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PubMed1988

[Color vision in cataract, aphakia and pseudophakia].

Color vision examinations were performed using a clinical test battery and two spectral laboratory methods. With aphakia and iris-clip lenses (ICL) there were slight acquired blue-yellow defects six months to three years after surgery, especially when the more sensitive laboratory methods were used. They were possibly caused by photochemical damage to the retina or by a barrier deprivation syndrome. Color vision with posterior chamber lenses (PCL) was superior to that with ICL, and in aphakic subjects it was quite normal. In the clinical tests no differences could be found between PCLs of clear PMMA material and those with UV absorbing properties. Slight acquired blue-yellow defects in the immediate postoperative phase after implantation of PCLs can be attributed to postoperative irritation and are reversible. Lasting and severe blue-yellow defects indicate inflammation or macular edema.

Adult↗

PubMed1991

[Ocular side effects of beta-pyridylcarbinol].

Derivatives of nicotinic acid such as beta-pyridylcarbinol play an important role in the therapy of lipoprotein disorders. In 1973, J.D. Gass reported the development of cystoid macular edema provoking metamorphosia during the course of nicotinic acid treatment. The aim of this study was to determine subtle changes in ocular function induced by beta-pyridylcarbinol. We investigated 16 patients prior to and after 6 months of beta-pyridylcarbinol treatment and compared the results of clinical and color vision tests in 9 patients after 2-25 years of continuous beta-pyridylcarbinol treatment. After 6 months, significant blue-yellow color vision changes (total error scores within normal ranges) were detected by the Farnsworth-Munsell 100 hue tests in all patients. One patient demonstrated macular edema. Fluorescein angiography, however, showed no evidence of fluorescein leakage. Beta-pyridylcarbinol treatment lasting for years led to diffuse color vision disturbances (total error score = 195). To the best of our knowledge neither macular edema nor color vision disturbances following beta-pyridylcarbinol treatment have been reported so far.

Adolescent↗

PubMed2005

Diagnosing protan heterozygosity using the Medmont C-100 colour vision test.

BACKGROUND: A surprisingly high 15 per cent of women in Caucasian societies are carriers of the genes for abnormal colour vision but there is no clinical method to identify them. It has long been known that heterozygotes for the protan colour vision deficiencies can demonstrate a reduced luminous sensitivity to red light. This is known as Schmidt's sign, which is thought to arise from mosaicism (Lyonisation). The Medmont C-100 colour vision test measures relative spectral sensitivity using flicker photometry to differentiate protans and deutans. It should be able to diagnose Schmidt's sign. METHOD: We tested six known protan heterozygotes (four whose sons have a protan colour vision deficiency and two whose fathers are protan) with the Medmont C-100 test. RESULTS: All six heterozygotes made average settings of -1.75 or more negative at the Medmont C-100 test, settings which are at or beyond the boundary of the distribution of settings made by observers with normal colour vision. There have been two previous cases reported in the literature of protan heterozygotes, who made protan settings on the Medmont C-100 or its predecessor test, the OSCAR. We also tested six daughters of the known heterozygotes, 50 per cent of whom are likely to be heterozygotes. Four of the six (66 per cent) made protan settings on the Medmont C-100. The other two made normal 0.0 settings. CONCLUSION: We conclude that the Medmont C-100 can be used clinically to diagnose carriers of protan colour vision deficiency.

Color Perception Tests↗

PubMed2002

Colour blindness in Italian art high school students.

To highlight the link between colour blindness and school achievement, the Ishihara and Farnsworth tests were administered to 3,565 high school art students (2,545 girls and 1,020 boys). Analysis showed colour defective students were discriminated against in theoretical subject matter, relative to orthochromate students, but not in the art-related subjects. This emphasizes the need to recognize youth with colour defective vision early.

Adolescent↗

PubMed1995

Colorimetric analyses of various light sources for the D-15 color vision test.

Colorimetric analyses were performed in both normal trichromatic and dichromatic color spaces to determine whether several light sources were suitable illuminants for the Farnsworth-Munsell Panel D-15 (D-15) color vision test. Results for fluorescent lamps showed that lamps with a correlated color temperature (CCT) of 7200 degrees K and a general color rendering index (GCRI) of at least 90 are acceptable substitutes for illuminant C. Predictions for filtered tungsten light indicated that lights with a color temperature near 5000 degrees K are unsuitable because of nonuniformities in the glass daylight filter transmittance. Conclusions based on these analyses are conservative because, with exception of the GCRI, color adaptation effects were not taken into account.

Adaptation, Ocular↗

PubMed1999

Evaluation of a new color vision test: "color vision testing made easy".

PURPOSE: A new pseudoisochromatic color plate test, "Color Vision Testing Made Easy" (CVTMET) has recently been introduced. Said to be designed for all age groups, including pre-school children, it uses the identification of simple shapes and objects to detect red-green color deficiencies. We evaluated the CVTMET to determine if the test is suitable for color vision screening of young children. METHODS: Forty-one adults predetermined to be color normal (n = 20) or to have hereditary red-green color deficiency (n = 21), served as subjects. A battery of color vision tests including the Ishihara, Panel D-15, and the anomaloscope were used for diagnosis and color deficiency classification. Subjects were then tested with Part I and Part II of the CVTMET test and results were compared to the Ishihara, Panel D-15, and anomaloscope. In addition, the CVTMET was used to screen for color vision deficiency in 152 kindergarten children 5 to 7 years of age. RESULTS: The pass/fail results for the adult subjects were the same for Parts I and II and compared favorably with the anomaloscope. There were no false positives (100% specificity) and only a few (2 of 21) false negatives (90.5% sensitivity). The two color-deficient subjects who passed the CVTMET had the mildest color deficiencies (simple deuteranomaly) and also passed the Ishihara test. Testability of kindergarten children was found to be 100%. Color vision deficiency occurred in 5.06% of the boys, which is about the same frequency found in older boys of similar ethnic background. CONCLUSION: This preliminary study indicates that the CVTMET appears to be an excellent screening instrument for red-green color deficiency in adults and has been shown to be useful for examining color vision in children 5 to 7 years of age.

Adult↗

PubMed1984

Assessment of children's colour vision using the Pickford-Nicolson anomaloscope.

The colour vision of 439 boys, aged 4-11 years, was measured by the Pickford-Nicolson anomaloscope and four pseudoisochromatic tests. Matching range and dispersion of mid match point were found to be larger than adult values, but did not decrease with age. However, younger children took longer to establish matching range. Twenty-eight (6.4%) colour defectives were found and it is concluded that the Pickford-Nicolson anomaloscope gives valid results with children.

Age Factors↗

PubMed1992

Scotopization and pseudoprotanomaly in blue-yellow/colour vision defects.

With a routine clinical colour vision test battery we found scotopization in 32% of retinal diseases presenting with pseudoprotanomaly as sign of an acquired type III blue-yellow colour vision defect. In blue-yellow colour vision defects of retinal origin scotopization is a transient phenomenon, present in early stages of the disease, but it is not an obligatory finding. There is no evident relationship between visual acuity and scotopization.

Color Perception Tests↗

PubMedDate not supplied

Incomplete achromatopsia in Alzheimer's disease.

We report that patients with Alzheimer's disease (AD) have a selective deficit in blue hue discrimination, as assessed with three clinical measures of color vision. The Farnsworth D-15 Test, the Lanthony New Color Test, and the City University Color Vision Test were administered to 32 patients with AD (ranging in dementia severity from mild to severe) and 32 age-matched normal control subjects (NCS). Of the AD patients, 11 who were representative of the larger group for age, education level, and dementia severity received a complete neuro-ophthalmological examination that ruled out obvious disorders of the anterior visual structures. AD patients made significantly more tritan (blue) errors than NCS on all three color vision tests but did not make more protan (red) or deutan (green) errors on two of the three tests. The results support the conclusion that there is a deficit in color discrimination in AD that is specific to blue hues, and oppose the hypothesis that AD does not deleteriously affect the color-opponent visual channel. In the absence of obvious damage to anterior visual structures, the likely substrates for the observed deficit are peristriate and inferotemporal visual cortices, which are subject to significant neuropathology in AD.

Aged↗

PubMed1997

The visual photopigments of simple deuteranomalous trichromats inferred from color matching.

Deuteranomalous trichromacy is the most common form of inherited color-vision deficiency. A modern description of its cause is a single abnormality: the normal middle-wave cone photopigment (M) is replaced by a shifted middle-wave pigment (M) that is shared by all deuteranomalous trichromats. This explanation, however, fails to account for the individual differences in color vision observed even within the sub-group of deuteranomals with good chromatic discrimination. An ensemble of color matches is used here to test whether these individual differences reflect differences in the wavelength of peak sensitivity (lambda max) of individual deuteranomals' cone photopigments. The results show variation in both the lambda max and the effective optical density of their cone pigments. The individual differences found in lambda max are in accord with recent molecular biological research that shows individual differences in the genes thought to encode deuteranomalous photopigments.

Adult↗

PubMed1990

The role of small-field tritanopia in two measures of colour vision.

The present work extends the findings of previous efforts examining the comparability of current colour-screening tests. Several popular tests are shown to differ greatly in the performance exhibited by colour-normal observers as well as in their differential sensitivity to experimental manipulations of viewing duration and viewing distance. Those tests designed to identify yellow-blue dichromacy are especially sensitive to the manipulation of viewing distance, which is interpreted as reflecting 'small-field tritanopia' and the asymmetry in retinal density of the three cone types. These findings are discussed in terms of factors that influence the comparability of current colour-screening devices and the particular need for close adherence to standardized conditions with such instruments.

Color Perception↗

PubMed1989

Note on color preference and color vision test performance.

The incidence of color deficient vision was investigated using the Pseudo-Isochromatic Plates on a relatively large and representative group. In the sample of 112 adults aged 20 to 80 yr. and comprised of 53% women and 12% minorities, 8% of men and 3% of women were color deficient. Over-all performance indicated no effects for sex or race. Nearly half of the plates were nondiscriminating among sex, minority/majority, and "normal" and "defective" color vision groups. Named color preferences within the "normal" group strongly favored blues and reflected no sex differences.

Adult↗

PubMed2005

Colour vision in migraine: selective deficits for S-cone discriminations.

Three studies are reported that explore colour perception in migraine. In each, sensitivity for colours detected selectively by the S-cones and the L- and M-cones was assessed separately. The first study assessed the discrimination of small colour differences using the Farnsworth-Munsell 100-hue test. The second assessed threshold detection for purple, yellow, red and green targets on five equiluminant background colours. The third examined supra-threshold colour scaling using two colour series, purple-yellow and red-green. Each study indicated that differences in colour perception between migraine and control groups were restricted to colours detected by the S-cones, there were no differences in performance for colours detected by the L- and M-cones. The results are discussed in terms of possible pathologies in the early visual pathways.

Adolescent↗

PubMed1994

[Methods for screening and surveillance of diabetic retinopathy].

PURPOSE: The aim of this study is to investigate the various tests which allow to detect and follow-up diabetic retinopathy (DR). METHODS: Sixteen patients without DR or with background retinopathy underwent, once every six months: a full ophthalmologic investigation; a fluorescein angiography; a color vision test; a central visual field investigation. RESULTS: The impairment of angiography preceeded damage of eye fundus in 27% of cases. Deficiency of color vision and visual field were found in 57% of cases and in 35% respectively. These preceeded the appearance of angiographic DR in 50% of cases and 32% of cases respectively. CONCLUSION: The importance of such tools in the evolution of DR especially in young diabetics is discussed.

Adult↗

PubMed1992

Detection of colour vision abnormalities in uncomplicated type 1 diabetic patients with angiographically normal retinas.

Colour vision function was assessed in 38 non-complicated type 1 diabetic patients in whom fluorescein angiography was normal, and was compared with that in 36 age-matched, non-diabetic controls. All of the patients were healthy and none were taking medication except insulin. The eye examination, which was normal in every patient, included the Ishihara and City University tests, measurement of Snellen acuity, slit-lamp examination, tonometry, and fundal photography as well as fluorescein angiography. Colour discrimination ability was measured with the Farnsworth-Munsell 100-hue test. Mean (SE) 100-hue test error score for the diabetic group was 86.8 (8.1) compared with 28.2 (3.3) for controls, p<<0.001. There was no relation between colour vision abnormalities and diabetes duration (r = 0, p>0.05), blood glucose at the time the colour tests were performed (r = 0.4, p > 0.05), most recent glycated haemoglobin result (r = 0.3, p>0.05), or the mean of all previous glycated haemoglobin results (r = 0, p>0.05). It is concluded that colour discrimination may be abnormal in uncomplicated type 1 diabetic patients before the onset of retinopathy, and that colour discrimination losses in diabetes may not be of vascular aetiology.

Adult↗

PubMed1995

Color vision and color pattern visual evoked cortical potentials in a patient with acquired cerebral dyschromatopsia.

We examined a 74-year-old man because of difficulty seeing green and the presence of prosopagnosia. His visual acuity was 0.8 in both eyes. He was not congenitally color blind, and there was no family history of color blindness. A left superior homonymous quadrantanopsia was found. The dyschromatopsia ws identical in both eyes. The patient showed red-green deficiency on testing with Ishihara plates a deutan defect with Tokyo Medical College plates, strong blue-yellow defects and medium red-green defects with Standard Pseudochromatic Plates II and a tritan defect with the Panel D-15. He failed the New Color separation test with scores of 160 and could not carry out the Farnsworth-Munsell 100-hue test, but his color naming test results were normal. Visual evoked cortical potentials to black-and-white checkerboard and color pattern reversal (Red and Blue-Green, Green and Red-Purple, Purple and Yellow-Green: isochromatic paired checks) stimuli were normal. Bilateral inferior occipital lesions were found by computed tomography and T2-weighted magnetic resonance imaging. Our findings suggested that luminance and color channels up to area 17 in our patient were intact. We believe that our patient's acquired cerebral dyschromatopsia is rare.

Aged↗

PubMed1990

Dyschromatopsia in heroin addicts.

The Farnsworth-Munsell 100-hue test was employed to determine whether there was any defect of colour vision in 29 confirmed male heroin addicts who had been successfully detoxified. Forty age-matched males served as controls. A typical normal error score on the FM test is about 40 and an error score of over 100 indicates poor colour discrimination; 86.2% of the eyes of the control group had an error score below 100 while only 17.2% of the eyes of heroin addicts had an error score below 100. The colour confusion among the heroin addicts was in the blue-purple (475-495 mu) range. These results indicate that colour vision defects are more common in heroin addicts.

Adult↗

PubMed1979

Lanthony's new color test--part III. The neutral zone.

With the New Color Test (NCT) the neutral zone was studied in hereditary and acquired dyschromatopsias. Acquired type III blue-yellow defects with neutral zone occurred in heredo-atrophies of the optic nerve, in edematous maculopathies, in choroidal atrophy, in myopia, in glaucoma and in retinitis pigmentosa. With exclusion of the autosomal dominant inherited cases, the type III neutral zone in retinitis pigmentosa becomes complicated by a type I neutral zone when the visual acuity dropped to 0.2.

Choroid↗