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At least 523 records · Page 29Linked to original sources
PubMed1987

The effect of season on the circulating concentrations of anterior pituitary, ovarian and adrenal cortex hormones and hormone binding proteins in the subarctic area; evidence of increased activity of the pituitary-ovarian axis in spring.

To evaluate the effects of season on the function of the pituitary-ovarian axis and the adrenal cortex in a northern area with great seasonal variation in the length of daylight, 10 healthy women were investigated over 1 menstrual cycle in spring (May-June), autumn (August-September), early winter (November-December) and late winter (February-March). Serum concentrations of LH, FSH, prolactin, estradiol, progesterone, total and free testosterone, cortisol, sex hormone binding globulin (SHBG) and cortisol binding globulin (CBG) were measured, and the indices of free estradiol (FEI), free androgen (FAI) and free cortisol (FCI) were calculated on cycle days 3-4, 6-7, 10-11, on the presumed day of ovulation, and 6-7 and 9-10 days after the presumed ovulation. Spring was the season that most significantly differed from the other seasons. It was characterized by a significantly decreased concentration of SHBG and an increased FAI throughout the whole menstrual cycle, an increased FSH concentration during the follicular phase, significantly increased estradiol concentration and an increased FEI, and significantly decreased concentrations of FSH and LH during the luteal phase of the cycle. The concentration of cortisol and the FCI were significantly increased in the autumn compared with late winter, both seasons having similar day-length. The present data demonstrate that spring, with a long photoperiod, seems to be associated with increased pituitary-ovarian axis activity and androgenic activity, whereas adrenal cortex function did not show any association with day-length.

Adrenal Cortex Hormones↗
PubMedDate not supplied

[Effect of adrenal cortex hormones on the ultrastructure of rat dermal fibroblasts in postnatal ontogeny. Electron microscopy morphometric data].

The effects of hydrocortisone and acetate deoxycorticosterone on the composition of the fibroblast population and the changes in their ultrastructure during the derma development in ontogenesis were studied using the electron morphometry. Hydrocortisone was shown to decrease in the derma of young animals the number of fibroblasts, which actively produce collagen, and to increase the number of fibrocytes and degenerating cells. Hydrocortisone reduced both in young and adult animals the total extent of the rough endoplasmic reticulum membranes, the total area of the rough endoplasmic reticulum and Golgi complex cisternae at the fibroblast section, as well as the area of the cytoplasm. The introduction of acetate deoxycorticosterone increased insignificantly the number of fiblroblasts, which actively produce collagen, in the derma of 2 month old animals. Under its influence the ultrastructure of the fibroblasts changed reliably only at the later stages of ontogenesis. Both the corticosteroids changed the bulk ratios of various organelles in the fibroblast's cytoplasm.

Adrenal Cortex Hormones↗
PubMed1977

[Role of adrenal cortex hormones in the realization of the biological action of cholerogen].

Experiments were conducted on albino mice, guinea pigs, and rabbits. The role played by glucocorticoids in the realization of the biological action of cholerogen--cholera exotoxin (edema of the limbs in mice and the skin test after Craig in guinea pigs and rabbits) was studied. Tissue sensitivity of these animals to the action of cholerogen diminished under the effect of stress factors (intramuscular injection of formalin, intraperitoneal injection of cholerogen, immobilization, hunger, laparatomy, etc.) and in case of glucocorticoid administration. A hypothesis is put forward that cholera exotoxin entering the blood from the intestine and activating the adrenal cortex function in vivo promoted the development of cholera intoxication.

Adrenal Cortex↗
PubMed1991

The effect of adrenal cortex hormones on heart and skeletal muscles amylases activity under conditions of alpha- and beta-adrenergic stimulation.

Influence of hydrocortisone (H), deoxycorticosterone (D), phenylephrine (F) and isoprenaline (I) on activity of alpha amylase (AA) = 4-glucohydrolase alpha-1, 4-glucan [3, 2, 1, 1] and glucoamylase (GA) = glucohydrolase alpha-1, 4-glucan [3, 2, 1, 3] was investigated in heart and skeletal muscle in rats, before and after adrenalectomy (AT). It was found that AT inhibits a rise in the activities of AA and GA caused by F in both investigated muscles, whereas the I given after AT loses its ability to inhibit GA activity in heart and skeletal muscle. Both H and D administered after AT recover the ability of F to an increase in the investigated enzyme activity whereas recovers I ability to inhibit amylase activity in the presence of H. It can be concluded that inhibition of amylase activity is muscles results from interaction between beta-adrenergic stimulation with glucocorticoids, while the increase in activities of these enzymes is a result of interaction between alpha-adrenergic stimulation with mineral tropic action of both D and H.

Adrenal Cortex↗