The behavioural pattern of the non-specific reaction to pollen in comparison with reactions to histamine, compound 48-80, serotonin and bradykinin.
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Animals↗
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Results for “COMPOUND 48-80”
Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.
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[Oxygen consumption of isolated rat peritoneal mast cells during histamine liberation by compound 48-80].
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Adenosine Triphosphate↗
Histamine release from lung tissue of rat induced by bee venom fractions and compound 48-80.
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Animals↗
[Studies on the effects of compound 48-80 on complement activity].
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Animals↗
[On the question of mast cell degranulation by compound 48-80. Tachyphylaxis of isolated mast cells against 48-80, inhibition of degranulation by heparinoids, modification of tachyphylaxis by protamine].
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Animals↗
In vivo histamine levels with hypoxia and compound 48-80.
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Acidosis↗
[Inhibition of giant cell formation by compound 48-80 after infection with Herpesvirus hominis. II. The activation of lysosomes].
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Animals↗
Action of polymixin and compound 48-80 on calcification in vitro.
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Bone and Bones↗
[Reciprocal dissociativity of DNA synthesis and giant cell formation in Herpesvirus hominis caused by cytosine arabinoside and compound 48-80].
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Animals↗
[Inhibition of giant cell formation with the so-called Compound 48-80 after infection with Herpesvirus hominis].
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Adsorption↗
Morphological effects of Compound 48-80, polymyxin B sulfate, and dextran on rat skeletal muscle.
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Animals↗
Effect of estrogens on the mast cell degranulating action of compound 48-80.
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Animals↗
Effect of histamine-releasing agent (compound 48-80) on extraction healing in rats.
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Animals↗
[Effects of compound 48-80, reserpine and carrageenan on 6-sulfanilaminoindazole arthritis].
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Animals↗
Studies on heart anaphylaxis. V. Cross-desensitisation between antigen, anaphylatoxin, and compound 48-80 in the guinea-pig papillary muscle.
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Anaphylaxis↗
[Effect of a synthetic isocoumarin (F 1375) with respect to compound 48-80 (author's transl)].
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Animals↗
The effect of beta-adrenergic blocking drugs and inhibitors of phosphodiestease on histamine release from isolated mast cells.
Differences in the histamine liberation from isolated rat mast cells after beta-adrenergic blocking drugs were demonstrated. In equimolar concentrations histamine release was induced by Kö 1124, Kö 1500, Kö 1560, Kö 1561 and propranolol. Alprenolol, oxprenolol, propranolol, and trimepranol significantly decreased thehistamine release induced by compound 48-80. The release of granules from cells was inhibited quantitatively more than the release of histamine. This enabled us to surmise the selective effect of beta-adrenergic blocking drugs on cell membranes of mast cells. The possible mechanisms of release reaction are discussed.
Adrenergic beta-Antagonists↗
Adrenal mast cells modulate vascular and secretory responses in the intact adrenal gland of the rat.
Mast cells were identified in the rat adrenal gland, located in the walls of arterioles at the point at which they penetrate the connective tissue capsule. The mast cell products, histamine and serotonin, both caused dose-dependent increases in rates of perfusion medium flow and steroid secretion in the isolated, perfused rat adrenal gland in situ. Compound 48-80, a mast cell degranulator, caused a significant increase in perfusion medium flow rate and steroid secretion by the in-situ perfused rat adrenal. Administration of disodium cromoglycate, a mast cell stabilizer, before administration of ACTH(1-24) virtually abolished the normal flow rate increment and significantly attenuated the corticosterone secretory response to ACTH(1-24). These observations strongly suggest that adrenal mast cells modulate both vascular and secretory responses in the intact adrenal gland of the rat.
Adrenal Glands↗