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At least 523 records · Page 29Linked to original sources
PubMed1990

[Clinico-morphological evaluation of pathogenetic mechanisms of bouginage and intubation on cicatricial esophageal stenosis after chemical burns].

The article deals with the clinico-morphological appraisal of bougienage and intubation of patients with cicatricial constriction of the esophagus after chemical burns. Endoscopic and morphological studies were carried out in 38 patients before and during treatment. Bioptic material collected from the esophageal mucosa was studied on a light-optic and ultrastructural level. The results of the study showed that bougienage of the epithelialized strictures causes protracted stimulation of intracellular regenerative processes and thus facilitates the development of leukoplakia of the squamous epithelium. The prevention of esophageal precancer in this case consists in radical maintenance bougienage and regular medical examination. Bougienage of ++non-epithelialized strictures stimulates the formation of young granulation tissue, while subsequent intubation stimulates reparative processes, creating in this manner conditions for adequate epithelialization of the ulcerated esophageal strictures.

Burns, Chemical↗
PubMed2002

Esophageal stenosis in childhood: dystrophic epidermolysis bullosa without skin blistering due to collagen VII mutations.

We report a 9-year-old girl who experienced recurrent dysphagia since infancy. Crohn's disease was suspected because she had aphthous ulcers of the mouth and anal dermatitis with hematochezia. After bougienages of esophageal stenoses and medication for inflammatory bowel disease proved unsuccessful, interdisciplinary re-examination revealed the cause of the symptoms to be an extracutaneous form of dystrophic epidermolysis bullosa, a genetic skin fragility disorder. Dystrophic epidermolysis bullosa is caused by mutations in the COL7A1 gene encoding collagen VII, a protein of the epidermal attachment complex, and typically manifests with trauma-induced skin blistering, scarring, nail dystrophy, and, in some cases, mucosal involvement. The present proband never developed skin blisters but had nail dystrophy and erosions of the oral, esophageal, and genitoanal mucosa, which healed with slight scarring. Mutation analysis disclosed compound heterozygosity for recessive mutations in the COL7A1 gene. The paternal mutation 425 A-->G caused abnormal splicing resulting in a premature stop codon. The maternal mutation G2775S led to the substitution of a glycine by a serine in the triple helical domain of collagen VII. This case shows that mucosal disease and esophageal strictures in childhood are not always acquired, but can also represent a genetic defect of dermal-epidermal adhesion, even in the absence of skin blistering.

Biopsy↗
PubMed2003

[Balloon dilatation in esophageal stenosis].

The most frequent causes of the stenosis are as follows: Schatzki rings, structures as the complications of GERB', postcorrosive and postoperative at nosis. The Balloon dilatation is one from the therapeutic procedures of stenosis of the esophagus. By the clinic research is included 12 patients with the stenoses of the esophagus of the various cause. It is used the balloon dilatator (Olympus) of the diameter 16 and 18 mm, and the analysis 40 mm. In 11 patients the balloon dilatation was successful and led to the walking of the esophagus for endoscope. Only in one patient the dilatation was unsuccesful, because was applied two years after the occurrence of the stenosis caused by the corrosive esophagisitis. It is concluded that the balloon dilatation of the therapeutic procedures of the choice in stenosis of the esophagus.

Adult↗
PubMed1999

[An unusual esophageal stenosis].

In the submitted case-history the authors describe the case of a child operated after delivery on account of oesophageal atresia type Vogt IIIb. After a two-year interval the child returned on account of acute dysphagia and endoscopy revealed oesophageal stenosis caused by a mucosal strangulating bridge. The latter was removed endoscopically and during a check-up examination 6 months later the endoscopic picture was negative.

Child, Preschool↗
PubMed1975

Radical surgical treatment of esophageal stenosis due to epidermolysis bullosa.

Stenotic lesions of the esophagus in epidermolysis bullosa are a rare cause of dysphagia. Even though 50 cases have been reported in the literature, only recently has surgical treatment been suggested for these patients. In our patients, who had an annular stenosis just distal to the pharyngoesophageal juncture, resection of a mucosal cylindrical segment with reanastomosis through a longitudinal esophageal myotomy was successful. This method of treatment is suggested for patients in whom the stenosis is localized.

Adult↗
PubMed1999

[Benign proximal esophageal stenosis--mostly a complication of gastroesophageal reflux disease].

BACKGROUND AND OBJECTIVE: Benign stenoses can occur anywhere in the oesophagus, but are most common in its distal part as a result of gastro-oesophageal reflux (GOR). It was the aim of this study to evaluate retrospectively the causes and incidence of benign stenosis of the proximal oesophagus (SPR) as well as its endoscopic and drug treatment. PATIENTS AND METHODS: Between December 1989 and December 1997 a total of 17,413 patients were referred to the authors' hospital for oesophago-gastroduodenoscopy, 1024 of them (6%) for clarification of heartburn, regurgitation and/or dysphagia. 53 of these patients (5%) were found to have benign stenosis of the oesophagus requiring bougie dilatation, located in the lower third in 29 (55%), in the middle third in six (11%) and in the upper third in 18 (34%) patients. Causes of stenosis in the upper third were peptic stricture in nine (50%), heterotopic gastric mucosa in three (17%), caustic corrosion in three (17%), post-radiation in two (11%), and the result of web formation in one (6%). Endoscopic bougie dilatation was performed in all these patients, those with GOR additionally receiving 40 mg omeprazole daily. RESULTS: In those patients with nonpeptic benign stenosis/stricture lasting improvement of symptoms was achieved with one to three dilatation. But those with GOR needed a mean of 13 dilatations during a follow-up period averaging 61 months. Barrett's oesophagus (replacement of squamous by columnar epithelium) was found in five patients. No case of dysplasia was discovered. Laparoscopic fundoplication was performed in one woman in whom bougie dilatation had failed. Remission was maintained, as needed, by bougie and omeprazole in eight patients. CONCLUSION: In benign stenosis of the upper oesophagus endoscopic dilatation is the treatment of choice. In cases of peptic aetiology the administration of proton pump inhibitors is the optimal adjuvant method.

Anti-Ulcer Agents↗
PubMedDate not supplied

Repair of traumatic cervical esophageal stenosis using microvascular free jejunum transfer.

Three cases of nonneoplastic stricture of the cervical esophagus and pharynx repaired by free jejunal transfer are reported. This technique permits repair of various circumferential defects of the pharynx and esophagus in a single stage without compromising laryngeal function. None of the patients required subsequent dilatation to restore satisfactory swallowing.

Adolescent↗