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Results for “DOMINANCE, CEREBRAL”

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At least 55 records · Page 3Linked to original sources
PubMed1987

Performance on a free-recall verbal dichotic listening task and cerebral dominance determined by the carotid amytal test.

Epileptic patients whose speech dominance had been ascertained using the carotid amytal technique were given a verbal dichotic listening test. Patients with left hemisphere speech dominance, like normal controls, showed a REA. Patients with right hemisphere speech representation, as a group, tended not to show a bias toward either ear. Moreover, repeated administrations of the verbal dichotic listening test yielded similar results.

Adolescent↗
PubMed1990

Tachistoscopic presentation of verbal stimuli for assessing cerebral dominance: reliability data and some practical recommendations.

Reliability data point to rather high test-retest correlations (greater than or equal to 0.65) for VHF data with four- and five-letter words as stimuli, but replicate previous findings that the first test score correlates poorly with later test scores. The same results are obtained for accuracy and latency data, though small differences exist. All laterality indices lead to the same conclusions and have high intercorrelations. The point-biserial correlation coefficient is, however, a slightly more reliable index of naming latency than the mere difference between LVF and RVF. No such superiority is found for the indices based on accuracy data. The results also point to the need to present a sufficient number of stimuli before firm conclusions can be drawn.

Adult↗
PubMed1983

An assessment of cerebral dominance in language--disordered children via a time-sharing paradigm.

A time-sharing paradigm was used to assess language lateralization in language-disordered and normal children aged 4-7 years. Several expressive language tasks as well as a vocal, but nonlinguistic, task were administered concurrently with unimanual finger tapping. Dependent variables were percent disruption scores and number of syllables produced per concurrent trial. All language concurrent tasks produced tapping reductions for both hands for both groups. This result contrasts to similar time-sharing studies claiming asymmetrical interference and hence language lateralization in children (N. White & M. Kinsbourne, 1980, Brain and Language 10, 215-223; J. Obrzut, G. Hynd, A. Obrzut, & J. Leitgeb, 1980, Brain and Language 11, 181-194). A multiple regression analysis revealed a significant interaction effect differentiating language-disordered from normal children. Normals exhibited a parallel response pattern for speech and tapping (both increased or decreased in rate) under all lateralization conditions. Language-disordered children exhibited an inverse response pattern (e.g., if speech output increased, tapping rate decreased) only under left-hemisphere time-sharing.

Child↗
PubMed1996

Ideomotor apraxia and cerebral dominance for motor control.

Ideomotor apraxia is a symptom of left hemisphere damage. Patients with ideomotor apraxia commit errors when imitating movements with their left, non-paralyzed hand. This has been taken as evidence for a motor dominance of the left hemisphere. It has been hypothesized that the left hemisphere is dominant for internal preprogramming of skilled movements of either hand. We investigated the kinematics of movement trajectories of imitation of meaningless gestures. Group analysis confirmed that hesitant, feedback-controlled movement prevail in patients with apraxia, but analysis of single cases revealed the existence of kinematically normal movements leading to apractic errors. Enhanced reliance on feedback-control appears to be a compensatory strategy rather than the source of apractic errors. In a second study we explored the alternative hypothesis that patients with apraxia lack a general concept of the human body which is necessary to mediate the translation of a target position seen on the model into a target position on the patient's body. Imitation of movements was examined on oneself and on a mannikin. Patients with apraxia who made errors when imitating on themselves committed errors also when imitating on the mannikin. Taken together, both studies support the view that the source of errors in the imitation of gestures is to be sought at a conceptual level. This casts doubts on the alleged dominance of the left hemisphere for motor control.

Apraxias↗