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At least 55 records · Page 3Linked to original sources
PubMed1981

[Projections of the amygdaloid body to the striopallidal structures of cats].

Horseradish peroxidase (HP) was injected (0.2-0.3 mkl, 40%) into the cat striopallidal nuclei. After treating the brain and its sections according to Messulam's technique, it was possible to reveal HP-positive neurons in some structures of the corpus amygdaloideum. When HP was injected into the caudate nucleus, the labelled neurons were found in the basal nucleus of the corpus amygdaloideum. When injected into the globus pallidus--the labelled neurons were revealed in the macrocellular part of the basal nucleus and in the anterior amygdaloid field. Owing to the data obtained, it is possible to suggest that not only the anterior amygdaloid field and the basal nucleus have projections on the putamen, but the medial, central and lateral nuclei of the corpus amygdaloideum, as well.

Amygdala↗
PubMed1984

[Comparative effects of lesion of the basolateral nucleus and lateral nucleus of the amygdaloid body on neophobia and conditioned taste aversion in the rat].

The effects of bilateral lesions of the basolateral and lateral nuclei of the amygdala on the neophobic response and LiCl-conditioned taste aversion to a saccharin solution were studied in rats. Compared to intact animals, rats with basolateral lesions did not exhibit neophobia to the novel stimulus, while rats with lateral lesions demonstrated an initial preference to the sweet solution over water. The LiCl-induced aversion was suppressed after basolateral lesions and was unchanged after lateral lesions. It is concluded that these two amygdaloid nuclei play an important but distinct role in neophobia and conditioned taste aversion.

Amygdala↗
PubMed2006

Increased volume and neuronal number of the basolateral nuclear group of the amygdaloid body in aggressive dogs.

Involvement of the basolateral nuclear group (BNG) in pathological canine aggression was assessed by stereological determination of the volume of the BNG and quantification of the numerical density and total number of BNG neurons in normally behaving and aggressive dogs. A bilateral BNG enlargement of 40% was observed in the aggressive group. This enlargement appeared to be caused by a significantly increased number of BNG neurons. Other alterations such as an increased vessel density, oedema and scar tissue were not observed in any of the examined BNGs. The potential role of neurotrophins and stress hormones in the increased number of BNG neurons is discussed.

Aggression↗
PubMed2003

Antioxidant ability and lipid peroxidation in the hippocampus with epileptogenesis induced by Fe3+ injection into the amygdaloid body of rats.

To analyze antioxidant ability and lipid peroxidation in the hippocampus of rats in an interictal state of FeCl3-induced epileptogenesis, the hippocampal eliminating decay ratio of exogenously applied nitroxide radical (3-carbamoyl-2,2,5,5-tetramethylpyrrolidine-1-oxyl (carbamoyl-PROXYL)) by electron paramagnetic resonance (EPR) spectroscopy, and the thiobarbituric reactive substances (TBARS) level in the hippocampus were measured. The prolonged half-life of electron paramagnetism of carbamoyl-PROXYL in the hippocampus of rats with chronic FeCl3-induced epileptogenesis revealed decreased antioxidant ability, which supports the vulnerability against oxidative stress. In addition, TBARS level (marker of lipid peroxidation) was increased in the hippocampus of rats injected with FeCl3 compared with that of control. This study revealed that repetitive seizures resulted in the decreased hippocampal antioxidant ability with lipid peroxidation and explained the regional vulnerability to oxidative stress in the limbic system with epileptogenesis.

Amygdala↗