Particle size sedimentation of dental amalgam alloy.
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In a cell with artificial saliva the electrochemical behaviour of amalgam specimens of Germadent, Duragam and ANA 2000 is analysed in dependence of the surface treatments carving, burnishing and polishing using potentiodynamic polarization with low polarization rate. The less roughness and content of gamma-2-phase the better the amalgam reacts to corrosion evident in a higher potential, a lower corrosion rate and a higher breakdown potential. Foremost the polish supplies the best resistance to corrosion. Duragam-amalgam is more resistant than Germadent-amalgam. Polished specimens of Duragam react likely specimens of ANA 2000.
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Clinicians are frequently confronted with the problem of ditched amalgam restorations. Materials which develop strong bonds to amalgam and tooth substance appear to be suitable for repair of such restorations and eliminate the need for complete replacement. This study evaluated the bond strengths of three glass-ionomer cements to amalgam made from conventional and high copper alloys. It was found that the bond strength values to amalgam were comparable with those to enamel and significantly higher than those to dentine. All bonds to the high copper amalgam failed in a cohesive mode; for the conventional amalgam, failure was either of the adhesive or cohesive type.
Samples from the central nervous system (occipital lobe cortex, cerebellar cortex and ganglia semilunare) and kidney cortex were collected from autopsies and analysed for total mercury content using neutron activation analyses. Results from 34 individuals showed a statistically significant regression between the number of tooth surfaces containing amalgam and concentration of mercury in the occipital lobe cortex (mean 10.9, range 2.4-28.7 ng Hg/g wet weight). The regression equation y = 7.2 + 0.24x has a 95% confidence interval for the regression coefficient of 0.11-0.37. In 9 cases with suspected alcohol abuse mercury levels in the occipital lobe were, in most cases, somewhat lower than expected based on the regression line. The observations may be explained by an inhibition of oxidation of mercury vapour. The regression between amalgams and mercury levels remained after exclusion of these cases. The kidney cortex from 7 amalgam carriers (mean 433, range 48-810 ng Hg/g wet weight) showed on average a significantly higher mercury level than those of 5 amalgam-free individuals (mean 49, range 21-105 ng Hg/g wet weight). In 6 cases analysis of both inorganic and total mercury was carried out. A high proportion (mean 77% SD 17%) of inorganic mercury was found. It is concluded that the cause of the association between amalgam load and accumulation of mercury in tissues is the release of mercury vapour from amalgam fillings.