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At least 55 records · Page 3Linked to original sources
PubMed1982

[Peptic esophageal stenosis and its relation to reflux esophagitis and esophageal cancer].

Carcinoma of the lower oesophagus can be a complication of a peptic oesophageal stricture caused by reflux oesophagitis. In most cases there is also a hiatal hernia. The main symptoms are dysphagia and epigastric retrosternal pain. The most important examinations are x-ray contrast film of the oesophagus and oesophagogastroscopy combined with biopsy.

Diagnosis, Differential↗
PubMed2006

[Application of portable esophageal dilator in treatment of esophageal stenosis at home].

OBJECTIVE: To explore an economical protocol for esophageal stenosis treatment at home and evaluate the effects of portable esophageal dilator. METHOD: Sixty-seven patients with esophageal stenosis were treated at home by themselves with portable esophageal dilator, which was designed by Prof Wang HT, one of the authors. Twenty-nine cases of esophageal stenosis were caused by chemical damage, 23 cases by esophageal tumor, 13 postoperation cases with hypopharyngeal and upper esophageal tumor and two cases by laryngeal thyroid gland recurrent tumor. Stricture happened at esophageal entrance and upper segment in 15 cases, at middle in 32 cases, both at middle and upper in 17 cases and at whole length of esophagus in three cases. Stricture was not complete atresia and dilators could insert into esophagus in all patients. RESULT: Significant effects were found in 43 patients and good effects in 24 patients. Seven deaths were caused by remote metastasis of recurrent cancer resulting in multiple organ failure or large blood vessels bleeding resulting of tumor erosion. Follow-up visit ranged from the shortest period of four months to the longest period of five years and 1 month, 9.8 months on average. CONCLUSION: Family portable dilator and self-catering dilation is an effective and safe method for treating esophageal stenosis. It is so simple, convenient, painless with low expense that it is especially useful in countryside hospitals and undeveloped rural areas.

Adolescent↗
PubMed1989

[Surgical treatment of esophageal stenosis with recessive dystrophic epidermolysis bullosa--esophageal dilatation using balloon catheter method].

We report the details of our treatment of 5 patients with recessive dystrophic epidermolysis bullosa (RDEB). Good results were obtained by esophageal dilatation using the balloon catheter method. Prior to the operation, we corrected the patients' nutritional status with oral supplementation of Hinex-R as well as intravenous drip infusion of Intra-lipid, based on the nutrition index obtained by body weight, serum albumin, rapid turnover proteins, and blood minimum essence. After sufficient anesthesia of the nasopharyngeal mucosa with xylocain spray, we inserted a Microvasive Rigiflex Balloon Dilator into the stenotic region while scanning the upper GI. Then we dilated gradually by barium injection. The patients' condition improved after this treatment and follow up X-ray examination revealed no recurrence of stenosis. There were several advantages to this method: 1) It was safer and could be done repeatedly. 2) It caused less stress for patients. 3) Its effectiveness appeared rapidly. We analyzed the frequency and severity of esophageal stenosis of 12 cases with EB in our department. Ten out of 12 cases complained of subjective esophageal symptoms. Esophageal stenosis was observed in all 8 cases examined by esophagogram. We concluded that this surgical procedure for esophageal stenosis was as important as digit reconstruction in the management of RDEB.

Adolescent↗
PubMed1997

[A new surgical management of esophageal stenosis without esophagectomy].

In many cases, reflux esophagitis following surgical treatment for esophageal stenosis is caused by the recurrence of that after esophagectomy and esophogogastrostomy. We performed a new management without esophagectomy for a 66-year-old man with sliding hiatal hernia and esophageal stenosis induced by reflux esophagitis. A Expanding Metalic Stent (MES) was inserted to the stenotic portion of the esophagus, and then Collis-Nissen's procedure was done through left thoracotomy and phrenotomy. The postoperative course was satisfactory, and no gastroes-ophageal reflux was detected with the use of 24h pH-monitoring of the esophagus after surgery.

Aged↗
PubMed2003

Restenosis following balloon dilation of benign esophageal stenosis.

AIM: To elucidate the mechanism of restenosis following balloon dilation of benign esophageal stenosis. METHODS: A total of 49 rats with esophageal stenosis were induced in 70 rats using 5 ml of 50% sodium hydroxide solution and the double-balloon method, and an esophageal restenosis (RS) model was developed by esophageal stenosis using dilation of a percutaneous transluminal coronary angioplasty (PTCA) balloon catheter. These 49 rats were divided into two groups: rats with benign esophageal stricture caused by chemical burn only (control group, n=21) and rats with their esophageal stricture treated with balloon catheter dilation (experimental group, n=28). Imaging analysis and immunohistochemistry were used for both quantitative and qualitative analyses of esophageal stenosis and RS formation in the rats, respectively. RESULTS: Cross-sectional areas and perimeters of the esophageal mucosa layer, muscle layer, and the entire esophageal layers increased significantly in the experimental group compared with the control group. Proliferating cell nuclear antigen (PCNA) was expressed on the 5th day after dilation, and was still present at 1 month. Fibronectin (FN) was expressed on the 1st day after dilation, and was still present at 1 month. CONCLUSION: Expression of PCNA and FN plays an important role in RS after balloon dilation of benign esophageal stenosis.

Animals↗
PubMed1992

Congenital esophageal stenosis due to tracheobronchial remnants: a rare but important association with esophageal atresia.

Congenital esophageal stenosis caused by tracheobronchial remnants occurred in eight children, six of whom had associated esophageal atresia and/or tracheoesophageal fistula. Symptoms usually began in early infancy but delayed diagnosis was a common feature. The mean lag period between presentation and definitive operation was 4.6 years (range, 1 month to 16 years). Errors in diagnosis were common. Six were initially diagnosed as having inflammatory strictures secondary to reflux esophagitis. Seven children were subjected to repeated esophagoscopy and bouginage of the "stricture" (mean no. = 3.4), with invariable failure to ameliorate dysphagia. Antireflux procedures were performed in three patients. In all children, symptoms were dramatically relieved following resection of the stenotic segment or esophageal replacement. Although a rare entity, congenital esophageal stenosis due to tracheobronchial remnants should be considered a possibility in patients with esophageal stricture, presumed to be inflammatory in nature, which fails to respond to standard therapy.

Adolescent↗
PubMed1998

Esophageal stenosis in children.

This article focuses on the special features of esophageal stenosis which pertain to children. In order to focus on stenoses intrinsic to the esophagus, esophageal stenosis due to extrinsic compression is excluded. While the causes of esophageal stenosis may be grouped as either congenital or acquired, congenital causes account for less than 5% of cases.

Child↗
PubMed1989

[Treatment of esophageal stenosis in children].

Esophageal stenoses, both congenital and acquired, represent a serious problem in childhood due to the deleterious effects, of these anomalies in the patients nutritional state and lifestyle. Choosing the best way to the solution of such problems is of primary importance, paying special attention to the techniques that may not compromise irreversibly the continuity of the alimentary tract. The authors experience on the treatment of 77 esophageal stenoses, 8 of which were congenital and 69 acquire, is reported. Of this group, 24 are sequelae of esophageal atresias, 33 are of caustic origin and 12 of peptic nature. In 3 congenital stenoses with a cartilaginous matrix, surgical repair was undertaken. In the remaining cases forward and retrograde dilatation guided by a thread, through the gastrostomy orifice were performed, in accordance to the length and severity of the stenoses. Duration of the treatment varied broadly, ranging from 3-5 anterograde sessions, in the cases of esophageal atresia sequelae, to several years with an average monthly periodicity of one session in cases of total pharyngoesophageal caustic burn. As for complications, there were three cases of mediastinitis and two of subcutaneous infiltrations at the cervical level that were solved with antibiotics. The authors believe, on the basis of their experience, the absence of mortality and the scarce morbidity that, in the pediatric patients the treatment of choice of esophageal stenoses consists of dilatations on their different modalities. A yearly follow-up must be strictly maintained until the patients have completed their growth. We reject esophageal coloplasties in the pediatric age because of their complications rate.(ABSTRACT TRUNCATED AT 250 WORDS)

Age Factors↗
PubMed1996

[Gore-tex covering of ultraflex stent and its usefulness for malignant esophageal stenosis: preliminary clinical results].

Malignant esophageal stenoses develop esophagorespiratory fistulae or perforations so frequently that esophageal stents must be required. We devised a covered stent with a thin Gore-Tex sheet and a nitinol stent system, Ultraflex, without increasing the size of the introducer assembly. Nine patients with malignant esophageal stenoses, including seven patients with perforation or fistulation, were carefully treated with the covered stent. All stent deployments were successfully carried out under fluoroscopic guidance. The average time required for full self-expansion was two weeks. The average grade of dysphagia improved from 3.7 to 1.2. Clinical symptoms due to esophagorespiratory fistulae were improved in three of four patients. No clinical or technical complications, such as migration, were observed, but a fistula developed at the bare site of the stent. This covering method for the flexible stent was simple and safe, and was considered to be useful in the treatment of malignant esophageal stenosis.

Adenocarcinoma↗
PubMed1989

[Long-term course of esophageal stenosis treated with pneumatic dilatation. A comparative study versus other methods].

Dilatation of benign esophageal strictures by means of a balloon catheter is a well established procedure in children. In this report a comparison of bouginage and balloon dilatation is made in eight patients treated by both procedures. The lesions treated included two strictures secondary to surgical anastomosis, one restrictive Nissen fundoplicature and five esophageal stenosis following caustic ingestion. These eight patients had a total of 44 bouginage procedures with a mean relapse free period of 80 days. There were two esophageal perforations. The clinical course was unsatisfactory in five cases. Only one patient had good results. In contrast, to date, the 15 balloon dilatations have resulted in a mean relapse free time of 174 days. There were no complications that required treatment. The clinical follow up shows four asymptomatics patients and the other four presenting difficulties to swallow with spontaneous resolution. We have the best results in younger patients with esophageal stenosis secondary to esophageal atresia repair. Balloon dilatation of esophageal stenosis in children is effective and safe, and should be considered before other methods of treatment are used.

Catheterization↗
PubMed1991

Probable congenital esophageal stenosis in a thoroughbred foal.

Esophageal stenosis was diagnosed in a 7-day-old Thoroughbred foal referred for evaluation of bilateral milky nasal discharge. Double-contrast radiography revealed concentric narrowing of the esophagus at the level of the fifth rib, overlying the base of the heart. Lateral displacement of the esophagus was not apparent. X-ray computed tomography of the cranial portion of the thorax confirmed normal cardiac and great vessel anatomy, ruling out vascular ring anomaly. A nasogastric tube was placed, extending past the stenosis and into the stomach, and the foal was fed mares' milk via this tube. On day 14, the foal was allowed to nurse the mare hourly and was made to stand, with the cranial half of its body elevated for 5 minutes after each feeding. On day 19, the foal was allowed to nurse ad libitum and eat hay and grain with the mare. Signs of regurgitation and aspiration were not apparent. On day 29, the foal was discharged to the owner.

Animals↗