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At least 55 records · Page 3Linked to original sources
PubMed1958

Cardiac output during excitation of chemoreflexes in the cat.

In cats under chloralose anaesthesia the reflex fall of blood pressure and heart rate caused by injection of veratrine, amidines, diphenhydramine, or ethyl acetoacetate was accompanied by a fall in cardiac output. After veratrine and amidines there was a fall in mean pulmonary arterial pressure and after veratrine no significant change in pulmonary vascular resistance. After diphenhydramine and ethyl acetoacetate there was a rise in mean pulmonary arterial pressure and after diphenhydramine an increase in pulmonary vascular resistance. The effects of veratrine and amidines, but not those of diphenhydramine and ethyl acetoacetate, were abolished by section of the vagi. The main change leading to the fall of cardiac output after amidines was bradycardia.

Animals↗
PubMed1958

Activation energy of ventricular contraction in anionicaliy modified solutions.

The activation energy of ventricular contraction and relaxation in chloride and nitrate-Ringer's solution was studied on the driven rat ventricle strip by measurement of the specific contraction and relaxation velocities over a 17 degrees C range of temperature. Nitrate ion produced a small but significant reduction in the activation energy of contraction.

Animals↗
PubMed1959

Release of autonomic mediators in cardiac tissue by suprathreshold stimulation.

Pharmacological evidence is presented supporting the theory that the increase in contractile strength of isolated cardiac muscle under suprathreshold stimulation is due to the release of an adrenergic mediator (norepinephrine). However, release of this material does not account for changes in contractile strength associated with changes in frequency of stimulation at threshold levels.

Autonomic Nervous System↗