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Results for “FLUORIDE POISONING”

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PubMedDate not supplied

[Effect of different kinds of Ocean paste on rat liver biochemical indices in sodium fluoride poisoning].

The paper is concerned with the effect of diets including three chemically different types of the "Ocean" paste on biochemical characteristics (glycolysis, the content of phosphofructokinase, dehydrogenase, glucoso-6- phosphate, demethylase, dimethylalaniline hydroxylase, ascorbic acid) of the liver of rats exposed to sodium fluoride overdosage. The test diets were compared with those containing conventional sources of protein (casein, beef). Acute exposure to sodium fluoride caused changes in the biochemical characteristics. The most appreciable changes were seen in the group of rats fed beef. As compared with control, the shifts in the biochemical characteristics were negligible provided the rats exposed to acute and chronic poisoning received the diet supplemented with the paste. No morphological changes were found in the internal organs. The data obtained indicate that the type C "Ocean" paste has a protective action in sodium fluoride poisoning.

Animals↗
PubMedDate not supplied

Disposition of fluoride in a fatal case of unsuspected sodium fluoride poisoning.

A case of suicidal ingestion of sodium fluoride roach powder by a 33-year-old black woman is presented. Disposition of fluoride (mg/l or mg/kg) was: bile, 3.4; gastric content, 225; kidney, 16; liver, 8.6 and urine, 295. No history of roach powder ingestion was available at autopsy. This case illustrates the need for extensive toxicological screening to determine if fatal poisoning has occurred when histopathological findings are unremarkable.

Adult↗
PubMed1980

Acute fluoride poisoning leading to fatal hyperkalemia.

Marked peaking of the T waves later recognized as being due to hyperkalemia was noted in a patient with acute fluoride intoxication before developing refractory ventricular fibrillation. Such T wave changes have not been previously described in fluoride intoxication and should alert one to the presence of hyperkalemia complicating this condition, the presence of which may herald the onset of lethal ventricular arrhythmias.

Acute Disease↗
PubMed1994

Peanut butter and fatal fluoride poisoning. A case of mistaken identity.

We report here a case of an adult seeking a late night peanut butter snack who mistook for peanut butter a fluoride containing glass etching compound. The glass etching compound was of a color and consistency similar to peanut butter and was packaged in a similar appearing container. The decedent survived for six hours after the fluoride ingestion. He was admitted to the emergency room in stable condition with severe gastrointestinal symptoms. His condition deteriorated rapidly prior to death with abrupt cardiac arrest and profound hypocalcemia (5.5 mg/dl). Autopsy showed only gastric hyperemia and discoloration with a postmortem blood fluoride level of 19 mg/L.

Arachis↗
PubMed2004

Fatality due to acute fluoride poisoning in the workplace.

We report a case of a 65-year-old worker who suffered a third-degree skin burn to 5% of his total body surface area as a result of being splashed in the face with hydrofluoric acid (HF). He died shortly thereafter without having received adequate first aid. His serum fluoride concentration was markedly increased at 6.38 mg/dl with hypocalcemia and hyperkalemia. We concluded that he died of HF poisoning. In this case he might have managed to avoid death if he had not been working alone and if he had received adequate first aid on the scene as soon as possible. We re-emphasize the need for the immediate initiation of first aid on the scene and the distribution of information on the risks of HF to workers.

Accidents, Occupational↗