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At least 73 records · Page 4Linked to original sources
PubMed1963

ACTINOMYCIN: INHIBITION OF CORTISONE-INDUCED SYNTHESIS OF HEPATIC GLUCONEOGENIC ENZYMES.

The cortisone-induced de novo synthesis of liver glucose-6-phosphatase, fructose-1, 6-diphosphatase, aldolase, and lactic dehydrogenase was prevented by injections of Actinomycin D in the rat. In the in vitro assay systems, addition of actinomycin exerted no effect on the enzyme activities examined. The evidence favors the concept that the increased gluconeogenesis induced by corticoids entails at a certain stage an increased rate in the synthesis of gluconeogenic enzymes.

Cortisone↗
PubMed1965

GLUCOSE-6-PHOSPHATASE: REEXAMINATION OF THE RNA-INDUCED ACTIVITY IN MOUSE ASCITES TUMOR CELLS.

The glucostat method for the measurement of glucose released by glucose-6-phosphatase has been reexamined; it is accurate and sensitive enough to measure glucose-6-phosphatase activity in mouse ascites tumor cells. In seven different experiments, treatment with RNA from liver led to increases in enzyme activity varying between 125 to 200 percent. The activity of induced enzyme is optimum from pH 6 to 6.4.

Animals↗
PubMed1965

CYTOENZYMATICAL DIFFERENTIATION OF NORMAL AND NEOPLASTIC PLASMA CELLS.

A histochemical method is described of differentiating between normal and neoplastic cells in plasma using the activity of various enzymes characteristic of plasma cells. It was found that in both types of cell increased or diminished activity of cytoenzymes is a useful means of distinguishing between normal and abnormal cells.

Adenosine Triphosphatases↗