrix (ECM) formations, and biomineralization. Regulatory processes associated with mandibular morphogenesis and specifically tooth formation are being investigated by the identification of when and where molecular determinants such as cell adhesion molecules (CAMs), substrate adhesion molecules (SAMs), and tissue-specific structural gene products are expressed during sequential developmental stages. Based upon in vitro organotypic culture studies in serumless, chemically defined medium, instructive and permissive signaling has been found to be required for both mandibular and dental morphogenesis and cytodifferentiation. For example, intrinsic developmental instructions (autocrine and paracrine factors), independent of long-range hormonal or exogenous growth factors, mediate morphogenesis from the initiation of the dental lamina through crown and initial root stages of tooth development. This review summarizes recent results using experimental embryology, organ culture, recombinant DNA technology, and immunocytology to elucidate mechanisms responsive to instructive epithelial-mesenchymal interactions associated with mandibular morphogenesis, tooth positional information, and subsequent tooth crown and initial root development.

Amelogenesis Imperfecta↗
PubMedDate not supplied

[Anomalies of enamel structure: description of a clinical case].

The anomalies of dental structure are quite common in pediatric dentistry; these anomalies can involve separately or in the same time enamel, dentin or cementum and they are due to many factors acting during odontogenesis and istogenesis. The Authors analyse these abnormalities and describe a personal case.

Adult↗
PubMed1990

Prevalence and distribution by tooth type and surface of developmental defects of dental enamel in a group of 15- to 16-year-old children in South Wales.

In 1984 the prevalence and distribution of developmental defects of dental enamel in 791 children aged 15-16 years was determined using the DDE index. The children were all residents of the county of South Glamorgan, Wales, which has a public water supply containing less than 0.1 mg fluoride/litre. The teeth were dried but not cleaned prior to examination and a dental operating light was used for illumination. Teeth with some type of defective enamel were seen in 50.1 per cent of children. White/single opacities were present in 28.3 per cent of children and white/diffuse, patchy opacities in 10.2 per cent of children. The enamel was abnormal in 5.71 per cent of all teeth with white/single opacities and white/diffuse, patchy opacities occurring in 1.81 per cent and 1.66 per cent of teeth respectively. There were no significant differences between boys and girls in terms of mouth or tooth prevalence of defects. Overall, 7.6 per cent of maxillary teeth were affected compared to 4.3 per cent of mandibular teeth. Most teeth were affected equally on right and left sides of the mouth except for maxillary lateral incisors and canines which had significantly more defects on the right (P less than 0.05 and P less than 0.005 respectively) and maxillary second molars, which had significantly more defects on the left (P less than 0.01). Maxillary central incisors (15.2 per cent) were affected by enamel defects most often followed by maxillary first molars (9.1 per cent) and maxillary lateral incisors and mandibular first molars (both 8.3 per cent).(ABSTRACT TRUNCATED AT 250 WORDS)

Adolescent↗
PubMed1975

The teeth.

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Amelogenesis Imperfecta↗
PubMed1989

Dental abnormalities associated with campomelic syndrome: case report.

Campomelic syndrome is a rare autosomal recessive disease. It is characterized by short stature with angulation and bowing of the lower limbs, hypoplasia of the facial bones, and various other skeletal anomalies. The facies are unusually flat with micrognathia, frequent cleft palate, hypertelorism, and micro-ophthalmia. Most infants with this syndrome die from severe respiratory distress within the first hours or days of life. Those who survive infancy are mentally retarded. This case report concerns the dental treatment of a 13-year-old female with campomelic syndrome.

Adolescent↗
PubMed1977

[A particular type of dominant hereditary enamel dysplasia?].

An enamel dysplasia which is obviously transmitted as a dominant trait, is observed in various members of one family over three generations. The picture of affected and non-affected teeth of both dentitions is different from the usual behavior of generalized enamel hypoplasia, as only a delimited period of enamel formation is affected. Findings of exogenous inflammatory changes in enamel make the nature of this hereditary odontopathia more understandable. The origin of this enamel hypoplasia is supposed to be a genetic phase-specific process in the stage of maximum metabolic activity (enamel formation). In different age groups, this disorder shows different degrees of severity, suggesting the existence of modifying factors. Among the subjects, the male sex is predominant, while the females exhibit more marked hypoplasias. Whether this is a new variant of hereditary dusturbance of enamel formation can be confirmed by examination of other large families.

Amelogenesis Imperfecta↗