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PubMed1992

Impact of lung surfactant therapy on chronic lung diseases in premature infants.

Bronchopulmonary dysplasia (BPD) and chronic lung disease remain common complications of prematurity. This article addresses the evolution of BPD since its description in 1967, and the impact of surfactant replacement therapy on the incidence and characteristics of BPD. It also addresses the emergence of a form of chronic lung disease now seen in surfactant-treated premature infants who had no acute lung disease.

Birth Weight↗
PubMed1998

[Follow-up of the premature infant: prevention of severe diseases and sudden death. Role of polysomnography].

Incidence and severity of apnea and bradycardia in preterm infants may be related to the immaturity of the respiratory control mechanisms. In addition, there is a steadily increasing risk for sudden death with decreasing birth weight or gestational age during the first year of age, justifying the search for a specific preventing strategy for very low birth weight (VLBW) infants. The purpose of this paper is to define practical preventive guidelines for VLBW infants at the time of discharge from the neonatal intensive care units. Polysomnography appears as a useful method to detect those among the VLBW infants who are at risk of life threatening events and may need home monitoring.

Apnea↗
PubMed2000

"What can I do to enhance the development of a premature infant with chronic lung disease?".

CASE. Timmy was born at 32 weeks of gestation after an uncomplicated pregnancy until there was a spontaneous rupture of the membranes and preterm labor associated with chorioamnionitis. A 2-month hospitalization in the neonatal intensive care unit (NICU) was associated with pneumonia, a Grade II intraventricular hemorrhage, chronic lung disease, and a slow weight gain in the nursery. He was discharged to home with plans for ongoing care by his pediatrician. The primary care pediatrician attended a multidisciplinary conference with the NICU staff and Timmy's parents. At the time of discharge from the nursery, at 38 weeks postconceptual age, Timmy still required oral diuretics and supplemental oxygen, as well as other medications such as iron. Timmy's respiratory rates were between 40 and 60 breaths per minute at rest, with mild intercostal retractions. He was discharged with a cardiorespiratory monitor. The discharge examination revealed mild to moderate symmetrical hypotonia with intact deep tendon reflexes, shoulder girdle weakness, and a mild head lag. Timmy would regard a human face and a bright object and would follow them briefly. He became active and would thrash his extremities with minimal tactile, bright light, or auditory stimulation. Typically, he settled slowly with swaddling and a pacifier. Nursing was slow to develop; he was currently receiving one half of his calories at the breast and the remainder of his calories from bottle-feeding of fortified expressed breast milk. As she prepared for the first office visit with Timmy and his parents, the pediatrician asked herself, "What can I do to enhance the developmental outcome for this child?"

Bronchopulmonary Dysplasia↗