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Results for “KIDNEY DISEASES/pathology”
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[Pathological anatomy of renal insufficiency].
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[Renal ptosis, anatomical study].
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[Study on congenital biliary fibroangiomatosis. Apropos of a case associated with a renal dysplasia].
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Renal tubular necrosis: its relation to norepinephrine administration.
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[Present knowledge concerning the correlation between the morphological and functional substrates in certain renal diseases].
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Renal amyloidosis: a clinicopathologic study of 21 cases.
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[Bilateral necrosis of the renal cortex. Study of a case as compared with the Shwarzman phenomenon].
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[Intrahepatic anatomo-functional changes after temporary interruption of the renal circulation].
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[Pathological anatomy of renal amyloidosis].
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[Relations between the functional capacity of the proximal tubule and the distal tubule in kidney diseases].
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[On nephrocalcinosis. Experimental research by electron microscope].
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[Retihal manifestations in the course of nephropathies].
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[Clinical and morphological aspects of renal amyloidosis and uremia].
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Electron microscopic studies of renal disease.
The nephrotic syndrome, glomerulonephritis, disseminated lupus erythematosus and the Fanconi syndrome show characteristic changes with electron microscopy. Experimental studies of animals were carried out to determine the significance of such changes by observing reactions that occur under carefully controlled conditions. A lesion with collagen deposition that was found in the centrolobular region of glomeruli sheds new light on the function of this region. This evidence must be considered in developing an understanding of how the production of urine is controlled. Fluid-filled compartments and various bodies associated with the ultrastructure of tubule cells can be produced under conditions which suggest that these structures play a role in tubular resorption.
[Amyloid nephropathies. Histological and electronic study. Apropos of 9 cases].
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[The glomerular basal membrane in glomerular nephropathies in the adult. Study by electron microscope].
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[The nephropathies caused by entombment in the earthquake at Agadir on the 1st of March 1960. Renal histopathological data on the fatal forms. Casablanca experience].
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