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Results for “Periapical Abscess”

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At least 685 records · Page 38Linked to original sources
PubMed1993

Factors associated with endodontic posttreatment pain.

One hundred six patients with pretreatment pain presenting for endodontic treatment participated in a controlled double-blind study of dexamethasone use. Endodontic therapy was performed and, after controlling for use of nonuse of dexamethasone, patient variables and treatment factors were evaluated for their effects on endodontic posttreatment pain. Patients with no radiographic periapical lesions had significantly more pain than patients with periapical lesions at 8-, 24-, 48-, and 72-h posttreatment regardless of whether they received placebo or dexamethasone. No other patient or treatment factors correlated with posttreatment pain. Twenty-two patients in the subgroup receiving placebo reported an 81% incidence of posttreatment pain, 73% of them required posttreatment pain medications. By 72-h posttreatment, the incidence and severity of pain in the placebo group was minimal.

Adult↗
PubMed1989

The use of bone imaging to detect a periapical lesion of endodontic origin.

This article reports the detection of a lesion of dental origin in the mandible of a 41-yr-old male patient during a routine bone scan used as a follow-up to cancer therapy. Unfortunately, more than 1 yr passed before the patient's signs and symptoms became severe enough for a definitive diagnosis to be made. A comprehensive dental examination is indicated for patients with positive bone scans in the area of the oral cavity. The use of bone imaging can be recommended to complement the dental examination of high risk patients with suspected metastasis or when all routine dental diagnostic measures are inconclusive.

Adult↗
PubMed1996

Immunohistochemical detection of prostaglandins E2, F2 alpha, and 6-keto-prostaglandin F1 alpha in experimentally induced periapical inflammatory lesions in rats.

The immunohistochemical localization of prostaglandin (PG) E2, PGF2 alpha, and 6-keto-PGF1 alpha (a stable metabolite of PGI2) was demonstrated in rat periapical inflammatory lesions induced by opening the pulp chamber. Two wk postoperatively, suppurative periapical lesions were formed, and active bone resorption was seen surrounding these lesions. Immunohistochemical examination showed that macrophages infiltrating in inflammatory tissue were positively stained for the examined PGs. In some lesions, wherein acute inflammatory changes subsided and proliferation of fibroblasts started, the fibroblasts were positively stained for 6-keto-PGF1 alpha. Osteocytes and osteoblasts were also positive for 6-keto-PGF1 alpha not only in experimental animals, but also in untreated animals. However the staining intensity of the PG in these cells was higher in periapical lesions than in normal condition. These findings suggested that the cellular sources of the PGs in the periapical lesions are mainly macrophages and fibroblasts, and that the PGs produced by these cells, and possibly osteoblast and osteocytes, may contribute to the osteolytic resorption of periapical lesions.

6-Ketoprostaglandin F1 alpha↗
PubMed1997

Endodontic treatment in three cases of dens invaginatus.

Three cases of dens invaginated teeth with periapical lesions are presented in this article. In the first case the maxillary canine was treated with conventional endodontic treatment. The second and third cases, in mandibular and maxillary central incisors respectively, were treated surgically. Six months postoperatively all cases appeared radiographically to be improved.

Adolescent↗