Congenital malformations as related to deficiencies of riboflavin and vitamin B12, source of protein, calcium to phosphorus ratio and skeletal phosphorus metabolism.
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Calcium↗
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Multiple congenital abnormalities resulting from transitory deficiency of pteroylglutamic acid during gestation in the rat.
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Animals↗
Multiple congenital abnormalities in the rat resulting from riboflavin deficiency induced by the antimetabolite galactoflavin.
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Animals↗
Teratogenic effects of pantothenic acid deficiency in the rat.
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Animals↗
Effect of variations of rations on the incidence of teratogeny in vitamin E-deficient rats.
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Animals↗
The anatomy and development of the mutants pirouette, shaker-1 and waltzer in the mouse.
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Animals↗
Immunological status and longevity of radiation-chimaeras.
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Animals↗
The influence of maternal age on development of the skeleton of the mouse.
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Animals↗
MALFORMATIONS induced by vitamin A deficiency.
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Congenital Abnormalities↗
Congenital malformations induced by maternal dietary deficiency.
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Congenital Abnormalities↗
EMBRYONIC malformations in vitamin E-deficient turkeys.
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Animals↗
The modification of the teratogenic action of cortisone by parity.
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Congenital Abnormalities↗
Congenital malformations produced by amniotic-sac puncture.
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Amnion↗
Developmental anomalies in offspring of pregnant mice treated with nicotine.
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Animals↗
Fate of frog embryos implanted into forelimbs of adults.
No teratoma formation and very little growth and differentiation followed implantation of late gastrula-stage frog embryos into forelimbs of adults. This is attributed to poor blood supply and rigid walls of muscle and bone surrounding the implants. The fate of embryos in nonamputated limbs is compared with that of regeneration-promoting implants in amputated limbs.
Animals↗
Uterine vascular clamping: new procedure for the study of congenital malformations.
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Congenital Abnormalities↗
Experimental study of teratogenic effect of emotional stress in rats.
The teratogenic effect of audiovisual and immobilization stress was studied in rats. Groups of 15 animals were subjected from the 9th to the 12th day of pregnancy to one or the other of these types of stress or to stress combined with administration of vitamin A. It was shown that the stresses alone had no effect on congenital malformations. Immobilization stress seemed to potentiate the teratogenic effect of vitamin A.
Animals↗
Lactic acid accumulation as a cause of hypoxia-induced malformations in the chick embryo.
Most hypoxia-induced malformations are caused by a syndrome involving tremendous edema followed by formation of clear blisters and hematomas. These, in turn, mechanically interfere with development. Studies of blood pH and lactic acid indicate that lactate accumulation initiates this syndrome. The effects of lactic acid injections parallel those of subjection to hypoxia, confirming this conclusion.
Animals↗