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A child affected by lack of eruption of the upper right primary cuspid from the presence of a compound odontoma was followed from 3 to 13 years of age. The early removal of the obstacle to eruption and the correct control of the case evolution allowed for a satisfactory alignment of the permanent teeth by means of minimal therapeutical intervention.
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Dens in dente (dens invaginatus, tooth within a tooth, dilated composite odontome) is an abnormal tooth form which occurs most frequently in the permanent maxillary lateral incisor region. Dens in dente may occur, however, in any tooth in the dental arch, although these other forms are comparatively rare. It may appear within both the coronal part of the tooth and the root, although coronal forms are more common. In this paper, a case of extreme dens in dente with pulpal involvement at an early stage of eruption is presented.
This paper describes the ultrastructure of the affected enamel and the clinical features in two siblings with the syndrome of nephrocalcinosis and amelogenesis imperfecta. Nephrocalcinosis was diagnosed by intravenous pyelography, and confirmed by ultrasonography and CT scan. Amelogenesis imperfecta AI was diagnosed clinically and histologically. Light microscopy showed that the affected enamel surfaces were rough and the enamel was hypoplastic and mainly positively birefringent. Scanning electron microscopy revealed a rough and extensively cracked enamel surface covered with oval shaped blister-like protrusions. TEM showed porous enamel consisting of loosely packed and randomly oriented thin ribbon-like crystals with little or no prismatic structure. Observations showed that hypoplasia together with hypocalcification and/or hypomaturation defects were present in the same tooth, indicating the possibility of an abnormality in interstitial matrix, leading to dystrophic calcification in the kidney and abnormal tooth enamel formation, or alternatively an involvement of two separate but closely linked genes.
In this report, a case of a mature tooth with abnormal crown morphology, which created plaque retentive areas was presented. The pulp was necrosed due to the periodontal disease, and the anomaly was thought to be an aberration of a talon cusp. Therapy involved the reshaping and the endodontic treatment of tooth.
There tooth abnormalities can be classified into four major groups, depending on the depth of the invagination. A typical example from group four is described. Thorough röntgenological examination reveals a frequent occurrence. The great diversity in nomenclature and the variety of morphogenetic and etiological theories show that genetic factors are very important in the development of an invaginated tooth and that knowledge concerning the pathomechanisms is very inadequate. A detailed inspection of the inner tooth morphology on röntgennogram is necessary as the surface morphology generally does not reveal the existence of an invagination. The irreversible pathological evolution occurring when diagnosis is neglected. Should incite the practitioner to look for invaginations on every set of röntgenograms. Five thereapeutic techniques are described. If an appropriate endodontic technique is applied, a successful root canal filling can be achieved.
Forty-five patients with Williams syndrome (WS) were evaluated for oral abnormalities. The mean age of the patients was 9.25 years, the median age was 6.7 years, and the majority (62.2%) were male. Hypodontia was present in 11.1% of the patients. Abnormal tooth morphology was noted in 12.5% of the primary dentitions and 40.7% of the permanent dentitions. With the exception of the primary mandibular central incisors of males, all mesiodistal incisor crown dimensions were statistically significantly smaller when compared with norms (P < 0.05). At least one hypoplastic enamel defect was present in 9.4% of patients with primary teeth and in 18.5% with permanent teeth. No patients exhibited generalized enamel hypoplasia. More than half of the patients (59.1%) were both caries and restoration free, while only 13.6% presented with clinically active caries. Tongue thrusting was present in 67.7% of the sample, while more than 50% of the patients present with excessive interdental spacing. Patients exhibited a higher than normal prevalence of Class II and III occlusions, open and deep bites and anterior crossbites. No single dental finding was pathognomonic of WS, however two constellations of findings, each occurring in approximately one-third of the sample, were observed: 1) microdontia, anterior crossbite, tongue thrusting, and excessive interdental spacing, and 2) microdontia, deep or open bite, and excessive interdental spacing.
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The purpose of the present investigation was to describe the formation, maturation and eruption of the dentition, including supernumerary teeth in a sample of patients with cleidocranial dysplasia. The dentition was evaluated from orthopantomograms, intraoral radiographs, cephalometric films, surgically removed teeth and intraoral photographs in 19 patients (9 men, 10 women), aged 3.5 to 34 years. Formation of primary teeth was normal, whereas all patients but one had supernumerary permanent teeth. Frequency of supernumerary teeth ranged from 22% in the maxillary incisor region to 5% in the molar regions. Supernumerary teeth were formed lingually and occlusally to the normal teeth. Maturation of the primary dentition was normal, while permanent teeth were delayed from 1 to 4 yr. Supernumerary teeth were delayed about 4 years in relation to normal permanent teeth. Eruption of primary teeth was normal, whereas all patients had severe eruption problems of permanent teeth. It was hypothesized that the dental lamina for both primary and permanent dentition is normal, but does not resolve completely and therefore may form supernumerary teeth. Abnormalities of tooth morphology is related to inadequate space and arrested eruption. Delayed or arrested eruption is probably caused by diminished resorption of bone and of primary teeth and to the presence of multiple supernumerary teeth.
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Explore the source record for details and available documents.
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Explore the source record for details and available documents.
Twenty-three patients who had completed orthodontic treatment for the resolution of unilateral palatal canine impactions were examined 2.3 years (mean) after all appliances were discarded. The patients were divided into two groups on the basis of whether the surgical exposure was "light" or "heavy." The whole group was also divided according to the type of orthodontic movement that was carried out--"light" for tipping, extrusive, and rotating movements and "heavy" for root movements. Final position of the teeth was classified as ideal or incomplete if rotations or spaces were present. The results showed marked deteriorative changes where the surgery had been more radical and where the tooth movement involved active alteration of root position. No change due to abnormal tooth position was seen. It is suggested that, in these cases, surgical procedures be limited in scope and that exposure of the cementoenamel junction be avoided.
The Blood-Vessel Thrust Theory is a new hypothesis regarding the forces which produce the normal eruption of teeth, and the movement of 'nonerupted' teeth through bone away from their normal position in the jaws. It points out that the flow of blood through the vessels of the dental pulp, and of the tissues surrounding the tooth, must produce hydrodynamic and hydrostatic forces within the blood vessels, and that these forces have a resultant towards the tooth crown, thus causing the tooth to move, crown first, through the bone during normal eruption or abnormal tooth migration.