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Results for “TRICUSPID STENOSIS”

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At least 73 records · Page 4Linked to original sources
PubMed1999

Predominant tricuspid stenosis secondary to bacterial endocarditis in a patient with permanent pacemaker and balloon dilatation of the stenosis.

In a 49-year-old woman with sick sinus syndrome and a permanent VVI pacemaker, severe tricuspid stenosis and its clinical consequences developed 4 years after the attack of endocarditis. Besides the quite unusual occurrence of lead related tricuspid stenosis, successful treatment with balloon dilatation is the unique feature of this case.

Catheterization↗
PubMed1991

Percutaneous balloon valvotomy for the treatment of isolated tricuspid stenosis.

A 46-year-old woman with isolated tricuspid stenosis complained of increasing fatigue and dyspnea on exertion. Exercise Doppler echocardiography reproduced her symptoms and revealed a marked increase in trans-tricuspid gradient. Successful percutaneous balloon tricuspid valvotomy was performed, with resolution of her symptoms.

Catheterization↗
PubMed2000

Pacemaker lead related tricuspid stenosis: a report of two cases.

Only four cases of tricuspid stenosis related to endocardial pacemaker leads have been reported. Two further cases associated with perforation of a tricuspid valve leaflet by a pacemaker lead are presented: a 46 year old woman and a 60 year old man. It is possible that tricuspid valve disease related to endocardial pacemaker and non-thoracotomy defibrillator leads is underrecognized. Diagnosis requires clinical suspicion and the use of Doppler echocardiography. Recent evidence of fibrosis affecting the tricuspid valve in hearts from patients who have had non-thoracotomy defibrillator implants suggests that this problem could be more common in the future.

Echocardiography, Doppler↗
PubMed1989

A prospective study comparing the haemodynamic with the cross-sectional echocardiographic diagnosis of rheumatic tricuspid stenosis.

The value of cross-sectional echocardiography in the diagnosis of tricuspid valve stenosis is not clearly established. We prospectively studied by cardiac catheterization 42 consecutive patients, with a mean age of 29 +/- 11 years, who exhibited the cross-sectional echocardiographic features of tricuspid valve stenosis, defined as: diastolic doming of all three tricuspid leaflets and leaflet thickening with restrictive motion. To expose occult and amplify borderline tricuspid diastolic gradients, simultaneous right atrial and right ventricular pressures were recorded in the basal state, after incremental infusions of normal saline to 200, 400, 500, 700 or 1000 ml until a mean right atrial pressure of 12 mmHg was achieved, and finally after intravenous administration of 0.6 mg of atropine. Eighteen patients, Group 1, (43%) exhibited mean tricuspid diastolic gradients greater than 2 mmHg after saline infusion, increasing from a mean of 4 +/- 2 to 9 +/- 3 mmHg, (P less than 0.001), 14 (33%) having gradients greater than 2 mmHg in the basal state, together with four (10%) increasing from 1.7 +/- 0.2 to 4.5 +/- 1.2 mmHg (P less than 0.01) after provocation with fluid challenge. In the remaining 24 patients, Group 2, (57%) the mean tricuspid diastolic gradient was less than 2 mmHg, both at rest and after provocative manoeuvres. We conclude that the cross-sectional echocardiographic features of tricuspid valve stenosis are not a precise indicator of tricuspid valve stenosis. Provocative manoeuvres during haemodynamic studies are required to expose occult or amplify borderline tricuspid diastolic gradients in a minority of patients with the cross-sectional echocardiographic features of tricuspid stenosis.

Adolescent↗
PubMed1989

Rheumatic tricuspid stenosis without involvement of the mitral valve.

We describe a case of rheumatic tricuspid stenosis without involvement of the mitral valve. Rheumatic disease of the tricuspid valve is very rare in the absence of concomitant mitral valvar disease. We suggest that the closing pressure at the valve is not the only factor governing localization of the rheumatic process.

Echocardiography↗
PubMed1990

Percutaneous double balloon tricuspid valvotomy for severe tricuspid stenosis: 3-year follow-up study.

We performed percutaneous double balloon tricuspid valvotomy in four patients (36 +/- 12 years of age), with severe symptomatic rheumatic tricuspid stenosis and mild mitral valve disease. Two 9F Meditech balloon catheters, 15-20 mm diameter, were positioned simultaneously across the tricuspid valve and inflated up to five atmospheres pressure achieving valvotomy. After balloon valvotomy there was symptomatic improvement, and the Doppler tricuspid valve area was increased from a mean of 1 +/- 0.2 to 2.2 +/- 0.2 cm2 (P less than 0.001). At 3-year follow-up the symptomatic improvement persisted and the mean tricuspid valve area was maintained at 2.2 +/- 0.1 cm2. During the follow-up period there was no increase in degree of tricuspid regurgitation assessed by colour Doppler. We conclude that 3 years after balloon valvotomy of the tricuspid valve, the 120% increase in valve area persists, with no objective evidence of restenosis.

Adult↗
PubMed1992

[A successful case of tricuspid valvulectomy for Ebstein's anomaly with severe tricuspid stenosis].

A 44-year-old male was admitted to our hospital complaining of general fatigue and dyspnea on exertion with congestive heart failure (NYHA IV). Cardiac catheterization and angiogram revealed Ebstein's anomaly with severe tricuspid stenosis and functioning small right ventricle. Because of tiny right ventricular chamber, the usual surgical treatment for Ebstein's anomaly i.e. tricuspid valve replacement or valvuloplasty were deemed to be inappropriate. Tricuspid valvulectomy was performed to relieve the inflow stenosis of the right ventricle and excellent symptomatic relief was achieved. It is very rare to seen an adult case of Ebstein's anomaly with severe tricuspid stenosis and functioning small right ventricle. Tricuspid valvulectomy may be justified to use in this rare condition.

Adult↗
PubMedDate not supplied

TRICUSPID stenosis.

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Constriction, Pathologic↗
PubMed1958

[Tricuspid stenosis].

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Constriction, Pathologic↗
PubMed1985

Uncommon pattern of tricuspid stenosis in Ebstein's anomaly.

The echocardiographic and cineangiographic features of an unusual type of tricuspid stenosis in Ebstein's anomaly are described. The displaced septal and mural leaflets of the tricuspid valve are inserted to an anomalous apical muscle shelf and cause a restrictive communication between the atrialized portion and the trabecular zone of the right ventricle. The antero-superior leaflet is not involved.

Adult↗
PubMed1992

Modified Fontan procedure for severe Ebstein's malformation with predominant tricuspid stenosis.

Two children, aged 7 and 6 years, had severe Ebstein's malformation of the tricuspid valve and underwent operation because of progressive cyanosis (saturation, 70%). Preoperative angiography demonstrated linear attachment of the distal edges of the displaced anterosuperior and mural leaflets, leaving only a "keyhole" communication between the atrialized and functional portions of the right ventricle. Operation was directed toward completely excising these dysplastic leaflets, which were obstructing the flow of blood. The continuity between the functional right ventricle and pulmonary trunk was left intact. In addition, an atriopulmonary truncal connection was established. Postoperatively, both children were in functional class I with arterial saturation of 95%. Angiography and Doppler echocardiography demonstrated that there was a dominant phase of systolic flow of blood up the pulmonary trunk from the right ventricle, and also diastolic filling of the pulmonary arteries through the atriopulmonary truncal connection. The filling patterns did not change over time for the follow-up period of 4 and 1.5 years, respectively. This modified Fontan procedure appears to be an effective surgical alternative for some patients with severe Ebstein's malformation and predominant tricuspid stenosis.

Child↗