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Results for “Uveitis, Intermediate”

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PubMed1992

[Intermediate uveitis].

In a group of 50 patients with intermediary uveitis (IU) the authors evaluated the clinical finding, diagnosis with which the patients were referred to the institute and considered the problem of corticotherapy. The majority of patients was referred with a vague and inaccurate diagnosis: uveitis chronica (34%) and uveitis posterior (20%). 96% patients were treated by locally applied corticosteroids which are justified only in case of marked participation of the anterior segment of the eye. The development of posterior subcapsular cataract in 20% of the patients is according to the authors associated with overdosage of local corticotherapy. They recommend general and parabulbar corticotherapy only if cystoid oedema of the macula and oedema of the papilla develops, or in case of massive formation of an exudate on the pars plana. In patients with IU corticosteroid treatment should be indicated with great care, as complications may develop which are more serious than the disease.

Adult↗
PubMed1990

[Retinal changes in intermediate uveitis].

The authors characterize the clinical unit of intermediary uveitis. They give an account of their own experience with the diagnosis and treatment of this diseases and summarize contemporary views on its aetiopathogenesis and therapy.

Adolescent↗
PubMed1989

[Introduction to the round table discussion on intermediate uveitis].

Intermediary uveitis is one of the most mysterious and difficult to treat among all inflammations of the eye. From a recent statistic stemming from 2 different series of 20 cases of our patients, the following data have been found: all cases seen after 4 years of evolution are still evolutive; with 8 years follow-up, 42% are still evolutive. Recent findings supplied with the help of angiofluorophotometry give interesting details concerning the physiopathology of the disease. So, the leakage through the hemato-ocular barriers would appear not only as the result of a defect in anterior or posterior barrier but a defect of the entire barrier. Even after the clinical healing of the disease, the barrier remains often permeable. Therefore, the prolongation of the evolution of an intermediary uveitis may be the consequence of not necessarily an immune process but of a definitive anatomic sequellae.

Chronic Disease↗